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>DATA Charmaine's HD:BSE - AUGUST 95-fill in bse position paper spec<


FROM THE DIRECTOR GENERAL

 

24 August, 1995

 

STRICTLY PRIVATE AND CONFIDENTIAL

 

 

Dear •salutation


UKASTA POLICY ON BSE

 

At the President's suggestion in the light of recent events, I have

reviewed the history of our policy on BSE so as to ensure that it fully

reflects the needs of our supporters In the feed Industry.

 

The paper enclosed with this letter is the result. For obvious reasons,

this is being circulated only to an extremely small circle within

UKASTA - basically, the National Executive Council.

 

If you have any comments on the policy, or the paper, I should be glad

to receive them under Private & Confidential cover.

 

Yours sincerely,

 

J.W. REED

 

JWR/cg


copied to SMT members - IJD; JN; JAS; REW

 

 

95/8.24/2.1

 

 

STRICTLY PRIVATE AND CONFIDENTIAL

 

UKASTA INTERNAL POSITION STATEMENT

BOVINE SPONGIFORM ENCEPHALOPATHY

 

POLICY AIMS

 

1.   These have been consistent, although unstated except In FEC discussions, since at least 1989:-

 

• To minimise the risk of farmers' claims for compensation from feed

compounders.

 

• To minimise the potential damage to compound feed markets

through adverse publicity.

 

• To maximise freedom of action for feed compounders. notably by

maintaining the availability of meat and bone meal as a raw

material in animal feeds, and ensuring time is available to make

any changes which may be required.

 

 

STRATEGY ADOPTED/SUCCESS ACHIEVED

 

2.   Strategy has depended upon the situation at a particular time.

UKASTA has sought to anticipate criticism from other industry sectors

and action by Government/Brussels as the epidemic has developed

and knowledge of the disease increased. Through dose liaison with

MAFF. we have to date avoided public statements seriously damaging

to the feed Industry and the adoption of policies likely to lead to such

damage.

 

3.    Successful examples of this strategy include:

 

• "Voluntary Ban" on SBO's In all MBM purchase contracts from

November 1989. matching the Government ban on SBO'S in

human food but anticipating the statutory ban on SBO's in feed

which came in only from September 1990;

 

• Pressing Government for full compensation to farmers, which was

finally conceded in February 1990;

 

• evidence (not Just on BSE) to the Lamming Committee in 1991/92

resulted in their recommending tighter controls over home

mixers/integrated operations, and over the processing of fallen

animals. Government eventually tightened the fallen animals

legislation in December 1992. Other Lamming recommendations

could yet be useful to us.

 

continued.....

 

95/8.24/2.2

 

2

 

• UKASTA pressure dissuaded MAFF from publicly linking voluntary

ELISA tests of feed on farms with BAB's to (possibly compulsory)

tests at compounders' premises in June/July 1994:

 

• in August 1995. while tightening the SBO Order and responding

to the EU Decision requiring introduction of a testing programme.

MAFF has accepted UKASTA proposals for the presentation of the

changes to a wider audience, including farmers, and accepted our

help in preparing for an EU Commission visit to inspect

procedures and controls.

 

 

THE FUTURE

 

4. BSE has for more than seven years posed the greatest single potential

threat to feed compounders' profitability. Although the epidemic is in

sharp decline (275 cases per week compared to 1000 at the peak).

MAFF remains under pressure in Brussels and is not skilled at

handling potentially explosive issues.

 

5. Tests may show that ruminant feeds have been sold which contain

illegal traces of ruminant protein.  More likely, a few positive test

results will turn up but proof that a particular feed mill knowingly

supplied it to a particular farm will be difficult if not impossible.

 

6. The threat remains real and it will be some years before feed

compounders are free of it.   The longer we can avoid any direct

linkage between feed milling practices and actual BSE cases, the more

likely it is that serious damage can be avoided. In issue management

terms, the aims and the strategy remain valid, but must be kept

under review in the light of further events.

 

JWR/cg/23.8.95

 

95/8.24/2.3

 



http://www.bseinquiry.gov.uk/files/yb/1995/08/24002001.pdf




From: TSS ()

Subject: THE MANIPULATION OF BSE/TSE SCIENCE IN PEER REVIEW JOURNALS ?

Date: July 11, 2005 at 11:23 am PST



1983



BSE CONSULTANT


APPROVAL OF MATERIAL FOR PUBLICATIONS


All material for publication including written works to be published in scientific journals, books, proceedings of scientific meetings, abstracts of verbally delivered papers and the like should be scrutinized for risk to the Ministry before dispatch to the publishers.............


full text;


http://www.bseinquiry.gov.uk/files/yb/1983/10/12001001.pdf



The BSE Inquiry / Statement No 67

Dr Iain McGill (scheduled to give oral evidence 08 June 98)

THE BSE INQUIRY

Statement by I S McGill

Curriculum Vitae

1. A brief CV is enclosed with this statement as Annex 1.

Periods particularly concerned with TSEs are as follows:

Spring/Summer 1988 Final Year Elective Research project

The astrocytic reaction in BSE and its comparison

with natural scrapie of sheep.

Royal Veterinary College (RVC), London University.

Performed at Pathology Dept, MAFF, CVL, Weybridge

Jan 1990 - Jun 1991 Veterinary Research Officer

Central Veterinary Laboratory (CVL), Weybridge.

Employer: MAFF

Funding Body: MAFF

Aug 1991 - Dec 1992 Post Doctoral Research Worker

Institute of Psychiatry, London University.

Employer: Institute of Psychiatry

Funding Body: AFRC

1994 - 1998 Director, Prion Interest Group

Independent Research Organisation.

Privately financed.

Incorporated as scientific wing of

Shift Ltd in 1995 : Company reg no 291 6731

Statement of Interests

2. I have no links of any nature with the farming community (other than

those from my work

with farms as a vet student or as a qualified vet) nor with the feedstock,

pet food or

rendering industries.

Advice to Governmental Committees

3. I have had no involvement in UK Governmental committees.I did, however,

act as

rapporteur for a conference of international experts held in 1990 at the

CVL. This was

held under the auspices of the Gibbs Committee, organised by Dr CJ Gibbs of

the National

2

Institutes of Health, Bethesda, USA. My draft report from this conference

was passed on

to MAFF. (The final report is at YB90/3.12/1.1)

How I became involved in work on TSEs.

4. In 1987/88, my final year at the RVC, the work of Gerald Wells and his

colleagues at the

CVL on BSE was the subject of much discussion. I approached Richard Barlow

Professor of Pathology at RVC, who had decades of experience in scrapie

research, with a

view to carrying out my final year elective research project outside the

RVC, with the

team at the CVL. He agreed to co-supervise a project with Gerald Wells.

5. The report of this work is available and also held in RVC library. The

external examiner

for this research was Dr W B Martin, at that time sitting on the Southwood

Committee.

