Mike
Well-known member
http://www.americanthinker.com/2011/08/the_racial_violence_that_dare_not_speak_its_name.html
Racist War In The U.S.
- Thread starter Mike
- Start date
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hypocritexposer
Well-known member
as per the obama plan.......
Sure hope everyone is intelligent enough to get rid of him in 2012.....last chance, for correction
Sure hope everyone is intelligent enough to get rid of him in 2012.....last chance, for correction
Mike said:
mike (Auburn U.) the racist bigot rides to incite again. is that all they taught you out there mikey? is that what you got your PhD's in, being a racist bigot, and a looser ta boot ???
old mikey has his hoody on again, and is riding to save the world from these pesky black folks. he thinks all blacks, hispanics, jews, asians, everything that is not white, is dirt. mikey, do you have your toy confederate and union soldiers out playing civil war again. old mike just can't wait for another civil war again. what a looser you are mike...
Bus stop beating becomes hate crime case
By ANITA HASSAN, HOUSTON CHRONICLE
Updated 10:28 p.m., Friday, September 2, 2011
The Harris County District Attorney's Office plans to prosecute the case of a white suspect accused of beating a black man at a downtown Houston bus stop last month as a hate crime, according to court records.
Prosecutors filed a motion Aug. 26 stating they believe 26-year-old Charles Allen Cannon - charged with misdemeanor assault in the beating of Yondell Johnson - committed the crime and intentionally selected the victim because of Cannon's "bias and prejudice against a group identified by race, color, national origin and ancestry," according to court documents obtained Friday by the Houston Chronicle.
State law specifies which offenses can be enhanced with hate crime allegations, according to authorities with the District Attorney's Office. For class A misdemeanors, as in this case, the maximum possible punishment - up to a year in jail - does not change with a hate crime finding. However, minimum jail time for the charge can be 180 days if the jury makes a finding of bias or prejudice. Otherwise, there is no minimum jail time for class A misdemeanor convictions.
Kristin Guiney, Cannon's attorney, declined to comment on the case.
It is unknown at this time whether three other suspects accused in the assault - Brian Kerstetter, 32, Michael Joe McLaughlin, 40, and Joseph Staggs, 48 - will also be prosecuted under the hate crime statute. Staggs and McLaughlin are scheduled to appear in court Tuesday, and Kerstetter's next court appearance is set for Sept. 21.
Cannon, McLaughlin and Staggs are in the Harris County Jail, and Kerstetter is reportedly in the City Jail. Bail for all four suspects is $50,000 each.
The incident occurred Aug. 13 at a bus stop at Travis and McKinney where Johnson, 29, was sitting shortly before midnight when the four men allegedly approached him. Johnson said one of the four men called him the N-word before the group proceeded to swing at him, eventually bringing him to the ground and kicking him in the face.
Johnson said he is glad prosecutors pursued the hate crime allegations and hopes the three other suspects will also be tried the same way. However, he still feels the punishment is not severe enough.
"A hate crime is something serious, and it could happen to anyone else," he said. "I don't think it should just be a misdemeanor charge."
Deric Muhammad, who held a news conference about Johnson's case in front of the Harris County Criminal Courthouse on Aug. 24, called the decision to prosecute the assault as a hate crime a "bittersweet" feeling.
"It is a relief, but it is disconcerting that the process was so slow," he said.
Muhammad echoed Johnson's sentiment, saying the punishment for all offenses prosecuted as hate crimes should be heftier.
"The system needs to be re-examined," he said. "Some lawmaker with a conscience should take a look at a way of writing a bill that would increase the punishment of a hate crime in the state of Texas."
[email protected].
http://www.chron.com/news/houston-texas/article/Bus-stop-beating-becomes-hate-crime-case-2153076.php
old mike and his old school buddies would just love another civil war. you really need to get a life mike...
Virulent Racism at Auburn Fraternities (posted 6 November 2001 · 1:38 pm)
Two all-white fraternities at Auburn, Delta Sigma Phi and Beta Theta Pi, have been temporarily suspended by University administration [AU] for vicious racial hatred at this year's Halloween party [SPLC]. Costumes included a hooded and robed Klansman, several white students in blackface, some wearing the shirts of a national Black fraternity, Omega Psi Phi, and one wearing blackface and a noose around his neck. Pictures included posed scenes of lynching and police brutality (all of the frat boys have big stupid grins on their faces—ha ha ha, genocide is really ******* funny, boys). I have known for a long time that the white fraternities at Auburn, and elsewhere, are some of the most reactionary networks on campus. There have been repeated problems from the fraternities for years in terms of racist, homophobic, and sexist discrimination, as well as a long record of repeated sexual violence against women. But this—this took me by surprise. And it disgusts me that anyone would feel like Auburn is a community that tolerates this ****.
http://radgeek.com/gt/2001/11/06/virulent_racism/
"All plaintiffs aver that the race discrimination practiced against them has been systematic, endemic and reflective of a long-term practice of intentional race discrimination practiced by defendant Auburn University against black employees, especially in the Athletic Department," the suit states.
http://blog.al.com/wire/2011/03/auburn_university_accused_of_r.html
http://radgeek.com/gt/2001/11/09/the_context/
http://withoutsanctuary.org/main.html
Posted: Fri Aug 12, 2011 7:22 am
Post
subject: Time To Get Tough Whitey!!!
