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OIE RECOGNITION OF BSE STATUS MEMBERS RESOLUTION XXI

flounder

Well-known member
Wednesday, June 11, 2008

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

snip...

CONSIDERING THAT

1. Adoption of subsequent Resolutions* since the 67th General Session of the OIE International Committee has established a procedure for annually updating a list of Members, categorised by their BSE risk according to the provisions of theTerrestrial Code,

2. During the 70th General Session, the International Committee adopted Resolution No. XVIII asking Members applying for a BSE risk evaluation to meet part of the costs sustained by the OIE Central Bureau in the evaluation process,

3. During the 72nd General Session, the OIE adopted Resolution No. XXI requesting the Director General to inform Delegates of Members whose country or zones are recognised with regard to their BSE risk status should annually confirm during the month of November whether their risk status and the criteria by which their status was recognised have remained unchanged,

4. Information published by the OIE is derived from declarations made by the official Veterinary Services of Members. The OIE is not responsible for inaccurate publication of a Member disease status based on inaccurate information, changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau, subsequent to the time of declaration of the BSE risk status.

THE COMMITTEE

RESOLVES THAT

1. The Director General publish the following list of Members recognised as having a negligible BSE risk in accordance with Chapter 2.3.13. of the Terrestrial Code:

Australia, Argentina, Finland, Iceland, New Zealand, Norway, Paraguay, Singapore, Sweden and Uruguay.

2. The Director General publish the following list of Members recognised as having a controlled BSE risk in accordance with Chapter 2.3.13. of the Terrestrial Code:

Austria Belgium Brazil Canada Chile Chinese Taipei Cyprus Czech Republic Denmark Estonia France Germany Greece Hungary Ireland Italy Latvia Lichtenstein Lithuania Luxembourg Malta Mexico Netherlands Poland Portugal Slovak Republic Slovenia Spain Switzerland United Kingdom United States of America

AND

3. The Delegates of these Members will immediately notify the Central Bureau if BSE occurs in their countries or their territories.

_________

(Adopted by the International Committee of the OIE on 27 May 2008)

* 67th General Session (GS) Resolution No (Res) XVI and Res XI; 69th GS Res XV, and 71st GS Res XXII, 72nd GS Res XXIV and Res XXI..

http://www.oie.int/eng/info/en_statesb.htm?e1d6

IN A NUT SHELL ;

(Adopted by the International Committee of the OIE on 23 May 2006)

11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,

http://www.oie.int/eng/Session2007/RF2006.pdf

bought and paid for by your local cattle dealer $$$

IN my opinion the WOAH/OIE is nothing more than a organized bunch of lobbyist for the members Countries in support of there INDUSTRY, bound together as one, with the only purpose of open trade for there precious commodities and futures. Speaking only of BSE, they failed at every corner, and then just said to hell with it, well just trade all strains of TSE globally.

snip...

NOW, ask yourself why not one single mad cow has been documented in the USA since the Honorable Phyllis Fong of the OIG did the end around Johanns, Dehaven et al ??? found two atypical BSE or BASE cases and they flat shut it down i tell you. IF the OIE gives a favorable rating, IF the OIE gives any other rating but the lowest, poorest possible BSE/TSE rating, the OIE will have sealed there fate once and for all, because most of the world knows the truth about the USA and there mad cows. THE OIE will then be able to stand side by side with the USA, and proudly claim to have sold there soul to the devil, all for a buck, commodities and futures, to hell with human health. A 'CONTROLLED' RATING IS EXACTLY what the OIE will get if that is what they classify the USA as a 'CONTROLLED RATING'. IT will be controlled by Johanns, Dehaven, and GW. IT WILL BE RIGGED in other words. but that is nothing new, it's been rigged for years. ...

snip...SEE FULL TEXT with facts and sources @ ;

http://usdavskorea.blogspot.com/2008/06/oie-recognition-of-bse-status-of.html

http://organicconsumers.org/forum/index.php?showtopic=1566

TSS

---------- https://lists.aegee.org/bse-l.html ----------
 

Mike

Well-known member
bought and paid for by your local cattle dealer $$$

Flounder there are lots of cattle dealers around that have never had a bit of interaction with the people running the BSE testing program.