This research achieved first place in the year in professional examinations

and contributed

to the award of the Cecil Aulden Second Prize (second place throughout

BVetMed

degree).

6. Once qualified, I worked for 18 months in practice, and then returned to

the Pathology

Department of the CVL in January 1990, to continue my work with Mr Wells.

Commissioning and Funding

Work at CVL, Weybridge, commissioned by MAFF

7. At CVL most of the projects I worked on were commissioned and directly

funded by

MAFF. All the research I pursued was as an employee of MAFF, excepting work

during

my final year at the RVC, when I was supported by a student grant.

8. I think it is worthwhile making a few salient points about my day to day

work for MAFF.

The facilities of the Pathology Department and the quality of technical and

administrative

support were in general excellent and the scientists I worked with were of a

uniformly

high calibre. Once the decision to fund a project had been reached, there

were no

restraints on rapidly seeking tangible results.

9. Publications arising from such work include references 2, 5, 7, 8 & 10 in

the list of

publications to be found at the end of this statement.

Uncommissioned Work at CVL, Weybridge

10. As I was not always encouraged to pursue work I regarded as essential, I

also worked on

projects (either practical or theoretical) beyond the narrow confines of

specific MAFF

commission. Often this was possible within my daily routine, but I would

periodically

have to 'clock off' from my MAFF job and work in my own time rather than

leave the

matter untouched.

11. Publications arising from non-commissioned work started whilst I was

still an employee of

MAFF include :

3

(i) Wood LJN, McGill IS, Done SH and Bradley R (1997). Neuropathology of

scrapie: a study of the distribution patterns of brain lesions in 222 cases

of natural

scrapie in sheep, 1982-1991; ref 12.

The project as a whole was commissioned by MAFF, but my own involvement was

voluntary (see para. 31).

(ii) Heretical Model of Scrapie (1991) paper to the annual conference of the

Association of veterinary Teachers and Research Workers, Scarborough 1991,

referred to in the Veterinary Record, 128, pp368-369.

See paras 52 et seq for details of research proposed to MAFF at this time.

(iii) Stack MJ, Aldrich AM, Davis LA (1997). Comparison of detergent and

protease

enzyme combinations for the detection of scrapie-associated fibrils from the

central

nervous system of sheep naturally affected with scrapie. Journal of

Comparative

Pathology, 1997, 116, pp.181-189 (J/CP/116/181).

12. Although this is not acknowledged in the published article, I introduced

Mick Stack to the

use of Subtilisin Carlsberg enzyme for SAF extraction, initially from

formalin-fixed

tissue. Following a theoretical discussion with Dr (now Professor) Ian Shaw

in 1991 I had

pioneered such use of this enzyme. My conceptual role (not commissioned by

MAFF) in

this work illustrates the sometimes unexpected fruits of the free pursuit of

scientific (as

opposed to Government) ideas.

Work at the Institute of Psychiatry funded by an AFRC Grant

13. Publications arising from this work include references 3, 4, 6 & 9 in

the attached list of

publications.

14. Two publications submitted to the Veterinary Record during this period

went unpublished.

(See para 22).

Constraints on publication of results

" Internal approval"

15. Every paper generated within MAFF is sent for approval by superiors; the

more serious

the topic (for example if the disease is zoonotic or notifiable), the higher

it is sent before

approval is given or refused. I only outline the system in operation within

MAFF at that

time, and it is not my intention to criticise individuals who were

performing their ascribed

roles within the structure of MAFF.

16. I would like to illustrate the process of 'approval' with reference to

the identification of

FSE and subsequent publication of findings. The first case of FSE was

discovered at

Bristol University by Janet Wyatt (now Bradshaw) working with Dr Geoff

Pearson and

others. Material was referred to Gerald Wells in April 1990 for his expert

opinion and he

passed histological sections to me for comment.

4

17. Over the next six months, the Bristol and Liverpool Veterinary Schools

(and possibly

others) and the CVL independently examined their archives of feline brains

to determine

whether this disease existed prior to the BSE epidemic, or whether it was a

new disease.

Neuropathological evidence suggested it was a new disease. This, along with

epidemiological and biochemical data, led Gerald Wells and myself to prepare

a paper for

an international TSE conference in Brussels including the indication that

there might be a

causal link between BSE and FSE. The abstract of this paper was faxed to

Brussels prior

to the conference for publication in a booklet for delegates. This abstract

(ref 5) includes

the suggestion of this link.

18. Following the conference, we prepared the full paper we had presented,

for publication in

a book of this European Commission-sponsored conference and sent it to our

superiors

for approval. (The correspondence which ensued in April and May 1991 is

found at

YB91/4.16/1.1;YB91/4.22/2.1;YB91/4.30/1.1;YB91/5.3/3.1). Specifically,

despite detailed

arguments supporting our statements, the following ultimatum was faxed to us

from the

then Assistant Chief Veterinary Officer, making it plain that he was taking

into account the

views of the then CVO:

"We are not willing for the paper to be published unless these references

are

removed. This may be unacceptable to the authors, in which case permission

to

publish is refused."

19. Despite protestations that the body of the text would no longer agree

with the already

published abstract, and our detailed knowledge on the subject

notwithstanding, the edict

stood. We were left with little alternative but to amend the paper, which by

this time had

missed the original deadline for submission and was in danger of not being

published at all

(see correspondence above).

20. Subsequent to its publication in the conference book (ref 5), the paper

was also published

in a refereed journal (ref 7). The original abstract from ref 5 was then

also altered to agree

with the altered text. Specifically the words "with BSE" were removed from

the phrase

"epidemiological association with BSE". I had left MAFF before this paper

was ever

published.

21. This episode was described in Dispatches (Channel 4, 9pm Thursday 11th

December

1997), and on two separate occasions in. The Independent newspaper

(YB97/12.11/1.1

and YB98/1.26/1.1).

Outright rejection of manuscripts submitted for publication, during

"Refereeing/Scrutineering" by Journals

22. The peer review system is in itself generally reasonable. However, an

issue of real concern

is that the Veterinary Record , the main channel of information for the

veterinary

profession, failed to provide an open forum for discussion of the TSEs

throughout the

period of the terms of reference of the Inquiry.

23. The following is a chronology of papers submitted to the Veterinary

Record, but which

went unpublished:

5

1988: Letter entitled 'Scrapie, Time to take HB Parry Seriously'

(YB88/6.8/4.1)

24. In this letter I stated that BSE had been officially confirmed as a TSE

(when much of the

veterinary profession still favoured a variety of alternate hypotheses). I

also suggested

that scrapie should be made a notifiable disease, and drew attention to the

work of HB

'James' Parry and the possibility that natural scrapie in sheep might be of

genetic origin.

25. I withdrew the letter following advice from Professor Barlow (who as far

as I can recall

had been contacted by MAFF and the Veterinary Record) that it might not be

in my

interests to pursue publication at that moment in time.