Mike
Rancher
Joined: 10 Feb 2005
Posts: 15527
Location: Montgomery, Al
http://ranchers.net/forum/viewtopic.php?p=532037
the real racist here is you bigot...
hypocritexposer
Well-known member
http://www.americanthinker.com/2011/08/the_racial_violence_that_dare_not_speak_its_name.html
hey flipper what does that article make the American Thinker, It is the one that wrote the article!!!!!!! Guess that means they are racist too????
What about those flash mobs, the fact they target white and Hispanic stores and people to attack means they are jut in the wrong place at the wrong time EH,
and you have the gall to point fingers the real racist is you
As someone suggested go back to the bull session you stunk it up already
SOOOOEEEEEEEEEE get outta here piggy
Terry is this you???
Terry Singletary -- A retired machinist and high school dropout, Terry Singletary suffered the tragic loss of his mother to "sporadic" Creutzfeldt-Jakob disease (CJD) in 1997. Desperate to find an explanation for his mother's death, he has devoted himself to the sad and fruitless task of connecting her death to her diet. Various reports confirm that Mrs. Singletary's life was claimed by the most common sub-type of CJD (one that accounts for 70 percent of "sporadic" cases). Sporadic CJD, unlike its newer "variant," is not linked to meat.
As the self-appointed international coordinator of CJD Watch, an organization he co-founded with social worker Deborah Oney, Singletary is cited in media reports as an apparent expert on tracking mad cow disease. This despite his lack of formal education and the absence for support from any credible academic, medical or scientific authority. His sensationalist allegations about the safety of U.S. beef have found their way into hundreds of newspapers and broadcasts. Singletary moderates a mad-cow discussion forum run by a vegetarian activist group; his contributions account for more than half the traffic on the "BSE-L" mailing list, which is generally read by real scientists. Animal rights activists and other food-scare artists frequently refer to him as "Dr. Terry Singletary," apparently an honorary degree as he has yet to finish high school.
OH REALLY
Like many activists, Singletary ignores overwhelming epidemiological and laboratory evidence that rules out a connection between sporadic CJD and beef. Relying entirely on shallow circumstantial evidence and frequent repetition of claims which have been publicly refuted as false, he also blindly insists upon a mad-cow with Alzheimer's, Parkinson's, and Lou Gehrig's disease. His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.
Terry note the the last paragraph,
words like ignores overwhelming evidence,,,, refuted by scientists,,,,,shallow circumstantial evidence ,,,, frequent repetition of publicly false statements,,,,, blindly insists
Good grief how can you look at yourself in the mirror terry
:roll: :roll: :roll: :roll: [/b]
What about those flash mobs, the fact they target white and Hispanic stores and people to attack means they are jut in the wrong place at the wrong time EH,
and you have the gall to point fingers the real racist is you
As someone suggested go back to the bull session you stunk it up already
SOOOOEEEEEEEEEE get outta here piggy
Terry is this you???
Terry Singletary -- A retired machinist and high school dropout, Terry Singletary suffered the tragic loss of his mother to "sporadic" Creutzfeldt-Jakob disease (CJD) in 1997. Desperate to find an explanation for his mother's death, he has devoted himself to the sad and fruitless task of connecting her death to her diet. Various reports confirm that Mrs. Singletary's life was claimed by the most common sub-type of CJD (one that accounts for 70 percent of "sporadic" cases). Sporadic CJD, unlike its newer "variant," is not linked to meat.
As the self-appointed international coordinator of CJD Watch, an organization he co-founded with social worker Deborah Oney, Singletary is cited in media reports as an apparent expert on tracking mad cow disease. This despite his lack of formal education and the absence for support from any credible academic, medical or scientific authority. His sensationalist allegations about the safety of U.S. beef have found their way into hundreds of newspapers and broadcasts. Singletary moderates a mad-cow discussion forum run by a vegetarian activist group; his contributions account for more than half the traffic on the "BSE-L" mailing list, which is generally read by real scientists. Animal rights activists and other food-scare artists frequently refer to him as "Dr. Terry Singletary," apparently an honorary degree as he has yet to finish high school.