I'm getting pretty sick of you puttin the BSE fiasco on the back of the U.S. cattleman when they haven't a clue of what's really going on and have no control if they did.

Why don't you pack your happy cattlemen downgrading azz up and clear out of here.

Maybe go back to school and get a diploma???????????????/ :roll:
 

flounder

Well-known member
Mike said:
bought and paid for by your local cattle dealer $$$

Flounder there are lots of cattle dealers around that have never had a bit of interaction with the people running the BSE testing program.

I'm getting pretty sick of you puttin the BSE fiasco on the back of the U.S. cattleman when they haven't a clue of what's really going on and have no control if they did.

Why don't you pack your happy cattlemen downgrading azz up and clear out of here.

Maybe go back to school and get a diploma???????????????/ :roll:


grow up mike. your childish 'words' will never hurt me. i will clear out when more people like you become more educated. you know you cannot argue with the science or the politics that dictates it. i will keep shoving both at ya until you and some others wake up and smell the BSe. have a nice day :tiphat: :wave:


p.s. about my diploma you and old fedup2, and a few others are so worried about are so worried about :cry2: this is a play of last resorts for those that have no science to throw back. your showing your in a desperate mode. everyone knows it. it's not becoming of you :lol:


again, you cannot argue with the science or the politics that dictate it.


i will remind you as i did old fedup2 ;


fedup2 said:
Do I dare answer your question! You are one arrogant sob. As I stated before, if Japan demands testing, test them and sell them. Why would it be any different here. If the American people demand testing, test them and sell them! But! The American people are not demanding testing for BSE. Despite your best efforts, only your peta and vegi-head sites are hearing your cry of wolf & they don’t eat beef anyway! As far as having bigger fish to fry, that is just your ego talking, floundered or should I say floundering! I believe most people can see through your bs substantiated only by more of your own bs. Oh ya, I forgot about your so called experts, “looks like, could be, maybe, this leans toward, blah blah! As far as your wanna-be insult, "you dance like a girl with a skirt on" :roll: :roll: :roll: I have to give you credit for one thing, that was the dumbest @ss wanna-be insult that I ever heard! :lol: :lol: :lol: Most people already have you figured out. http://consumerfreedom.com/article_detail.cfm/article/138 Terry Singletary -- A retired machinist and high school dropout, Terry Singletary suffered the tragic loss of his mother to “sporadic” Creutzfeldt-Jakob disease (CJD) in 1997. Desperate to find an explanation for his mother’s death, he has devoted himself to the sad and fruitless task of connecting her death to her diet. Various reports confirm that Mrs. Singletary’s life was claimed by the most common sub-type of CJD (one that accounts for 70 percent of “sporadic” cases). Sporadic CJD, unlike its newer “variant,” is not linked to meat. As the self-appointed international coordinator of CJD Watch, an organization he co-founded with social worker Deborah Oney, Singletary is cited in media reports as an apparent expert on tracking mad cow disease. This despite his lack of formal education and the absence for support from any credible academic, medical or scientific authority. His sensationalist allegations about the safety of U.S. beef have found their way into hundreds of newspapers and broadcasts. Singletary moderates a mad-cow discussion forum run by a vegetarian activist group; his contributions account for more than half the traffic on the “BSE-L” mailing list, which is generally read by real scientists. Animal rights activists and other food-scare artists frequently refer to him as “Dr. Terry Singletary,” apparently an honorary degree as he has yet to finish high school. Like many activists, Singletary ignores overwhelming epidemiological and laboratory evidence that rules out a connection between sporadic CJD and beef. Relying entirely on shallow circumstantial evidence and frequent repetition of claims which have been publicly refuted as false, he also blindly insists upon a mad-cow with Alzheimer’s, Parkinson’s, and Lou Gehrig ’s disease. His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.

Hello there fedup2. i thought i must comment on your latest BSe, with a few scientific facts. i know how much you hate to hear about them.