26. I received a letter from the then editor, Edward Boden, questioning my

permission to

release the information that BSE was indeed a proven TSE. I had no

permission, though

was unaware that any was needed, to inform my profession of this urgent and

important

fact.

1992: McGill and Wood

27. This paper summarises views as to why an open debate on TSEs and in

particular scrapie

were and remain essential. We drew attention to the work of Parry, Prusiner

and others,

and outlined novel explanations for recent research findings in light of

such work. We

suggested that not all the relevant questions were being asked in the

interpretation of data.

In particular, the possibility that the infectious agent was being generated

de novo from the

genome (the PrP gene) in certain families of sheep, was still not being

considered, despite

a body of scientific data going back over 30 years. It was to be a further 5

years before

publications from Government laboratories would start to cite Parry's work

as a possibly

correct theory.

28. The refereeing process for this work was at the time not transparent,

and I have yet to be

informed as to why this remains unpublished.

29. 1992 Book review commissioned - on "Sub-Acute Spongiform

Encephalopathies" Eds.

Bradley, Savey & Marchant, Kluwer Academic Publishers, Dorchelt, for the

Commission

of the European Communities.

30. On 13th May 1992, I was commissioned by the Veterinary Record to review

this book

(YB92/5.13/1.1). After approximately 100 hours work for this review, an

editorial

decision was taken not to publish. Ironically, this book contained the very

paper by

Gerald Wells and myself over which "censorship" has been alleged.

Prolonged delay during "Refereeing/Scrutineering" of manuscripts submitted

for

publication

1997 Wood McGill Done and Bradley (ref 12).

31. This work was started in 1990 to screen for putative BSE in sheep, by

James Wood, a

colleague in the Pathology Department at CVL, although it was not finally

published until

1997. James sought my assistance in light of my greater experience in TSE

pathology. I

worked many many months to get this paper into print (YB95/6.29/2.1;

YB96/9.19/2.1)

6

refereeing process took two years, hardly an acceptable delay for crucial

work in this

field. The referees' comments (YB95/6.29/2.2) themselves require scrutiny.

One

scrutineer seemed to referee the paper in a balanced way, whilst the other

seemed more

intent on pushing his/her own opinions onto the paper.

32. Publication was finally expedited in the summer of 1996, when the

politically sensitive

question of whether or not BSE had indeed gone into the sheep population

started to be

asked in the public domain. This paper finally appeared (with some important

omissions

and watering down) in 1997, seven years after it was started, and two years

after it was

submitted. It was jointly funded by MAFF and the Prion Interest Group.

33. Had my ongoing research into sheep scrapie been funded and/or the McGill

and Wood

1992 paper been published, stimulating debate and further investigations,

this paper would

most certainly have appeared by 1995. Further work based on it could have

determined

by 1997 whether or not, and if so to what extent, BSE had gone into sheep.

34. In addition, the work may by now have led to a rapid diagnostic test and

a great deal of

information on the actual (as opposed to the theoretical/experimental)

causes of sheep

scrapie and the fundamental biology of this entire group of diseases. Some

of the work

suggested in 1991 has still not been started.

Aspects of TSE work with which I was involved

Analysis of the astrocytic response in BSE and its comparison with natural

scrapie.

35. I worked as a neuropathologist with Gerald Wells to establish that

astrocytic reaction, one

of the fundamental triad of neuropathological changes occurring in TSEs, was

indeed

present in BSE. This work was accomplished using antibodies to GFAP (a

structural

component characteristic of astrocytes) to quantify previously qualitative

interpretations

that an astrocytic reaction was present. The astrocytic reaction in natural

sheep scrapie

was assessed in parallel.

Published: 1988 RVC library.

1991 (Wells, Wilesmith and McGill) - details of astrocytic response in BSE

1997 (Wood, McGill, Done and Bradley) - details of astrocytic response in

natural sheep

scrapie

Surveillance for emerging scrapie-like diseases in animals in the UK

36. Working with Gerald Wells and other pathologists from the State

Veterinary Service, I

was involved with surveillance for neurological disease of animals in the

UK. This was

with particular reference to surveillance for, and subsequent confirmation

of TSEs.

During my time of employment, novel TSEs arose in domestic cats and in

exotic ungulates

in zoological collections. I also became involved in the investigation of a

putative TSE in

hound packs detected by Robert Higgins.

FSE, and BSE in exotic ungulates published in reviews:

1991 (Wells and McGill) ref 5

7

1992 (Wells and McGill) ref 7

FSE discussed in para 15.

37. Putative TSE in hounds - work started 1990 –(see para 41)

Robert Higgins, a Veterinary Investigation Officer at Thirsk, had been

working on a

hound survey in 1990. Gerald Wells and I myself received histological

sections from this

survey along with the accompanying letter (YB90/11.28/1.1) dated November

1990. This

letter details spongiform changes found in brains from hunt hounds failing

to keep up with

the rest of the pack, along with the results of SAF extractions from fresh

brain material

from these same animals. SAFs were not found in brains unless spongiform

changes were

also present. The spongiform changes were not pathognomonic (ie. conclusive

proof) for

prion disease, as they were atypical, being largely present in white matter

rather than grey

matter in the brain and spinal cord. However, Tony Scott, then head of

electron

microscopy work on TSEs, had no doubt that these SAFs were genuine and that

these

hounds therefore must have had a scrapie-like disease. I reviewed all the

sections myself

(original notes appended) and although the pathology was not typical, I

could not exclude

the possibility that this was a scrapie-like disorder, as white matter

vacuolation is seen in

TSEs and Wallerian degeneration was also present in the white matter of the

hounds,

another feature of scrapie.

38. I reviewed the literature on hound neuropathology, and discovered that

micrographs and

descriptive neuropathology from papers on 'hound ataxia' mirrored those in

material from

Robert Higgins' hound survey. Dr Tony Palmer (Cambridge) had done much of

this

work, and I obtained original sections from hound ataxia cases from him.

This enabled me

provisionally to conclude that Robert Higgins had in all probability

detected hound ataxia,

but also that hound ataxia itself was possibly a TSE. Gerald Wells confirmed

in 'blind'

examination of single restricted microscopic fields that there was no

distinction between

the white matter vacuolation present in BSE and scrapie cases, and that

occurring in

hound ataxia and the hound survey cases.

39. Hound ataxia had reportedly been occurring since the 1930's, and a known

risk factor for

its development was the feeding to hounds of downer cows, and particularly

bovine offal.

Circumstantial evidence suggests that bovine offal may also be causal in

FSE, and TME in

mink. Despite the inconclusive nature of the neuropathology, it was clearly

evident that

this putative canine spongiform encephalopathy merited further

investigation.

40. The inconclusive results in hounds were never confirmed, nor was the

link with hound

ataxia pursued. I telephoned Robert Higgins six years after he first sent

the slides to CVL.