OH REALLY
Like many activists, Singletary ignores overwhelming epidemiological and laboratory evidence that rules out a connection between sporadic CJD and beef. Relying entirely on shallow circumstantial evidence and frequent repetition of claims which have been publicly refuted as false, he also blindly insists upon a mad-cow with Alzheimer's, Parkinson's, and Lou Gehrig's disease. His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.
Terry note the the last paragraph,
words like ignores overwhelming evidence,,,, refuted by scientists,,,,,shallow circumstantial evidence ,,,, frequent repetition of publicly false statements,,,,, blindly insists
Good grief how can you look at yourself in the mirror terry
:roll: :roll: :roll: :roll: [/b]
Mike
Well-known member
I just hope like heck flipper don't think he's hurting my feelings by calling me a racist....................................................................
Mike
Well-known member
flounder said:
Wrong Einstein. There are racists that come in every color.
If you think all racists are white, you're MUCH, MUCH more stupid than the average idiot we thought you were..
hopalong said:hey flipper what does that article make the American Thinker, It is the one that wrote the article!!!!!!! Guess that means they are racist too????
What about those flash mobs, the fact they target white and Hispanic stores and people to attack means they are jut in the wrong place at the wrong time EH,
and you have the gall to point fingers the real racist is you
As someone suggested go back to the bull session you stunk it up already
SOOOOEEEEEEEEEE get outta here piggy
Terry is this you???
Terry Singletary -- A retired machinist and high school dropout, Terry Singletary suffered the tragic loss of his mother to "sporadic" Creutzfeldt-Jakob disease (CJD) in 1997. Desperate to find an explanation for his mother's death, he has devoted himself to the sad and fruitless task of connecting her death to her diet. Various reports confirm that Mrs. Singletary's life was claimed by the most common sub-type of CJD (one that accounts for 70 percent of "sporadic" cases). Sporadic CJD, unlike its newer "variant," is not linked to meat.
As the self-appointed international coordinator of CJD Watch, an organization he co-founded with social worker Deborah Oney, Singletary is cited in media reports as an apparent expert on tracking mad cow disease. This despite his lack of formal education and the absence for support from any credible academic, medical or scientific authority. His sensationalist allegations about the safety of U.S. beef have found their way into hundreds of newspapers and broadcasts. Singletary moderates a mad-cow discussion forum run by a vegetarian activist group; his contributions account for more than half the traffic on the "BSE-L" mailing list, which is generally read by real scientists. Animal rights activists and other food-scare artists frequently refer to him as "Dr. Terry Singletary," apparently an honorary degree as he has yet to finish high school.
OH REALLY
Like many activists, Singletary ignores overwhelming epidemiological and laboratory evidence that rules out a connection between sporadic CJD and beef. Relying entirely on shallow circumstantial evidence and frequent repetition of claims which have been publicly refuted as false, he also blindly insists upon a mad-cow with Alzheimer's, Parkinson's, and Lou Gehrig's disease. His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.
Terry note the the last paragraph,
words like ignores overwhelming evidence,,,, refuted by scientists,,,,,shallow circumstantial evidence ,,,, frequent repetition of publicly false statements,,,,, blindly insists
Good grief how can you look at yourself in the mirror terry
:roll: :roll: :roll: :roll: [/b]
hopalong, your as dumb as a door knob just like your soul mate there mikey. i already told mike, i love it when ya'll get mad, have nothing credible to come back with, so you post this consumer freedom report on me, which i love, because since then, i have proven them wrong on all fronts. so thank you, you just keep on posting it dummy :lol:
however, let's take a closer look at just whom the center for the concumer freedom ccf, and just whom they really are shall we ;
The Center for Consumer Freedom (CCF) (formerly called the "Guest Choice Network (GCN)") is a front group for the restaurant, alcohol and tobacco industries. It runs media campaigns which oppose the efforts of scientists, doctors, health advocates, environmentalists and groups like Mothers Against Drunk Driving, calling them "the Nanny Culture -- the growing fraternity of food cops, health care enforcers, anti-meat activists, and meddling bureaucrats who 'know what's best for you.'"
CCF is registered as a tax-exempt, non-profit organization under the IRS code 501(c)(3). Its advisory board is comprised mainly of representatives from the restaurant, meat and alcoholic beverage industries.
http://www.sourcewatch.org/index.php?title=Center_for_Consumer_Freedom
http://en.wikipedia.org/wiki/Center_for_Consumer_Freedom
What Is the Center for Consumer Freedom, and Why Is It Attacking PETA?