1st off, if i wanted to insult you, you would know it. your child like insults and name calling only show what little of a man you really are. like i said before, call me what you like, but answer the question. in which you kinda finally did. but at the same time, you tried to make like the USA consumer does not want there beef tested for BSE. you claim that USA costumer does not want this, and i beg to differ. i could give you a list of about 500+ on cjdvoice that would challenge you on that claim. me being one of them. i even know ranchers that want USA beef testing for BSE.

2ndly, i want to personally thank you for posting that article on consumerfreedom site. i love that article, and every time someone tries to discredit me with it, all it does is give me more credit, because everything i wrote in the journal of neurology (peer review), has come to pass. i can't believe those folks still have that article up. let's take another look at it ;

Terry Singletary -- A retired machinist and high school dropout, Terry Singletary suffered the tragic loss of his mother to “sporadic” Creutzfeldt-Jakob disease (CJD) in 1997. Desperate to find an explanation for his mother’s death, he has devoted himself to the sad and fruitless task of connecting her death to her diet. Various reports confirm that Mrs. Singletary’s life was claimed by the most common sub-type of CJD (one that accounts for 70 percent of “sporadic” cases). Sporadic CJD, unlike its newer “variant,” is not linked to meat.<<<

well, in part that statement is wrong and right. i have consistently said that i have no idea what the source and route of the hvCJD my mother had, but it could be iCJD i.e. friendly fire, and it could be eating beef, or both, considering that the USA has no documented cases of the BSE, the last two cases were _atypical BSE_, (BSE being only documented in imported case from Canada;-) so it is therefore possible and most likely that the USA strain of CJD from consumption of beef, would not look like the nvCJD of the UK BSE. one thing i do know, it did not just happen spontaneously, like everyone wanted me to believe. and indeed, science has shown that some of these atypical BSE cases, some of these atypical BSE cases have shown that they are more like some subtypes of sporadic CJD. this has been well documented in science literature and i have posted it one this site more times than i care to count. so, in fact, these comments have come to pass as being correct. and the statement that i am a high school drop out is correct, however i did go on to get my GED in the Navy, and i did get a few hours of junior college under my belt, but still no PhDs. BUT, the statement here is simply wrong ;

Sporadic CJD, unlike its newer “variant,” is not linked to meat.<<<

the meat of the issue i.e. facts are, they simply do not know yet, but have more evidence that in fact, there are more than one strain of BSE in the bovine, one that looks more like some sub-types of sporadic CJD. AS most folks have figured out by now, sporadic CJD is not a single strain, it is simply a term used for CJD of unknown routes and sources, of many strains, and they are mounting. and why would this be so difficult to understand, when Scrapie, the key model to all TSEs, over 20 typical strains are documented, with atypical strains growing. the routes and sources of these TSE will be known eventually, once the government and the industry allow science to move along.

This study further confirms that BASE is caused by a distinct prion isolate and discloses a novel disease phenotype in cattle, closely resembling the phenotype previous reported in scrapie-inoculated cattle

*** and in some subtypes of inherited and sporadic Creutzfeldt-Jakob disease.

http://www.prion2007.com/pdf/Prion%20Book%20of%20Abstracts.pdf

The Italian cases (11 and 15 years of age) originally named bovine amyloidotic spongiform encephalopathy (BASE) were characterized by an unglycosylated protein band with a lower molecular mass (thus named L cases) and the predominance of the monoglycosylated band. In addition, immunohistochemical detection of PrPres in these cases found greater deposits in the cerebral cortex and thalamus versus the brain stem. The French cases found a higher molecular mass associated with the unglycosylated protein band and were called H cases (see figure 1). *** The different “strains” are now called atypical BSE. ...

full text, skroll down to page 6 ;

http://www.usaha.org/committees/reports/2006/report-fe-2006.pdf

MAD COW DISEASE terminology UK c-BSE (typical), atypical BSE H or L, and or Italian L-BASE

http://bse-atypical.blogspot.com/2008/03/mad-cow-disease-terminology-uk-c-bse.html