I was informed that despite his submitting a yearly report to the CVO

including the

suggestion that the hound work be continued, no further work had been done

since 1991.

This was surprising, to say the very least.

41. The hound work could have provided valuable evidence that a scrapie-like

agent may have

been present in cattle offal long before the BSE epidemic was recognised.

The MAFF

hound survey remains unpublished.

Histopathological support to various other published MAFF experiments

8

42. These included neuropathological examination of material from

experiments studying the

attempted transmission of BSE to chickens and pigs (CVL 1991) and to mice

(RVC

1994).

Neuropathological findings in cattle with clinically suspect but

histologically unconfirmed

bovine spongiform encephalopathy

43. This was my main project during my employment at MAFF.

44. At this time, approximately 10% of cattle suspected of having BSE were

not being

diagnosed as BSE-positive. The purpose of this work was to establish what

other diseases

were being clinically mistaken for BSE and causing these cattle to be taken

as suspects

under the BSE Order.

45. Upon closer examination, three of the 200 'BSE-negative' brains proved

positive for

spongiform changes diagnostic of BSE (see YB87/12.14/1.2; YB87/12.15/2.1).

This

represents an overall diagnostic accuracy of 99.85%, exceeding the 99.6%

previously

published for the same standard diagnostic technique. Despite this, at the

behest of MAFF

managers, the emphasis of the study and its provisional title had to be

changed, from

accurately representing the whole negative 10%, to a study examining this

10% minus any

mention whatsoever of BSE-affected cattle going undiagnosed. I therefore had

to

reluctantly locate and analyse three new BSE-negative suspect brains.

46. Discussion of this would according to MAFF officials have resulted in

'lack of clarity' and

opened up debate as to the accuracy of diagnosis.

47. Although this may seem a minor consideration, it illustrates the

kneejerk and perhaps

unnecessary culture of secrecy operating within MAFF at that time.

48. As it was also a theoretical possibility that cases of BSE might exist

without the

characteristic spongiform changes, a further purpose of this work was to

examine selected

cases using immunocytochemistry for PrP to determine if any had BSE but

lacked the

characteristic pathology. Although the sensitivity of the technique used has

increased

dramatically since then, none were found at this time, and this was one of

the important

findings of the paper which was published.

49. In a number of informal conversations at that time, managers within MAFF

let me know

that the upper echelons of MAFF "had had it up to here with you scientists

finding out

about new diseases". As a Veterinary Research Officer employed in disease

surveillance, I

had considered that to be my job.

Published 1993 (McGill and Wells) ref 10

Theoretical models of TSE diseases

50. Published in the Veterinary Record 1991 (J/VR/128/368) as editorial of

AVTRW

conference. In 1991 Martin Alder, new editor of the. Veterinary Record ,

published a very

favourable account of my theoretical paper presented at AVTRW 1991 in

Scarborough

under the heading "Heretical Model of Scrapie". The Chairman of this session

was Bill

9

Blakemore, Cambridge Vet School. It was printed (after consultation with me)

in an

editorial article "Fruits of Research On Show in Scarborough". He devoted

considerably

more column inches to work by myself and Kenton Morgan than to work reported

by

NPU, although they had presented far more papers. It was to be the last time

my name

would appear in the Veterinary Record until 1997.

Unpublished 1988, 1992

Establishing that human prion disease can exist without characteristic

pathology.

51. This was the first conclusive proof that prion diseases can indeed exist

without any of the

characteristic pathology, extending the phenotypic diversity of prion

disease.

Published 1992 (Lantos, McGill et al) ref. 6

Setting up in vitro models of human prion diseases (GSS, familial CJD) in

neuroblastoma

cells in culture

(Resigned half way through project)

Neuropathology of natural sheep scrapie.

Started 1990, submitted 1995, published 1997 (Wood, McGill et al) ref 12

See para 11 for details of this work.

Contact with / Advice to Government

Contact with CVL / MAFF

52. I maintained regular contact with scientists at the CVL until 1997.

53. I cannot catalogue all the information, advice or recommendations I

offered to MAFF or

CVL between 1988 and 1997, as there is too much to include. However, I could

illustrate

with the following summary of two substantive suggestions for research.

Research on the biochemical/physical nature of "strains"

54. Prior to their publication as an editorial in the Veterinary Record, a

summary of these

ideas was presented to the CVL management for funding as a 'blue sky' PhD

project in

Spring 1991. The proposal was not taken up. This was the first occasion on

which I

proposed research to the Government in writing.

An abattoir survey for incidence of BSE

55. I suggested in 1990 that to improve the provision of control material I

should collect 20

cattle heads from a local abattoir.The purpose of this was to provide

BSE-negative

material to act as controls for our (CVL's) BSE work. However,

neuropathological

examination of these brains might also have given an indication of the

number of cattle

incubating BSE which were entering the human food chain. This research had

actually

been recommended in the Interim Report of the Tyrrell Committee, June 89.

10

56. I was instructed a few days after suggesting this to my head of

department that I was not

the first person to have thought of that, and that a decision had been taken

not to do that

research. I was also instructed, for some reason, not to put it in writing.

57. Budgets could hardly have been an issue contributing to the rejection of

this proposal, as

tongueless cattle heads were free, being banned from human consumption.

Contact with AFRC

58. I had contact with the AFRC in several capacities:

My work at the Institute of Psychiatry was funded by an AFRC grant.

Attending BSE Programme conferences in 1992 and 1994.

Submitted a further grant application to the AFRC in 1991.

59. This proposal was to continue research on natural scrapie, with which I

had been involved

at CVL (eventually published 1997; ref 12). I was to collaborate with John

Powell

(molecular neurobiologist) and David Male (co-author of the standard

Immunology text

worldwide: Roitt, Brostoff and Male). All five referees gave positive

statements about the

proposal, which was alpha-rated (see YB92/12.10/1.1 and YB92/12.17/1.1).

60. This was the second time that I suggested substantial investigations on

the TSEs to the

government in writing. Once more the proposal was not taken up.

61. In view of the continuing uncertainties as to the degree to which BSE

has affected the

sheep population, it would perhaps have been wise to fund this application

at that time.

62. Some of this work has still not been initiated, although the paper

(Wood, McGill et al

1997), after a two year delay from submission to publication, and the

original 1992 AFRC

grant submission, both described a unique series of characterised sheep

brains affected

with naturally occuring TSEs. The majority of them are natural scrapie

although further

work on this series of brains would give an indication of whether BSE was

also occurring

in sheep in the 1980s and early 1990s. Events have moved forward since this

grant

application was submitted, both in the nvCJD and scrapie fields, but this

still represents a

crucial question in the epidemiology of both scrapie and BSE which remains

unanswered.

This work should, in my opinion, be initiated forthwith, and further work

based on these

results pursued vigorously as results are obtained.Refer to discussion also

at para 31.

