fedup2 said:
Flounder. You have been running around the internet screaming "The sky is falling" for a long time. I am sorry for your loss. Most all of us have lost loved ones but not all of us become obsessed with it.
I can google tss singletary and get nine full pages to sites where you have been screaming "Mad Cow"! Including veggie heads. You have been on pet sites telling people their pets are dying from mad cow, you have been on hunting sites, most every site where people will put up with you trying to tie every disease, sickness, & death to mad cow disease! I saw where one reader described you as a "raving lunatic".
I also saw the response from the chairman - woman of that Korean political party where her response to you included:
{"Through the internet, many people here in Korea have learnt about the story of your family and it has created a great deal of concern. Your experience had particular relevance for the citizens here, for the Korean government, in connection to the free trade agreement it is negotiating with the United States, is about to restart the sale of the unsafe American beef to the general public.}"
Who the hell says its unsafe beef? You? And people wonder why beef demand is dropping! :roll:
i will address this BSe in order ;
>>>Most all of us have lost loved ones but not all of us become obsessed with it<<<
IF wanting justice for corporate and policital homicide is being obsessed, oh well, i guess i am obsessed. i want answers. you want to cover it up. not gonna happen. ...
>>>I can google tss singletary and get nine full pages to sites where you have been screaming "Mad Cow"! Including veggie heads. You have been on pet sites telling people their pets are dying from mad cow, you have been on hunting sites, most every site where people will put up with you trying to tie every disease, sickness, & death to mad cow disease! <<<
not so, i only try to tie TSE related disease to mad cow.
so i post to more than one site, and some include veggie sites, so what. so i post the truth to more than one site. so i post to hunting sites, and to pet sites. don't we have CWD in deer and elk running rampant. don't we have pet food killing our dogs and cats. and how many of those cats and dogs in the USA ever get tested for a TSE. don't you imagine since our federal government is willing to cover-up mad cow disease in Texas, they can do it in cats, and dogs, by simply not looking. it works for cattle. let me remind you of a few things about this following the part you said about the Koreans. you just posted a short part of that, let me post the rest ;
>>>I also saw the response from the chairman - woman of that Korean political party where her response to you included:
{"Through the internet, many people here in Korea have learnt about the story of your family and it has created a great deal of concern. Your experience had particular relevance for the citizens here, for the Korean government, in connection to the free trade agreement it is negotiating with the United States, is about to restart the sale of the unsafe American beef to the general public.}" <<<
here is the full text,
Name: Terry S. Singeltary Sr. Date: Jan 26, 2007
Dear Terry S. Singeltary Sr.
My name in Seoungwon Lee and I work for National Assemblywoman (MP) Sang-Jeong Sim, in South Korea. Below is a message from Mr. Kwon. Please get back to us regarding the letter.
Thank you.
Seoungwon Lee
Legislative Assistant
National Assembly
Republic of Korea
Dear Terry S. Singeltary Sr.
Greetings from Korea.
I have learned about what you and your family have been through a internet. Being the father of a small family, I can only imagine the sense of loss that you and your family still must feel regarding your mother.
Through the internet, many people here in Korea have learnt about the story of your family and it has created a great deal of concern. Your experience had particular relevance for the citizens here, for the Korean government, in connection to the free trade agreement it is negotiating with the United States, is about to restart the sale of the unsafe American beef to the general public.
The Korean government has already imported the first load American beef and is currently going through the inspection period. There has been a great deal of controversy regarding the safety of these products, and a precipitous increase in public interest regarding the causes and symptoms of vCJD.
It is in this context that we are preparing a session together with National Assembly members and relevant NGOs to hear from those who have had direct experience with the human variant of BSE. We are very interested in hearing about the symptoms that your mother showed, your views on the response from the US government to the many deaths that resulted from this disease, the reaction from the general public, and any other area that you would wish to speak about. There would also be experts and academics in the area to speak at the session as well. We believe that it would greatly contribute to raising awareness about the issue, as well as help politicians and civic activists to consider the repercussions of the beef import issue.
We would like to request your help in this regard. We are planning for the session to be held in Seoul on the 23rd of this November. We would very much like to have you present in order to help prevent such tragic incidents from happening in Korea. We would, of course, pay for the trip and accommodations for the duration of your stay. If you were to participate, we could also meet with members from the agricultural committee of the National Assembly and from other related organizations to urge more interest to the issue.
Please do not hesitate to offer suggestions or ask us any questions that you might have. We look forward to a positive response and to meeting you in Seoul.
Thank you.
Sincerely,
Sim Sang-Jeong
Head of the Democratic Labor Party Parliamentary Committee on the Korea-US FTA
Member of the National Assembly
Republic of Korea
http://www.fpif.org/fpiftxt/3940
===================
SO they wanted to know the truth. i cannot help that. they cannot seem to get the truth from this administration. ...