The Center for Consumer Freedom is a nonprofit corporation run by lobbyist Richard Berman through his Washington, D.C.-based for-profit public relations company, Berman & Co. The Center for Consumer Freedom, formerly known as the Guest Choice Network, was set up by Berman with a $600,000 "donation" from tobacco company Philip Morris.
Berman arranges for large sums of corporate money to find its way into nonprofit societies of which he is the executive director. He then hires his own company as a consultant to these nonprofit groups. Of the millions of dollars "donated" by Philip Morris between the years 1995 and 1998, 49 percent to 79 percent went directly to Berman or Berman & Co.
Richard Berman is an influence peddler. He has worked out a scheme to funnel charitable donations from wealthy corporations into his own pocket. In exchange, he provides a flurry of disinformation, flawed studies, op-ed pieces, letters to the editor, and trade-industry articles, as well as access to his high-level government contacts, who are servants of the industries he represents.
Berman's name might sound familiar. In 1995, Berman and Norm Brinker, his former boss at Steak and Ale Restaurants, were identified as the special-interest lobbyists who donated the $25,000 that disgraced then-House Speaker Newt Gingrich, who was hauled before the House Ethics Committee for influence-peddling over the money. Berman and Brinker were lobbying against raising the minimum wage.
Richard Berman is a spin doctor. For example, he has argued against a Mothers Against Drunk Driving (MADD) initiative to lower the blood alcohol content (BAC) limit for drivers by claiming that the stricter limits would punish responsible social drinkers. He has claimed that U.S. Centers for Disease Control and Prevention (CDC) warnings about salmonella-related food poisoning are just "whipping up fear over food."
Here's how an internal Philip Morris memo described Berman's spin: "His proposed solution would broaden the focus of the 'smoking issue,' and expand into the bigger picture of over-regulation." Smoking won't kill you; over-regulation will.
Berman is "a one-man wrecking crew on important issues." His approach has been described as "misleading" and "despicable." Berman has been called "a tobacco company whore," but he's branched out since then.
Using "freedom of choice" as his battle cry, Berman has now taken on PETA and a number of other groups and organizations whose points of view could have an impact on the profits of his clients by waking consumers up. Berman's Guest Choice Network has an "advisory panel" whose members in 1998 included officials representing companies ranging from Cargill Processed Meat Products and Outback Steakhouse to Minnesota Licensed Beverage Association and Sutter Home Winery. Berman's clients are companies with vested interests in low employee wages; cheap, unhealthy restaurant-chain food, particularly meat; and tobacco, soft drink, and alcohol consumption—companies like Ruth's Chris Steakhouse, Armour Swift, and Philip Morris, whose product line includes Kraft Foods and everything from Marlboro cigarettes to Oscar Meyer wieners and which is a major shareholder in its former subsidiary Miller Brewing, now known as SABMiller.
PETA's recent successes in gaining fast-food industry concessions for more humane conditions for farm animals have sent ripples of fear through the food and beverage service industry. About the same time that McDonald's buckled to PETA's demands, Richard Berman changed his front group's name and stepped up his attacks.
The key to Berman's aggressive strategy is, in his own words, "to shoot the messenger ... we've got to attack their credibility as spokespersons,"—an interesting remark from someone whose background and funding so severely challenge his own credibility.
http://www.consumerdeception.com/index.asp
please see full text and more here mike ; Thursday, December 23, 2010
Alimentary prion infections: Touch-down in the intestine, Alzheimer, Parkinson disease and TSE mad cow diseases $ The Center for Consumer Freedom
http://betaamyloidcjd.blogspot.com/2010/12/alimentary-prion-infections-touch-down.html
Saturday, June 25, 2011 Transmissibility of BSE-L and Cattle-Adapted TME Prion Strain to Cynomolgus Macaque
Emmanuel Comoy,1,† Valérie Durand,1 Evelyne Correia,1 Sophie Freire,1 Jürgen Richt,2 Justin Greenlee,3 Juan-Maria Torres,4 Paul Brown,1 Bob Hills5 and Jean-Philippe Deslys1
1Atomic Energy Commission; Fontenay-aux-Roses, France; 2Kansas State University; Manhattan, KS USA; 3USDA; Ames, IA USA; 4INIA; Madrid, Spain; 5Health Canada; Ottawa, ON Canada†Presenting author; Email: [email protected].
The epidemiology of Transmissible mink encephalopathy (TME) indicates an alimentary origin. Several inter-species transmission experiments have not succeeded in establishing with certainty any natural reservoir of this prion strain, although both ovine and bovine sources have been suspected. Cattle exposed to TME develop a spongiform encephalopathy that is distinct from classical Bovine Spongiform Encephalopathy (c-BSE).