Additional Comments

63. I could perhaps sum up MAFF's approach to BSE with an observation which

is by no

means original:

"Absence of evidence" is not the same as "evidence of absence"

Publications

1. McGill IS (1986) The Shortcut to Elitism. The Guardian, December 1st,

p12.

11

2. Wells GAH, Wilesmith, JW & McGill IS (1991) Bovine spongiform

encephalopathy - a neuropathological perspective. Brain Pathology, 1, 69-78

3. McGill IS (1991) Bovine Spongiform Encephalopathy. In: Practical Food

Hygiene,

Ed. Dickens T, Croner Publications Ltd, Kingston, UK, pp. 435-436

4. McGill IS & Whatley SA (1991) Understanding the causes of brain disease.

The

Independent, August 16th, p. 20

5. Wells GAH & McGill IS (1991) Recently described scrapie-like

encephalopathies of

animals - case definitions. In: Sub-acute Spongiform Encephalopathies, Eds.

Bradley

R, Savey M & Marchant B, Kluwer Academic Publishers, Dorchelt, pp. 11-24.

6. Lantos P, McGill IS, Janota I, Doey J, Collinge J, Bruce M, Whatley SA,

Anderton BH, Clinton J, Roberts GW & Rosser N (1992) Prion protein

immunocytochemistry helps to establish the true incidence of prion disease.

Neuroscience Letters, 147, 67-71

7. Wells GAH & McGill IS (1992) Recently described scrapie-like

encephalopathies of

animals - case definitions. Research in Veterinary Science, 53, 1-10

8. Pollin MM, McGill IS & Wells GAH (1992) The differential

neurohistological

diagnoses of clinically suspect but unconfirmed BSE. Neuropathology and

Applied

Neurobiology, 18, 633 (abstract)

9. Guha M & McGill IS (1992) Book review of Black's Veterinary Dictionary

(17th

Edition), Ed, West GP, A & C Black, London. Reference Reviews, 6, 26

10. McGill IS & Wells GAH (1993). Neuropathological findings in cattle with

clinically

suspect but histologically unconfirmed bovine spongiform encephalopathy

(BSE).

Journal of Comparative Pathology, 108, 241-260

11. McGill IS (1995) Ayurvedic Medicine - The Documentary. Natural Medicine

Society

News, Spring 1995

12. Wood LJN, McGill IS, Done SH and Bradley R (1997) Neuropathology of

scrapie:

a study of the distribution patterns of brain lesions in 222 cases of

natural scrapie in

sheep, 1982-1991. Veterinary Record 140, 167-174

13. McGill IS, Hobson J (1998) Multi-centre evaluation of a herbal skin gel

for veterinary

practice - a questionnaire survey. Veterinary Times, 28, 1, 20-21

14. McGill IS (1998) BSE and Censorship. The Independent January 26th 1998,

p14

(YB98/1.26/1.1).

12

ANNEX 1:

CURRICULUM VITAE: IAIN STEWART McGILL

Education & Qualifications

1975 - 1982 Southend High School for Boys

GCE O-Levels (1980): 9 (6 A, 3 B)

GCE A-Levels (1982): Biology (A), Physics (A), Chemistry (A)

GCE S-Level (1982): Biology (2)

1982 - 1984 Royal Veterinary College, University of London

1984 - 1985 Kings College, University of London

B.Sc(Hons), II(i) Neuroscience and Immunology

1985 - 1988 Royal Veterinary College, University of London

B.Vet.Med., MRCVS.

Distinctions : Medicine , Clinical Pathology (Elective Subject)

First place for research project

Cecil Aulden Second Prize

Professional Experience

1988 - 1989 Veterinary Surgeon -- Blue Cross Animal Hospital, Victoria,

London

In addition to clinical duties, I upgraded clinical pathology services

within the hospital and established an interpretive service for laboratory

data for other clinicians.

1990 - 1991 Veterinary Research Officer -- MAFF Central Veterinary

Laboratory,

Weybridge, Surrey.

In this post I worked as a neuropathologist with Gerald Wells and William

Hadlow, in a large

interdisciplinary team researching the prion diseases of animals.

My work concentrated on the neuropathological characterisation of Bovine

Spongiform

Encephalopathy (BSE), the prion diseases of other animals and their

differential diagnosis.

This gave me good general experience of neurological disease, its diagnosis

and pathological

characteristics in a wide range of animals and an introduction to many

fields of neuroscience

research. Although broad-based, my research in these varying disciplines was

centred on the

Prion protein and its gene, and associated molecular pathology in the prion

diseases.

I described, amongst other things, the first cases in the UK of a chlamydial

disease of cattle

putatively equivalent to Sporadic Bovine Encephalomyelitis (see McGill and

Wells, 1993).

13

Additional responsibilities included:

Liaison with the Consultant Pathology Unit for neuropathological

surveillance, including

rabies diagnosis for the British Isles and characterisation of novel

diseases such as blue eared

pig disease.

Conducting occasional seminars introducing scrapie and BSE diagnosis for

visiting scientists

from abroad.

Rapporteur for The Gibbs Committee on Subacute Spongiform Encephalopathies

(held at

CVL in summer 1990).

Papers presented at AVTRW conferences at Scarborough (1990 & 1991), at

European

Community Seminar on Spongiform Encephalopathies, Brussels, (1991) and

International Pig

Veterinary Society, Holland, (1991).

Aug 91 - Dec 92 Research Worker (post-doctoral level)

Department of Neuroscience, Institute of Psychiatry, London

In this post I continued to follow my interest in the prion diseases, and

gained a good

grounding in both theoretical and practical molecular biology. I cloned PrP

genes from blood

samples taken from individuals with PrP mutations causal of familial CJD or

GSS and

transfected them into neuroblastoma cells in culture to investigate the

disease process in vitro.

I continued to work with colleagues from other disciplines, particularly

Neurology and

Neuropathology, and with Professor Peter Lantos and others established for

the first time that

prion disease can exist without its characteristic pathology (Lantos, McGill

et al, 1992).

Positions of responsibility included:

Lecturing on a course entitled "Molecular Mechanisms of Neurodegeneration",

to both

internal and external scientists, and as part of the London University M.Sc.

Neuroscience

course.

Lecturing on scrapie-like diseases as part of the London University M.Sc. in

Animal Health at

the RVC.

Liaison and research collaboration between the IOP and my previous employers

at CVL,

Weybridge.

Sole responsibility for the organisation and funding of the 1992/1993

seminar series for the

Department of Neuroscience, in which leading researchers from around the UK

were invited

to give seminars.

Paper presented at AFRC BSEP meeting, Reading, April 1992.

1994 - Present Scientific and Veterinary Consultant

14

(Spring 94) Acted as neuropathological consultant for research on the

transmissibility of BSE in collaboration with Dr David White and

Professor Neil Eddington at the Royal Veterinary College, University

of London.