AS implied in the Inset 25 we must not _ASSUME_ that transmission of BSE to other species will invariably present pathology typical of a scrapie-like disease.

snip...

http://www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf

NOT to forget the 5 cases of the NOR-98 atypical scrapie documented in the USA in 2007, in five different states. WHICH pathologically looks like some sub-types of sporadic CJD, of which Stanely Prusiner warns of a public health risk ;

***The pathology features of Nor98 in the cerebellum of the affected sheep showed similarities with those of sporadic Creutzfeldt-Jakob disease in humans.

http://www.prion2007.com/pdf/Prion%20Book%20of%20Abstracts.pdf

Here we report that both Nor98 and discordant cases, including three sheep homozygous for the resistant PrPARR allele (A136R154R171), efficiently transmitted the disease to transgenic mice expressing ovine PrP, and that they shared unique biological and biochemical features upon propagation in mice. These observations support the view that a truly infectious TSE agent, unrecognized until recently, infects sheep and goat flocks and may have important implications in terms of scrapie control and public health.

Edited by Stanley B. Prusiner, University of California, San Francisco, CA, and approved September 12, 2005 (received for review March 21, 2005)

http://www.pnas.org/cgi/content/abstract/0502296102v1

http://nor-98.blogspot.com/

NEXT

As the self-appointed international coordinator of CJD Watch, an organization he co-founded with social worker Deborah Oney, Singletary is cited in media reports as an apparent expert on tracking mad cow disease. This despite his lack of formal education and the absence for support from any credible academic, medical or scientific authority. His sensationalist allegations about the safety of U.S. beef have found their way into hundreds of newspapers and broadcasts. Singletary moderates a mad-cow discussion forum run by a vegetarian activist group; his contributions account for more than half the traffic on the “BSE-L” mailing list, which is generally read by real scientists. Animal rights activists and other food-scare artists frequently refer to him as “Dr. Terry Singletary,” apparently an honorary degree as he has yet to finish high school. <<<

well lets see there fedup2, for a high school drop out, etc. etc. let's look at a few peer review journals ;

Journal of American Medical Association

Diagnosis and Reporting of Creutzfeldt-Jakob Disease Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.

Terry S. Singeltary, Sr Bacliff, Tex

1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323. FREE FULL TEXT

http://jama.ama-assn.org/

2 January 2000 British Medical Journal

U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well

http://www.bmj.com/cgi/eletters/320/7226/8/b#6117

15 November 1999 British Medical Journal vCJD in the USA * BSE in U.S.

http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406

JOURNAL OF NEUROLOGY

MARCH 26, 2003

RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States

Terry S. Singeltary:

http://www.neurology.org/cgi/eletters/60/2/176#535

NEXT

Like many activists, Singletary ignores overwhelming epidemiological and laboratory evidence that rules out a connection between sporadic CJD and beef. Relying entirely on shallow circumstantial evidence and frequent repetition of claims which have been publicly refuted as false, he also blindly insists upon a mad-cow with Alzheimer’s, Parkinson’s, and Lou Gehrig ’s disease. His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.<<<

THIS is my favorite part. please let me explain why.

first off, let's look at exactly what that article said ;

JOURNAL OF NEUROLOGY

MARCH 26, 2003

RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob

disease in the United States

Email Terry S. Singeltary:

[email protected]

I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey b y intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?

http://www.neurology.org/cgi/eletters/60/2/176#535

secondly, it was not epidemiological or laboratory evidence i was ignoring, it was the junk science the cattle industry and the bush administration is putting out, i.e. 'trust us, were your government speaking, and it's the truth'. TO date, i have proven them wrong with 'overwhelming epidemiological and laboratory evidence' of the atypical TSEs in human and animal. the shallow evidence they speak of has become stronger and stronger. the evidence in question has NOT been refuted as false i.e. common links to TSE and Alzheimers, in fact, the evidence is mounting. but all i stated in the JAMA journal and the Neurology journal was that CJD was being misdiagnosed and undercounted, due to Alzheimer's, and the fact CJD is not being reportable in every state, of every age group. ...