NOW, about those pets you dont seem to think that can get mad cow disease i.e. TSE, and then show us where we are testing to see if any of the reported canine or feline dementia related deaths are a TSE here in the USA ? we know how they test for cattle :roll:
GAH WELLS (very important statement here...TSS)
HOUND STUDY
AS implied in the Inset 25 we must not _ASSUME_ that
transmission of BSE to other species will invariably
present pathology typical of a scrapie-like disease.
snip...
http://www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf
76 pages on hound study;
http://www.bseinquiry.gov.uk/files/sc/seac16/tab04.pdf
The spongiform changes were not pathognomonic (ie.
conclusive proof) for prion disease, as they were atypical,
being largely present in white matter rather than grey matter in
the brain and spinal cord. However, Tony Scott, then head of
electron microscopy work on TSEs, had no doubt that these
SAFs were genuine and that these hounds therefore must have
had a scrapie-like disease. I reviewed all the sections
myself (original notes appended) and although the pathology
was not typical, I could not exclude the possibility that this was
a scrapie-like disorder, as white matter vacuolation is seen
in TSEs and Wallerian degeneration was also present in the
white matter of the hounds, another feature of scrapie.
38.I reviewed the literature on hound neuropathology, and
discovered that micrographs and descriptive neuropathology from
papers on 'hound ataxia' mirrored those in material from
Robert Higgins' hound survey. Dr Tony Palmer (Cambridge) had
done much of this work, and I obtained original sections
from hound ataxia cases from him. This enabled me provisionally to
conclude that Robert Higgins had in all probability detected
hound ataxia, but also that hound ataxia itself was possibly a
TSE. Gerald Wells confirmed in 'blind' examination of single
restricted microscopic fields that there was no distinction
between the white matter vacuolation present in BSE and
scrapie cases, and that occurring in hound ataxia and the hound
survey cases.
39.Hound ataxia had reportedly been occurring since the 1930's,
and a known risk factor for its development was the feeding
to hounds of downer cows, and particularly bovine offal.
Circumstantial evidence suggests that bovine offal may also be
causal in FSE, and TME in mink. Despite the inconclusive
nature of the neuropathology, it was clearly evident that this
putative canine spongiform encephalopathy merited further
investigation.
40.The inconclusive results in hounds were never confirmed,
nor was the link with hound ataxia pursued. I telephoned Robert
Higgins six years after he first sent the slides to CVL.
I was informed that despite his submitting a yearly report to the
CVO including the suggestion that the hound work be continued,
no further work had been done since 1991. This was
surprising, to say the very least.
41.The hound work could have provided valuable evidence
that a scrapie-like agent may have been present in cattle offal long
before the BSE epidemic was recognised. The MAFF hound
survey remains unpublished.
Histopathological support to various other published
MAFF experiments
42.These included neuropathological examination of material
from experiments studying the attempted transmission of BSE to
chickens and pigs (CVL 1991) and to mice (RVC 1994).
http://www.bseinquiry.gov.uk/witness/htm/stat067.htm
It was thought likely that at least some, and probably all, of the cases
in zoo animals were caused by the BSE agent. Strong support for this
hypothesis came from the findings of Bruce and others (1994)
( Bruce, M.E., Chree, A., McConnell, I., Foster, J., Pearson, G. &
Fraser, H. (1994) Transmission of bovine spongiform encephalopathy and
scrapie to mice: strain variation and species barrier. Philosophical
Transactions of the Royal Society B 343, 405-411: J/PTRSL/343/405
), who demonstrated that the pattern of variation in incubation period
and lesion profile in six strains of mice inoculated with brain
homogenates from an affected kudu and the nyala, was similar to that
seen when this panel of mouse strains was inoculated with brain from
cattle with BSE. The affected zoo bovids were all from herds that were
exposed to feeds that were likely to have contained contaminated
ruminant-derived protein and the zoo felids had been exposed, if only
occasionally in some cases, to tissues from cattle unfit for human
consumption.
snip...
http://www.bseinquiry.gov.uk/files/ws/s324.pdf
2005
DEFRADepartment for Environment,Food & Rural Affairs
Area 307, London, SW1P 4PQTelephone: 0207 904 6000Direct line: 0207 904 6287E-mail: h.mcdonagh.defra.gsi.gov.uk
GTN:FAX:
Mr T S SingeltaryP.O. Box 42BacliffTexasUSA 77518
21 November 2001
Dear Mr Singeltary TSE IN HOUNDS
Thank you for e-mail regarding the hounds survey. I am sorry for the long delay in responding.