(Summer 94) Veterinary Surgeon, Blue Cross Animal Hospital (Victoria,

London)

(Oct 94 - Dec 94) Lecturer in anatomy and histology, Optics Department, City

and

Islington College (London).

(1995) Re-established the Prion Interest Group (originally founded at the

Institute of Psychiatry in 1991) as a private organisation, continuing

research on prions.

Filmed and directed a documentary in India/Europe about Ayurvedic

medicine.

Acted as a locum veterinarian for the PDSA and the Veterinary Centre

Caterham.

(Jan 96 - Present) Veterinary Consultant to Ayuvet (UK) Ltd., co-ordinating

clinical and

laboratory research into the Ayurvedic system of medicine and its

application in European veterinary medicine. Continued co-ordination

of the Prion Interest Group.

Issued on behalf of the witness by:

The BSE Inquiry Press Office

6th Floor Hercules House

Hercules Road

London SE1 7DU

Tel: 0171 261 8377 / 8383

Fax: 0171 803 0893

Website: http://www.bse.org.uk

email: inquiry@bse.org.uk


http://www.bseinquiry.gov.uk/files/ws/s067.pdf


TSS


----- Original Message -----

From: "Terry S. Singeltary Sr." <flounder9@VERIZON.NET>

To: <BSE-L@aegee.org>

Sent: Monday, July 11, 2005 1:31 PM

Subject: THE MANIPULATION OF BSE/TSE SCIENCE IN PEER REVIEW JOURNALS ?



#####################  Bovine Spongiform Encephalopathy

#####################


From: TSS ()

Subject: THE MANIPULATION OF BSE/TSE SCIENCE IN PEER REVIEW JOURNALS ?

Date: July 11, 2005 at 11:23 am PST



1983



BSE CONSULTANT


APPROVAL OF MATERIAL FOR PUBLICATIONS


All material for publication including written works to be published in

scientific journals, books, proceedings of scientific meetings, abstracts of

verbally delivered papers and the like should be scrutinized for risk to the

Ministry before dispatch to the publishers.............


full text;


http://www.bseinquiry.gov.uk/files/yb/1983/10/12001001.pdf



- 10 -


19. On 18th February, 1987 (YB87/2.18/1.1) I reported to Dr Watson and Dr

Shreeve on a further


case which we had received from Truro VIC. The brain had shown neuronal

vacuolation and


in brain extracts there were fibrils that were similar in size and

appearance to SAFs from sheep


with scrapie. The Virology Department was studying the brain further and

considering a


transmission study. A few weeks before this, I had discussed the possibility

of a transmission


study with Michael Dawson, a research officer in the Virology Department and

an expert in


viral diseases in sheep, and we were considering carefully the safety

aspects. In my note I


raised the question of whether we should disclose the information we had

more widely to the


VIS because this may assist in getting any other cases referred to CVL but

there was the


difficulty that we knew very little about the disorder and would be unable

to deal with queries


that might be raised.


20. On 23rd February, 1987 (YB87/2.23/1.1) I sent Mr Wells a note asking him

to prepare a


statement for publication in Vision, the in-house newsheet prepared by the

VIS for the SVS,


setting out details of what we had discovered. On 24th February, 1987

(YB87/2.25/2.1) Gerald


Wells indicated in a note to me that he had discussed the proposed article

with Mr Dawson and


they both believed that it could be damaging to publish anything at that

stage. They believed


cases would be referred to CVL in any event because they were unusual and

they did not feel


"Vision" was an appropriate publication because its confidentiality was

questionable and might


lead to referrals to veterinary schools rather than CVL. Gerald Wells was

also concerned


about the resources available in his section to deal with referred cases. I

replied


(YB87/2.25/2.1) indicating a draft statement was needed by the Director

before a decision on


publication could be made. Gerald Wells prepared a draft statement

(YB87/3.2/2.1) and sent it


to me on 2nd March, 1987. In his cover note (YB87/3.2/1.1) he commented that

he believed


the distribution of any statement about the new disease outside of CVL to be

premature


because there was so little information available about the new disease. I

passed on a copy of


Gerald Wells' note to Dr Watson (YB87/3.2/3.1). I discussed the matter of

publication with Dr


Watson. No decision had been taken to publish any material at that stage and

I sent a note to


Gerald Wells letting him know the position and confirming that his views and

those of Michael


Dawson would be taken into account when a decision was taken.


- 11 -


21. In March, 1987 serious consideration was given to possible transmission

(e.g. to hamsters) and


other experiments (other than the collection of epidemiological data by the

VIS and


clinicopathology which had been in progress since the first cases were

recognised in November,


1986).


22. On 23rd April, 1987 I sent a report (YB87/4.23/1.1) to Dr Watson and Dr

Shreeve informing


them that nine control brains were being examined for SAFs and a cow which

appeared to be


affected with BSE had been purchased for observation. The cow had come from

the farm


where the original cases had developed and had arrived at CVL on 22nd April,

1987.


23. On 15th May, 1987 Dr Watson informed me that the proposed "Vision" draft

would be


circulated to VICs in England and Wales if it was approved by management. On

22nd May,


1987 I was copied in on a note (YB87/5.22/2.1) from B.M Williams, (who I

believe was Head


of the VIS at this time but retired shortly after this), to Dr Watson. This

confirmed that the


draft prepared for publication in Vision was approved but that the final

paragraph should be


amended to make it clear that knowledge of the new disease should not be

communicated to


other research institutes or university departments. At a meeting with Dr

Watson on 2nd June,


1987 he informed me that no communication should be made with NPU until

after the meeting


with the CVO on 5th June, 1987 (see my note of 3rd June, 1987 –

YB87/6.3/1.1). We needed


much more data and information to answer inevitable queries. ...



http://www.bseinquiry.gov.uk/files/ws/s071.pdf


http://www.publications.parliament.uk/pa/cm199900/cmselect/cmsctech/465/465m

48.htm



The Cultural Politics of Science and Decision-Making


An Anglo-German Comparison of Risk Political Cultures


 The BSE Case





by




Kerstin Dressel


sine-Institute Munich, Germany


kerstin.dressel@sine-institut.de.




The following report include excerpts of a thesis submitted in fulfilment of

the requirements for the degree of


Doctor rerum politicarum (Dr. rer. pol.)


at the Ludwig-Maximilians-University of Munich


supervisor: Prof Dr Ulrich Beck


Institute for Sociology, Munich, Germany




The British case study was prepared at the Centre for the Study of

Environmental Change at Lancaster University, UK,


Supervisor: Prof Dr Brian Wynne


kindly supported by a grant of the Economic and Social Research Council, UK




Munich, 2nd October 2000



© Kerstin Dressel, 2000  all rights reserved.



http://bse.airtime.co.uk/dressel.htm#9




suppressed peer review of Harvard study October 31, 2002


http://www.fsis.usda.gov/oa/topics/BSE_Peer_Review.pdf




----- Original Message -----

From: "Galyean, Michael"

To:

Cc:

Sent: Saturday, May 28, 2005 3:34 PM

Subject: FW: Prion biology relevant to bovine spongiform encephalopathy

(ANIMALSCI Feedback Form)



Dr. Novakofski:


I recevied the following message and comments regarding your recent paper on

prion biology published in the Journal of Animal Science. I hope you will

take the time to look over the comments and respond to Mr. Singeltary.