www.cjdsupport.net Newsletter issue 17 april 2008

the importance of gene types

We have known for many years that a common variation in a gene, known as the prion protein gene, is very important in determining the risk of developing prion diseases and how long it takes for the disease to develop when someone becomes infected. There are three genetic types in the UK population known as MM, VV and MV. So far, vCJD has only affected people with MM genetic type. Around 40% of healthy people in the UK are MM, about 50% are MV and around 10% are VV. It is likely that BSE prions will infect people of the VV and MV types also, but they may have much longer incubation periods (the time taken from being infected with prions until the brain disease becomes apparent) and may also develop a pattern of disease which may be different to vCJD. We suspect this again as result of research in laboratory mice wher e those that had the VV and MV genes had a different type of disease and different types or ‘strains’ of prions developed.

snip...

This unusual finding reminds us of the importance of keeping alert to the possibility that BSE prions will cause disease in individuals with different genetic types, who may develop a disease that may resemble sporadic CJD, or vCJD, or have a new pattern of disease.

Prion disease and gene types

Dr Simon Mead and Professor John Collinge, NHS National Prion Clinic

Reference: Creutzfeldt-Jakob Disease, Prion Protein Gene Codon 129VV, and a Novel PrPSc Type in a Young British Woman Simon Mead; Susan Joiner; Melanie Desbruslais; Jonathan A. Beck; Michael O’ Donoghue; Peter Lantos; Jonathan D. F. Wadsworth; John Collinge Arch Neurol. 2007;64(12):1780-1784.

NEXT

Alzheimer's, CJD, and prions

Association between Deposition of Beta-Amyloid and Pathological Prion Protein in Sporadic Creutzfeldt-Jakob Disease

Laura Debatin a Johannes Streffer b Markus Geissen c Jakob Matschke c Adriano Aguzzi a Markus Glatzel a, c

a Institute of Neuropathology, and b Division of Psychiatry Research, University Hospital Zurich, Zurich , Switzerland; c Institute of Neuropathology, University Medical Center Hamburg-Eppendorf, Hamburg , Germany

Copyright © 2008 S. Karger AG, Basel

Abstract

Background: Alzheimer’s disease (AD) and prion diseases such as sporadic Creutzfeldt-Jakob disease (sCJD) share common features concerning their molecular pathogenesis and neuropathological presentation and the coexistence of AD and CJD in patients suggest an association between the deposition of the proteolytically processed form of the amyloid precursor protein, B -amyloid (A B ), which deposits in AD, and the abnormal form of the prion protein, PrP Sc , which deposits in sCJD. Methods: We have characterized sCJD patients (n = 14), AD patients (n = 5) and nondemented controls (n = 5) with respect to the deposition of PrP Sc and A B morphologically, biochemically and genetically and correlated these findings to clinical data. Results: sCJD-diseased individuals with abundant deposits of A B present with a specific clinicopathological profile, defined by higher age at disease onset, long disease duration, a genetic profile and only minimal amounts of PrP Sc in the cerebellum. Conclusion: The co-occurrence of pathological changes typical for sCJD and AD in combination with the inverse association between accumulation of A B and PrP Sc in a subgroup of sCJD patients is indicative of common pathways involved in the generation or clearance of A B and PrP Sc in a subgroup of sCJD patients.

Copyright © 2008 S. Karger AG, Basel

http://content.karger.com/produktedb/produkte.asp?typ=fulltext&file=000121389

Regarding Alzheimer's disease

(note the substantial increase on a yearly basis)

http://www.bseinquiry.gov.uk/files/yb/1988/07/08014001.pdf

snip...

The pathogenesis of these diseases was compared to Alzheimer's disease at a molecular level...

snip...

http://www.bseinquiry.gov.uk/files/yb/1990/03/12003001.pdf

And NONE of this is relevant to BSE?