Inoculation of c-BSE to cynomolgus macaque provided early evidence of a possible risk to humans, and remains an important model to define the risk of both primary (oral transmission from cattle to primate) and secondary (intravenous intra-species transmission) exposures. We have also evaluated the transmissibility of other cattle prion strains to macaques, including L- and H- atypical forms of BSE, namely BSE-L and BSE-H, and cattle-adapted TME.
BSE-L induced a neurological disease distinct from c-BSE. Peripheral exposures demonstrate the transmissibility of BSE-L by oral, intravenous, and intra-cerebral routes, with incubation periods similar to c-BSE. Cattle-adapted TME also induced a rapid disease in cynomolgus macaque. The clinical features, lesion profile, and biochemical signature of the induced disease was similar to the features observed in animals exposed to BSE-L, suggesting a link between the two prion strains. Secondary transmissions to a common host (transgenic mouse overexpressing bovine PrP) of cattle-TME and BSE-L before or after passage in primates induced diseases with similar incubation periods: like the c-BSE strain, these cattle strains maintained their distinctive features regardless of the donor species and passages.
If the link between TME and BSE-L is confirmed, our results would suggest that BSE-L in North America may have existed for decades, and highlight a possible preferential transmission of animal prion strains to primates after passage in cattle.
=====================end...tss====================
Risk.16: Clinical Disease in Cattle Experimentally Inoculated with All Types of BSE
Catherine Graham,1,† Michel Levy,2 Ed Pajor,2 Garth McGregor,1 Rheana Flitton1 and Stefanie Czub1
1Canadian Food Inspection Agency; Lethbridge, AB Canada; 2Faculty of Veterinary Medicine; University of Calgary; Calgary, AB Canada†Presenting author; Email: [email protected].
Background. Classical, or C-type, bovine spongiform encephalopathy (BSE) has been extensively described in the literature. Recently, two novel forms of BSE, termed atypical BSE, have been reported in a number of countries. These new forms show differences in the biochemical characteristics of the prion protein and, where reported, tend to occur in aged animals but descriptions of clinical presentation are incomplete or absent.
Materials and Methods. Female Hereford/Angus cross calves were intracranially challenged at approximately five months of age with 1 ml of a 10% brain homogenate originating from Canadian field cases of BSE which had been previously classified as C-, L-, or H- type.
The animals were monitored during incubation period, and clinical disease is described using a standardized examination protocol. Incubation period, description and progression of clinical signs was recorded and videotaped for objective evaluation.
Results. All L- and H- type atypical BSE challenged animals began to display signs of clinical disease at approximately 11 months post inoculation, and disease progression was slow but constant until animals were euthanized. Clinical signs in all atypical BSE inoculated animals included hesitation at doors and gates, spontaneous muscle fasciculations and sensitivity to touch. Teeth grinding and excessive salivation are occasionally noted. Animals with L-type BSE are very anxious and show high levels of sensitivity to hand movement. One H-type animal shows periods of somnolence. Both H-type inoculated animals go down during handling and have difficulty rising and show sensitivity to movement around their head and neck area, but to a lesser degree than the L-type BSE inoculated animals. Interestingly, no locomotor abnormalities have been observed in either group.
C-type challenged animals remain normal at approximately 18 months post inoculation. Clinical disease in C-type inoculated animals from a previous transmission study was typically slow and intermittently displayed during the initial stages and after a period of two to four months was more consistent and progressive. Clinical signs in C-type BSE were as previously reported in the literature.
Discussion. The spectrum of clinical signs for all three types of BSE examined is similar. Incubation period is shorter for H- and L-type BSE as compared with C-type. Once clinical signs begin, progression is slow but relentless in atypical BSE, and more insidious in classical BSE. A summary of clinical signs presented in the three different types of BSE will be presented, and video of clinical disease will be displayed.
link url not available, please see PRION 2011 ;
http://www.prion2011.ca/files/PRION_2011_-_Posters_(May_5-11).pdf
=========================end...tss================
PLEASE NOTE *
Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.
snip...