Regards,


Michael Galyean

Editor-in-Chief

Journal of Animal Science


________________________________


From: Terry S. Singeltary Sr. [mailto:flounder9@verizon.net]

Sent: Thu 5/26/2005 9:20 AM

To: Amanda Kolling

Cc: Galyean, Michael

Subject: Re: Prion biology relevant to bovine spongiform encephalopathy

(ANIMALSCI Feedback Form)



I have forwarded this to Dr. Michael Gaylean as suggested.

thank you........


please note new email address flounder9@verizon.net


any questions or follow ups, please do not hesitate to write...


thank you,


kindest regards,

Terry S. Singeltary Sr.



----- Original Message -----

From: Amanda Kolling

To: Terry S. Singeltary Sr.

Sent: Thursday, May 26, 2005 8:11 AM

Subject: RE: Prion biology relevant to bovine spongiform encephalopathy

(ANIMALSCI Feedback Form)



Dear Mr. Singeltary,


Thank you for your comments. Contrary to a previous e-mail sent to you by an

employee of HighWire, the Journal of Animal Science does accept Letters to

the Editor. If you are interested in submitting this as a letter to the

editor, I urge you to contact our editor-in-chief, Dr. Michael Gaylean at

michael.galyean@ttu.edu..


Best regards,


Amanda Kolling

Technical Editor,

Journal of Animal Science



At 09:11 AM 5/17/2005 -0700, Terry S. Singeltary Sr. wrote:

>------------------------------------------------------------

>Comments sent via JAS Feedback Page

>------------------------------------------------------------

> NAME: Terry S. Singeltary Sr.

> EMAIL: flounder@wt.net

> IP ADDRESS: 216.119.139.23

> HOSTNAME: pool139-23.dial-p1.hou.wt.net

> PREVIOUS PAGE: http://jas.fass.org/cgi/content/abstract/83/6/1455

> BROWSER: Mozilla/5.0 (Windows; U; Win98; en-US; rv:1.0.2)

> Gecko/20030208 Netscape/7.02

> PROMOTIONAL USE: Granted

>------------------------------------------------------------

>COMMENTS:

>J. Anim. Sci. 2005. 83:1455-1476

>© 2005 American Society of Animal Science

>SPECIAL TOPICS

>Prion biology relevant to bovine spongiform encephalopathy1

>J. Novakofski*,2, M. S. Brewer{dagger}, N.

>Mateus-Pinilla{ddagger}, J. Killefer* and R. H. McCusker*

>

>* Departments of Animal Sciences and {dagger} Food

>Science and Human Nutrition, University of Illinois at

>Urbana­Champaign 61801-4737; and {ddagger} Illinois

>Natural History Survey, Center for Wildlife and Plant

>Ecology, Champaign, IL 61820

>

>2 Correspondence: 1503 South Maryland Dr. (phone:

>217-333-6181; e-mail: Jnova@uiuc.edu).

>

>Bovine spongiform encephalopathy (BSE) and chronic

>wasting disease (CWD) of deer and elk are a threat to

>agriculture and natural resources, as well as a human

>health concern. Both diseases are transmissible

>spongiform encephalopathies (TSE), or prion diseases,

>caused by autocatalytic conversion of endogenously

>encoded prion protein (PrP) to an abnormal, neurotoxic

>conformation designated PrPsc. Most mammalian species

>are susceptible to TSE, which, despite a range of

>species-linked names, is caused by a single highly

>conserved protein, with no apparent normal function. In

>the simplest sense, TSE transmission can occur because

>PrPsc is resistant to both endogenous and environmental

>proteinases, although many details remain unclear.

>Questions about the transmission of TSE are central to

>practical issues such as livestock testing, access to

>international livestock markets, and wildlife

>management strategies, as well as intangible issues

>such as consumer confidence in the safety of the meat

>supply. The majority of BSE cases seem to have been

>transmitted by feed containing meat and bone meal from

>infected animals. In the United Kingdom, there was a

>dramatic decrease in BSE cases after neural tissue and,

>later, all ruminant tissues were banned from ruminant

>feed. However, probably because of heightened awareness

>and widespread testing, there is growing evidence that

>new variants of BSE are arising "spontaneously,"

>suggesting ongoing surveillance will continue to find

>infected animals. Interspecies transmission is

>inefficient and depends on exposure, sequence homology,

>TSE donor strain, genetic polymorphism of the host, and

>architecture of the visceral nerves if exposure is by

>an oral route. Considering the low probability of

>interspecies transmission, the low efficiency of oral

>transmission, and the low prion levels in nonnervous

>tissues, consumption of conventional animal products

>represents minimal risk. However, detection of rare

>events is challenging, and TSE literature is

>characterized by subsequently unsupported claims of

>species barriers or absolute tissue safety. This review

>presents an overview of TSE and summarizes recent

>research on pathogenesis and transmission.

>

>Key Words: Bovine Spongiform Encephalopathy . Chronic

>Wasting Disease . Prion

>

>http://jas.fass.org/cgi/content/abstract/83/6/1455

>

> >there is growing evidence that new variants of BSE are

>arising "spontaneously,"

>

>

>THERE is NO evidence of a 'spontaneous' TSE anywhere that

>is infectious and shows the pathology of any natural TSE.

>if i have missed something, could someone please site this

>science to me please.

>

>

> >Considering the low probability of interspecies

>transmission, the low efficiency of oral transmission,

>and the low prion levels in nonnervous tissues,

>consumption of conventional animal products represents

>minimal risk.

>

>

>I DISAGREE with all of the above. all one has to do is

>read transmission

>studies. scrapie infected sheep and goats, CWD infected

>deer and

>elk (who knows how many strains) and undocumented TSEs

>in the

>USA bovine have been rendered and fed to animals for

>humna/animal

>consumption for decades. it's only a pipe dream that

>none of this

>was infectious. to think of a 'spontaneous' TSE as just

>popping

>up from nowhere, is like believing in Santa Claus. remember

>the USA scrapie research in Mission, Texas. IT did NOT look

>like BSE...

>

>

>1: J Infect Dis 1980 Aug;142(2):205-8

>

> Oral transmission of kuru, Creutzfeldt-Jakob

>disease, and scrapie to nonhuman primates.

>

> Gibbs CJ Jr, Amyx HL, Bacote A, Masters CL,

>Gajdusek DC.