There is also the matter whether the spectrum of ''prion disease'' is wider than that recognized at present.

http://www.bseinquiry.gov.uk/files/yb/1990/07/06005001.pdf

Human BSE

snip...

These are not relevant to any possible human hazard from BSE nor to the much more common dementia, Alzheimers.

snip...

http://www.bseinquiry.gov.uk/files/yb/1990/07/09001001.pdf

IN STRICT CONFIDENCE

TRANSMISSION OF ALZHEIMER-TYPE PLAQUES TO PRIMATES

http://www.bseinquiry.gov.uk/files/yb/1993/01/05004001.pdf

please see full text ;

http://betaamyloidcjd.blogspot.com/

PLEASE see the old Duke, Pa, and other studies proving CJD being misdiagnosed as Alzheimer's

http://betaamyloidcjd.blogspot.com/2008/03/association-between-deposition-of-beta.html

Completely Edited Version

PRION ROUNDTABLE

PARKINSON'S, ALS, AND PRIONS STANLEY PRUSINER NOBEL PRIZE WINNER

snip...page 5

Dr. Prusiner made some comments about Parkinson's disease. He said a protein being improperly handled in the substantia nigra cells is what causes Parkinson's disease because it causes a decrease in dopamine production. His theory is that Parkinson’s disease is a prion-related problem, that Parkinson's disease is the direct result of those cells being killed because of prion accumulation that he believes is a familial transfer.

"Can it be transmitted by blood transfusions?" His answer is that We don't know.

We believe that ALS, or Lou Gehrig's disease, is a prion-related disease.

The last question that was asked of Dr. Prusiner was a simple question. The person said, "Dr. Prusiner, do you eat meat?" He answered immediately "Not in Europe." He was asked to elaborate, and he said these prion-related diseases are all over Europe. He said wherever you go, if you eat meat, you have the potential of picking them up. He believes the prions are in the muscle tissue. He does not believe they're just in the spinal column.

My son is an M.D. and they are getting a lot of information on these prion-related diseases and most of it is coming from Dr. Prusiner. The medical community has an understanding about prion-related diseases that's much different from that of the veterinary community, from what I can determine. ...

Accomplished this day, Wednesday, December 11, 2003, Denver, Colorado

The roundtable presentations and discussions were recorded. A transcript will be made available to the Academy of Veterinary Consultants, the American Association of Bovine Practitioners, and the Colleges of Veterinary Medicine throughout the United States and Canada. A condensed version translated for the livestock industry will be made available to educate livestock producers about prion related diseases.

http://www.r-calfusa.com/Newsletter/2004January.pdf

His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.<<<

AND several months later, my allegations were substantiated of the under counting of CJD victims, and Schonberger et al 'conceded' i was correct. i wonder why the consumerfreedom site does not post that $$$ probably because they are nothing more than a bunch of industry goons $$$ but here are the facts ;

THE PATHOLOGICAL PROTEIN

Hardcover, 304 pages plus photos and illustrations. ISBN 0-387-95508-9

June 2003

BY Philip Yam

CHAPTER 14 LAYING ODDS

Answering critics like Terry Singeltary, who feels that the U.S. under- counts CJD, Schonberger conceded that the current surveillance system has errors but stated that most of the errors will be confined to the older population.

http://www.thepathologicalprotein.com/

AND now in 2008 we find that indeed, my 'specific allegations', were confirmed, and that includes the Journal of American Medical Association, where i merely stated that CJD was being undercounted due to the fact in some cases, CJD was being misdiagnosed as Alzheimer's. That proven in many studies. BUT now we find evidence that there may be a common link between the two. hopefully one that will unlock all the unknowns to date. ...

The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html

***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.***

Progress Report from the National Prion Disease Pathology Surveillance Center

An Update from Stephen M. Sergay, MB, BCh & Pierluigi Gambetti, MD

April 3, 2008

http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45

Communicated by: Terry S. Singeltary Sr. <[email protected]>

[In submitting these data, Terry S. Singeltary Sr. draws attention to the steady increase in the "type unknown" category, which, according to their definition, comprises cases in which vCJD could be excluded. The total of 26 cases for the current year (2007) is disturbing, possibly symptomatic of the circulation of novel agents. Characterization of these agents should be given a high priority. - Mod.CP]

http://pro-med.blogspot.com/2007/11/proahedr-prion-disease-update-2007-07.html

http://www.promedmail.org/pls/askus/f?p=2400:1001:6833194127530602005::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1010,39963

There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection.