The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...
http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf
snip...see full text ;
Saturday, June 25, 2011
Transmissibility of BSE-L and Cattle-Adapted TME Prion Strain to Cynomolgus Macaque
"BSE-L in North America may have existed for decades"
http://transmissiblespongiformencephalopathy.blogspot.com/2011/06/transmissibility-of-bse-l-and-cattle.html
Sunday, June 26, 2011
Risk Analysis of Low-Dose Prion Exposures in Cynomolgus Macaque
http://transmissiblespongiformencephalopathy.blogspot.com/2011/06/risk-analysis-of-low-dose-prion.html
Thursday, June 23, 2011
Experimental H-type bovine spongiform encephalopathy characterized by plaques and glial- and stellate-type prion protein deposits
http://transmissiblespongiformencephalopathy.blogspot.com/2011/06/experimental-h-type-bovine-spongiform.html
Wednesday, June 15, 2011
Galveston, Texas - Isle port moves through thousands of heifers headed to Russia, none from Texas, Alabama, or Washington, due to BSE risk factor
http://transmissiblespongiformencephalopathy.blogspot.com/2011/06/galveston-texas-isle-port-moves-through.html
Sunday, August 28, 2011
Rick Perry, Texas, BSE aka mad cow disease, CJD, and 12 years of lies there from
http://sciencebushwhacked.blogspot.com/2011/08/rick-perry-texas-bse-aka-mad-cow.html
Sunday, September 6, 2009
MAD COW USA 1997 SECRET VIDEO
http://madcowusda.blogspot.com/2009/09/mad-cow-usa-1997-video.html
Tuesday, August 30, 2011
A Review of Archived Canadian Scrapie Cases for Evidence of Bovine Spongiform Encephalopathy
http://transmissiblespongiformencephalopathy.blogspot.com/2011/08/review-of-archived-canadian-scrapie.html
Wednesday, March 31, 2010
Atypical BSE in Cattle
To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE.
When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.
This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle.
http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2
Thursday, August 12, 2010
Seven main threats for the future linked to prions
First threat
The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.
***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.
Second threat
snip...
http://www.neuroprion.org/en/np-neuroprion.html
http://prionpathy.blogspot.com/2010/08/seven-main-threats-for-future-linked-to.html
http://prionpathy.blogspot.com/
Rural and Regional Affairs and Transport References Committee
The possible impacts and consequences for public health, trade and agriculture of the Government's decision to relax import restrictions on beef Final report June 2010
2.65 At its hearing on 14 May 2010, the committee heard evidence from Dr Alan Fahey who has recently submitted a thesis on the clinical neuropsychiatric, epidemiological and diagnostic features of Creutzfeldt-Jakob disease.48 Dr Fahey told the committee of his concerns regarding the lengthy incubation period for transmissible spongiform encephalopathies, the inadequacy of current tests and the limited nature of our current understanding of this group of diseases.49
2.66 Dr Fahey also told the committee that in the last two years a link has been established between forms of atypical CJD and atypical BSE. Dr Fahey said that: They now believe that those atypical BSEs overseas are in fact causing sporadic Creutzfeldt-Jakob disease. They were not sure if it was due to mad sheep disease or a different form. If you look in the textbooks it looks like this is just arising by itself. But in my research I have a summary of a document which states that there has never been any proof that sporadic Creutzfeldt-Jakob disease has arisen de novo-has arisen of itself. There is no proof of that. The recent research is that in fact it is due to atypical forms of mad cow disease which have been found across Europe, have been found in America and have been found in Asia. These atypical forms of mad cow disease typically have even longer incubation periods than the classical mad cow disease.50
http://www.aph.gov.au/senate/committee/rrat_ctte/mad_cows/report/report.pdf
Based on the results obtained with in vitro conversion assays, the opinion concludes that there is probably no absolute molecular barrier to transmission of TSE agents between mammalian species. Results also suggest that these assays may be developed as a tool for quantifying the transmission barriers between species for different TSE agent strains; however, there is no means at the moment to transpose in vitro results into the likelihood of in vivo interspecies transmission.
-- Communicated by: Terry S Singeltary Sr
[ProMED-mail thanks Terry S Singeltary Sr for drawing attention to this comprehensive document which provides a current evaluation of experimental work designed to explore the zoonotic potential of the various recently recognised TSEs of domestic and other animals.
It is concluded that at present the only TSE agent demonstrated to be zoonotic is the classical BSE agent. Nor can it be entirely excluded at the present time that a small proportion of cases of sporadic CJD may be environmentally acquired. - Mod.CP]
******
http://www.promedmail.org/pls/apex/f?p=2400:1202:888892554804923::NO::F2400_P1202_CHECK_DISPLAY,F2400_P1202_PUB_MAIL_ID:X,88784
[Terry S. Singeltary Sr. has added the following comment:
"According to the World Health Organisation, the future public health threat of vCJD in the UK and Europe and potentially the rest of the world is of concern and currently unquantifiable. However, the possibility of a significant and geographically diverse vCJD epidemic occurring over the next few decades cannot be dismissed
http://whqlibdoc.who.int/publications/2003/9241545887.pdf
http://www.promedmail.org/pls/apex/f?p=2400:1001:568933508083034::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,82101
14th ICID International Scientific Exchange Brochure -
Final Abstract Number: ISE.114
Session: International Scientific Exchange
Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America update October 2009
T. Singeltary
Bacliff, TX, USA
Background:
An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.