>

> Kuru and Creutzfeldt-Jakob disease of humans and

>scrapie disease of sheep and goats were transmitted to

>squirrel monkeys (Saimiri sciureus) that were exposed

>to the infectious agents only by their nonforced

>consumption of known infectious tissues. The

>asymptomatic incubation period in the one monkey

>exposed to the virus of kuru was 36 months; that in the

>two monkeys exposed to the virus of Creutzfeldt-Jakob

>disease was 23 and 27 months, respectively; and that in

>the two monkeys exposed to the virus of scrapie was 25

>and 32 months, respectively. Careful physical

>examination of the buccal cavities of all of the

>monkeys failed to reveal signs or oral lesions. One

>additional monkey similarly exposed to kuru has

>remained asymptomatic during the 39 months that it has

>been under observation.

>

>PMID: 6997404

>http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_u

ids=6997404&dopt=Abstract

>

>

>3. You will recall that the advice provided by

>Professor Smith in

>1993 and by Dr. Gore this month used the sub-population

>of dairy

>farm workers who had had a case of BSE on their farms -

>63,000, which is approximately half the number of dairy

>farm

>workers - as a denominator. If the above sums are

>repeated using

>this denominator population, taking an annual incidence

>in the general

>population of 1 per million the observed rate in this

>sub-population

>is 10 TIMES, and taking an annual incidence of 0.7 per

>million,

>IT IS 15 TIMES (THE ''WORST CASE'' SCENARIO) than

>that in the general population...

>

>http://www.bseinquiry.gov.uk/files/yb/1995/01/31004001.pdf

>

>

>It was, however, performed in the USA in 1979, when it

>was shown that

>cattle inoculated with the scrapie agent endemic in the

>flock of Suffolk

>sheep at the United States Department of Agriculture in

>Mission, Texas,

>developed a TSE quite unlike BSE. 32 <

>

>http://www.bseinquiry.gov.uk/report/volume2/chaptea3.htm#820543

>

>The findings of the initial transmission, though not of

>the clinical or

>neurohistological examination, were communicated in

>October 1988 to Dr

>Watson, Director of the CVL, following a visit by Dr

>Wrathall, one of

>the project leaders in the Pathology Department of the

>CVL, to the

>United States Department of Agriculture. 33

>

>

>http://www.bseinquiry.gov.uk/report/volume2/chaptea3.htm#820546

>

>

>The results were not published at this point, since the

>attempted

>transmission to mice from the experimental cow brain

>had been

>inconclusive. The results of the clinical and

>histological differences

>between scrapie-affected sheep and cattle were

>published in 1995.

>Similar studies in which cattle were inoculated

>intracerebrally with

>scrapie inocula derived from a number of

>scrapie-affected sheep of

>different breeds and from different States, were

>carried out at the US

>National Animal Disease Centre. 34

>

>

>http://www.bseinquiry.gov.uk/report/volume2/chaptea3.htm#820549

>

>

>The results, published in 1994, showed that this source

>of scrapie

>agent, though pathogenic for cattle, did not produce

>the same clinical

>signs of brain lesions characteristic of BSE.

>

>

>http://www.bseinquiry.gov.uk/report/volume2/chaptea3.htm

>

>

>Visit to USA ... info on BSE and Scrapie

>

>

>http://www.bseinquiry.gov.uk/files/yb/1988/10/00001001.pdf

>

>

>HOUND STUDY

>

>AS implied in the Inset 25 we must not _ASSUME_ that

>transmission of BSE to other species will invariably

>present pathology typical of a scrapie-like disease.

>

>snip...

>

>http://www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf

>

>

>In Confidence - Perceptions of unconventional slow

>virus diseases

>of animals in the USA - APRIL-MAY 1989 - G A H Wells

>

>

>http://www.bseinquiry.gov.uk/files/mb/m11b/tab01.pdf

>

>WHY is USA insisting _now_ not to use WB, when on the

>1st _confirmed_

>case Dec. 23, 2003

>USA mad cow, WB was used ???

>

>maybe this is the reason ;

>

>JAPAN BSE # 8 & 9 cow

>

>8. 6/10/2003 Holstein Steer 13/10/2001 23 mths

>No clinical signs WB+, IHC-, HP-

>

>

>9. 4/11/2003 Holstein Steer 13/1/2002

>21 mths No clinical signs WB+, IHC-, HP-

>

>===========

>

>More information on the first 11 Japanese BSE-cases can

>be found on the


>website of the Japanese Embassy in the US:

>

>http://www.us.emb-japan.go.jp/english/html/fafacts/bse/bse.htm

>

>

>IN fact, the new strain of TSE in cattle BaSE, does not

>look like nvCJD in humans, but very similar

>to the sporadic CJD;

>

>

>BASE in cattle in Italy of Identification of a second

>bovine amyloidotic spongiform encephalopathy: Molecular

>similarities with sporadic Creutzfeldt-Jakob disease

>

>http://www.pnas.org/cgi/content/abstract/0305777101v1

>

>

>Adaptation of the bovine spongiform encephalopathy

>agent to primates and comparison with Creutzfeldt-

>Jakob disease: Implications for human health THE

>findings from Corinne Ida Lasmézas*, [dagger] ,

>Jean-Guy Fournier*, Virginie Nouvel*, Hermann Boe*,

>Domíníque Marcé*, François Lamoury*, Nicolas Kopp

>[Dagger ] , Jean-Jacques Hauw§, James Ironside¶, Moira

>Bruce [||] , Dominique Dormont*, and Jean-Philippe

>Deslys* et al, that The agent responsible for French

>iatrogenic growth hormone-linked CJD taken as a control

>is very different from vCJD but is similar to that

>found in one case of sporadic CJD and one sheep scrapie

>isolate;

>

>

>http://www.pnas.org/cgi/content/full/041490898v1

>

>

>Characterization of two distinct prion strains derived

>from bovine spongiform encephalopathy transmissions to

>inbred mice

>

>

> http://vir.sgmjournals.org/cgi/content/abstract/85/8/2471

>

>

>USA BSE GBR III

>

>http://www.efsa.eu.int/science/efsa_scientific_reports/gbr_assessments/scr_

annexes/574/sr03_biohaz02_usa_report_annex_en1.pdf

>

>http://www.fda.gov/ohrms/dockets/dockets/03n0312/03N-0312_emc-000001.txt

>

>https://web01.aphis.usda.gov/regpublic.nsf/0/eff9eff1f7c5cf2b87256ecf000df0

8d?OpenDocument

>

>https://web01.aphis.usda.gov/BSEcom.nsf/0/b78ba677e2b0c12185256dd300649f9d?

OpenDocument&AutoFramed

>

>

>Terry S. Singeltary SR.

>P.O. Box 42

>Bacliff, Texas USA 77518


~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~


La Vonne Gallo

HighWire Press

1454 Page Mill Road

Palo Alto, CA 94304

fax: 650.725.9335

lgallo@highwire.stanford.edu


~~~~~~~~~~~

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