He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf

SO there fedup2, once again i have shown just how full of BSe you really are, and there is not a field or pasture that could hold it all.

for once, please bring something to the table that is not just BSe.

p.s. LOL, those fried up flounder were gooooood last night, wish you could have been here. ...

TSS


Mike, you are like fedup2, and a few others here, all mouth. again, bring something to the table worth reading. if i were you i would be pushing to test all cattle for BSE, and all livestock producing animals for TSE, for both humans and animals. close all the loopholes and then maybe pushing for the FDA and USDA to enforce the regulations they have on the books. and i would push for the feed ban especially. you know how they boast of that being the fire wall that protected all those children in the USDA DEADSTOCK DOWNER COW FOOD LUNCH PROGRAM.


Tuesday, May 27, 2008

FDA BSE/Ruminant Feed Inspections Firms Inventory Report Texas Legend Ranch
OAI 05/10/2008

http://madcowfeed.blogspot.com/2008/05/fda-bseruminant-feed-inspections-firms.html


Tuesday, April 29, 2008

Interference at the EPA - Science and Politics at the U.S. Environmental
Protection Agency

please see full text ;

http://sciencebushwhacked.blogspot.com/

DOWNER COW SCHOOL LUNCH PROGRAM

http://downercattle.blogspot.com/

http://downercattle.blogspot.com/2008/04/gao-report-on-humane-methods-of.html

http://downercattle.blogspot.com/2008/03/usda-certified-dead-stock-downer-cow.html

http://downercattle.blogspot.com/2008/03/usda-still-pandering-to-industry-still_27.html

http://downercattle.blogspot.com/2008/03/usda-still-pandering-to-industry-still.html

http://downercattle.blogspot.com/2008/03/recalled-beef-from-chino-slaughterhouse.html

http://downercattle.blogspot.com/2008/03/mad-cow-disease-typical-vs-atypical.html

http://downercattle.blogspot.com/2008/03/downer-cow-blues-senators-want.html

http://downercattle.blogspot.com/2008/03/mr-will-hueston-dvm-on-school-lunch.html

http://downercattle.blogspot.com/2008/03/california-downer-cow-meat-worker-i-was.html

http://downercattle.blogspot.com/2008/03/usda-questions-and-answers.html

http://downercattle.blogspot.com/2008/03/usda-to-hallmark-we-want-our-plaque.html

http://downercattle.blogspot.com/2008/03/house-committee-subpoenas.html

http://downercattle.blogspot.com/2008/03/california-lists-possible-recipients-of.html

http://downercattle.blogspot.com/2008/03/to-hard-working-employees-of-usda-and.html

http://downercattle.blogspot.com/2008/02/beef-recall-nationwide-school-lunch.html

http://downercattle.blogspot.com/2008/02/transcript-technical-briefing.html

[Docket No. 03-025IFA] FSIS Prohibition of the Use of Specified Risk
Materials for Human Food and Requirement for the Disposition of
Non-Ambulatory Disabled Cattle

03-025IFA 03-025IFA-2 Terry S. Singeltary

9/13/2005

http://www.fsis.usda.gov/OPPDE/Comments/03-025IFA/03-025IFA-2.pdf

[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE)

http://www.fsis.usda.gov/OPPDE/Comments/2006-0011/2006-0011-1.pdf

second line of lies... i mean defense i.e. fda mad cow feed ban ;

Friday, April 25, 2008

Substances Prohibited From Use in Animal Food or Feed [Docket No.
2002N-0273] (Formerly Docket No. 02N-0273) RIN 0910-AF46

http://madcowfeed.blogspot.com/2008/04/substances-prohibited-from-use-in.html



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