Methods:
12 years independent research of available data
Results:
I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.
Conclusion:
I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.
http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf
Monday, May 23, 2011
Atypical Prion Diseases in Humans and Animals 2011
Top Curr Chem (2011)
DOI: 10.1007/128_2011_161
# Springer-Verlag Berlin Heidelberg 2011
Michael A. Tranulis, Sylvie L. Benestad, Thierry Baron, and Hans Kretzschmar
http://bse-atypical.blogspot.com/2011/05/atypical-prion-diseases-in-humans-and.html
Sunday, August 21, 2011
The British disease, or a disease gone global, The TSE Prion Disease
(see video here)
http://transmissiblespongiformencephalopathy.blogspot.com/2011/08/british-disease-or-disease-gone-global.html
Tuesday, August 30, 2011
Compliance Guidelines for Use of Video or Other Electronic Monitoring or Recording Equipment in Federally Inspected Establishments
http://transmissiblespongiformencephalopathy.blogspot.com/2011/08/compliance-guidelines-for-use-of-video.html
Saturday, July 23, 2011
CATTLE HEADS WITH TONSILS, BEEF TONGUES, SPINAL CORD, SPECIFIED RISK MATERIALS (SRM's) AND PRIONS, AKA MAD COW DISEASE
http://transmissiblespongiformencephalopathy.blogspot.com/2011/07/cattle-heads-with-tonsils-beef-tongues.html
Saturday, November 6, 2010
TAFS1 Position Paper on Position Paper on Relaxation of the Feed Ban in the EU Berne, 2010 TAFS
INTERNATIONAL FORUM FOR TRANSMISSIBLE ANIMAL DISEASES AND FOOD SAFETY a non-profit Swiss Foundation
http://madcowfeed.blogspot.com/2010/11/tafs1-position-paper-on-position-paper.html
Archive Number 20101206.4364 Published Date 06-DEC-2010 Subject PRO/AH/EDR> Prion disease update 2010 (11)
PRION DISEASE UPDATE 2010 (11)
http://www.promedmail.org/pls/apex/f?p=2400:1001:5492868805159684::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,86129
AND NOW THE SPECIFIED RISK MATERIAL (SRM) FIRE WALL BURNT DOWN.
the so called triple mad cow fire wall that the USDA, FSIS, APHIS, FDA, all boast about, in reality was, BSe.
BUT, the mad cow agent continues to spread, and apparently, it's been spreading in North America for decades...
what is Alzheimer's anyway ?
strictly NOT private and confidential $$$
Saturday, January 22, 2011
Alzheimer's, Prion, and Neurological disease, and the misdiagnosis there of, a review 2011
http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/alzheimers-prion-and-neurological.html
Friday, September 3, 2010
Alzheimer's, Autism, Amyotrophic Lateral Sclerosis, Parkinson's, Prionoids, Prionpathy, Prionopathy, TSE
http://betaamyloidcjd.blogspot.com/2010/09/alzheimers-autism-amyotrophic-lateral.html
Wednesday, January 5, 2011
ENLARGING SPECTRUM OF PRION-LIKE DISEASES Prusiner Colby et al 2011
Prions
David W. Colby1,* and Stanley B. Prusiner1,2
----- Original Message -----
From: David Colby
To: [email protected]
Cc: stanley@XXXXXXXX
Sent: Tuesday, March 01, 2011 8:25 AM
Subject: Re: FW: re-Prions David W. Colby1,* and Stanley B. Prusiner1,2 + Author Affiliations
Dear Terry Singeltary,
Thank you for your correspondence regarding the review article Stanley Prusiner and I recently wrote for Cold Spring Harbor Perspectives. Dr. Prusiner asked that I reply to your message due to his busy schedule. We agree that the transmission of CWD prions to beef livestock would be a troubling development and assessing that risk is important. In our article, we cite a peer-reviewed publication reporting confirmed cases of laboratory transmission based on stringent criteria. The less stringent criteria for transmission described in the abstract you refer to lead to the discrepancy between your numbers and ours and thus the interpretation of the transmission rate. We stand by our assessment of the literature--namely that the transmission rate of CWD to bovines appears relatively low, but we recognize that even a low transmission rate could have important implications for public health and we thank you for bringing attention to this matter.
Warm Regards, David Colby
--
David Colby, PhDAssistant ProfessorDepartment of Chemical EngineeringUniversity of Delaware
====================END...TSS==============
re-ENLARGING SPECTRUM OF PRION-LIKE DISEASES Prusiner Colby et al 2011 Prions
CWD to cattle figures CORRECTION
Greetings,
I believe the statement and quote below is incorrect ;
"CWD has been transmitted to cattle after intracerebral inoculation, although the infection rate was low (4 of 13 animals [Hamir et al. 2001]). This finding raised concerns that CWD prions might be transmitted to cattle grazing in contaminated pastures."
Please see ;
Within 26 months post inoculation, 12 inoculated animals had lost weight, revealed abnormal clinical signs, and were euthanatized. Laboratory tests revealed the presence of a unique pattern of the disease agent in tissues of these animals. These findings demonstrate that when CWD is directly inoculated into the brain of cattle, 86% of inoculated cattle develop clinical signs of the disease.
http://www.ars.usda.gov/research/publications/publications.htm?seq_no_115=194089
"although the infection rate was low (4 of 13 animals [Hamir et al. 2001])."
shouldn't this be corrected, 86% is NOT a low rate. ...
kindest regards,
Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518
Thank you!
Thanks so much for your updates/comments. We intend to publish as rapidly as possible all updates/comments that contribute substantially to the topic under discussion.
http://cshperspectives.cshlp.org/letters/submit
re-Prions David W. Colby1,* and Stanley B. Prusiner1,2 + Author Affiliations
1Institute for Neurodegenerative Diseases, University of California, San Francisco, San Francisco, California 94143 2Department of Neurology, University of California, San Francisco, San Francisco, California 94143 Correspondence: [email protected]
http://cshperspectives.cshlp.org/content/3/1/a006833.full.pdf+html
snip...full text ;
Wednesday, January 5, 2011
ENLARGING SPECTRUM OF PRION-LIKE DISEASES Prusiner Colby et al 2011 Prions
David W. Colby1,* and Stanley B. Prusiner1,2
http://cshperspectives.cshlp.org/content/3/1/a006833.full.pdf+html
http://betaamyloidcjd.blogspot.com/2011/01/enlarging-spectrum-of-prion-like.html
http://betaamyloidcjd.blogspot.com/
Saturday, March 19, 2011
Familial prion disease with alzheimer disease-like tau pathology and clinical phenotype
http://transmissiblespongiformencephalopathy.blogspot.com/2011/03/familial-prion-disease-with-alzheimer.html
Mike
Well-known member
Hey Hoppy, Have you noticed that when flipper gets mad he posts a million miles of unimportant BS that he thinks is getting back at us? :lol: :lol: :lol:
He's a hoot. A dumbazz hoot, but a hoot nevertheless.
He's a hoot. A dumbazz hoot, but a hoot nevertheless.
Yep and not one particle of it really makes any sense Guess that is why they call him filppring flounder the full of it fish that stinks
Guess the thinks the more he cuts and pastes the more it makes him look presentable.. all the REAL people recognize him for that he is a bigger PHONEY than anyone else on this forum,,,
A machinist that dropped out of high school. people laughingly calling him DR.
He makes a mockery of his poor departed mother. the only one who fails to see that is TERRY himself,,,,,,if he were to ever try to see what a fool he is and the pain he must cause his research he would disappear in a hear beat
just google TERRY SINGELTRY you will see how little respect he has in the real world
Guess the thinks the more he cuts and pastes the more it makes him look presentable.. all the REAL people recognize him for that he is a bigger PHONEY than anyone else on this forum,,,
A machinist that dropped out of high school. people laughingly calling him DR.
He makes a mockery of his poor departed mother. the only one who fails to see that is TERRY himself,,,,,,if he were to ever try to see what a fool he is and the pain he must cause his research he would disappear in a hear beat
just google TERRY SINGELTRY you will see how little respect he has in the real world
Mike
Well-known member
hopalong said:
Yea, and he's ugly too. :lol: :lol:
Faster horses
Well-known member
- Joined
- Feb 11, 2005
- Messages
- 30,238
- Reaction score
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Who read all that? Not me...never do, in fact. What a waste of time.
hypocritexposer
Well-known member
I read this part FH
and then I saw this homophobic bigotry in another thread. I'm pretty sure flounder was just trying to incite others......have a nice ride flounder
flounder said:
and then I saw this homophobic bigotry in another thread. I'm pretty sure flounder was just trying to incite others......have a nice ride flounder
flounder said:
Mike
Well-known member
hypocritexposer said:
Little things like spelling simple words, not to be confused with typo errors, are very telling in his vocabulary and about his education, or lack of.
By the way what's a "looser"? :lol: :lol: :lol: :lol: :lol: :lol: :lol:
About 90% of the people who respond to his garbage on other internet communications think he's an idiot also and he doesn't even realize it. :roll:
Personally, I think he's had too many of those supplements that he thinks killed his mother.