As you note, the hound survey remains unpublished. However the Spongiform Encephalopathy Advisory Committee (SEAC),
the UK Government's independent Advisory Committee on all aspects related to BSE-like disease, gave the hound study
detailed consideration at their meeting in January 1994. As a summary of this meeting published in the BSE inquiry noted,
the Committee were clearly concerned about the work that had been carried out, concluding that there had clearly been
problems with it, particularly the control on the histology, and that it was more or less inconclusive. However was agreed
that there should be a re-evaluation of the pathological material in the study.
Later, at their meeting in June 95, The Committee re-evaluated the hound study to see if any useful results could be
gained from it. The Chairman concluded that there were varying opinions within the Committee on further work. It did
not suggest any further transmission studies and thought that the lack of clinical data was a major weakness.
Overall, it is clear that SEAC had major concerns about the survey as conducted. As a result it is likely that the
authors felt that it would not stand up to peer review and hence it was never published. As noted above, and in the
detailed minutes of the SEAC meeting in June 95, SEAC considered whether additional work should be performed to
examine dogs for evidence of TSE infection. Although the Committee had mixed views about the merits of conducting
further work, the Chairman noted that when the Southwood Committee made their recommendation to complete an
assessment of possible spongiform disease in dogs, no TSEs had been identified in other species and hence dogs
were perceived as a high risk population and worthy of study. However subsequent to the original recommendation,
made in 1990, a number of other species had been identified with TSE ( e.g. cats) so a study in hounds was less
critical.
For more details see-
http://www.bseinquiry.gov.uk/files/yb/1995/06/21005001.pdf
As this study remains unpublished, my understanding is that the ownership of the data essentially remains with the original researchers.
Thus unfortunately, I am unable to help with your request to supply information on the hound survey directly. My only suggestion is
that you contact one of the researchers originally involved in the project, such as Gerald Wells. He can be contacted at the following address.
Dr Gerald Wells, Veterinary Laboratories Agency, New Haw, Addlestone, Surrey, KT 15 3NB, UK
You may also wish to be aware that since November 1994 all suspected cases of spongiform encephalopathy in animals and poultry were made notifiable.
Hence since that date there has been a requirement for vets to report any suspect SE in dogs for further investigation. To date there has never
been positive identification of a TSE in a dog.
I hope this is helpful
Yours sincerely 4
HUGH MCDONAGHBSE CORRESPONDENCE SECTION
==========================================================
some 100+ _documented_ TSE cats of all types later...tss
on occassions, materials obtained from slaughterhouses
will be derived from sheep affected with scrapie or
cattle that may be incubating BSE for use in petfood manufacture...
http://www.bseinquiry.gov.uk/files/yb/1989/05/03007001.pdf
Meldrum's notes on pet foods and materials used
http://www.bseinquiry.gov.uk/files/yb/1989/05/16001001.pdf
http://www.bseinquiry.gov.uk/files/yb/1989/05/16002001.pdf
IN CONFIDENCE CJD TO CATS...
http://www.bseinquiry.gov.uk/files/yb/1989/05/18002001.pdf
Confidential BSE and __________________
http://www.bseinquiry.gov.uk/files/yb/1989/05/22012001.pdf
1st case natural FSE
http://www.bseinquiry.gov.uk/files/yb/1990/05/09002001.pdf
FSE and pharmaceuticals
http://www.bseinquiry.gov.uk/files/yb/1990/05/10005001.pdf
confidential cats/dogs and unsatisfactory posture
MAFFs failure to assure key research
http://www.bseinquiry.gov.uk/files/yb/1990/06/14006001.pdf
can't forget about the mad man and his mad cat;
Deaths of CJD man and cat linked
http://news.bbc.co.uk/1/hi/health/184558.stm
In October 1998 the simultaneous occurrence of spongiform encephalopathy
in a man and his pet cat was reported. The report from Italy noted that
the cat did not display the same clinical features as FSE cases
previously seen. Indeed, the presence of a new type of FSE was
suggested. The man was diagnosed as having sporadic CJD, and neither
case (man nor cat) appeared to be affected by a BSE-related condition.
http://www.defra.gov.uk/animalh/bse/bse-science/level-4-othertses.html
Image] Research letters Volume 352, Number 9134 [Image] 3 October1998[Previous] [Next] [Image][Image]
Simultaneous occurrence of spongiform encephalopathy in a manand his cat in Italy
[Image] Gianluigi Zanusso, Ettore Nardelli, Anna Rosati, GianMaria Fabrizi, SergioFerrari, Antonella Carteri, Franco De Simone, Nicola Rizzuto, SalvatoreMonaco
Transmissible spongiform encephalopathies (TSE) encompass inherited,acquired, and sporadic mammalian neurological disorders, and arecharacterised by the conversion of the cellular prion protein (PrP) in aninsoluble and protease-resistant isoform (PrPres). In human TSE, four typesof PrPres have been identified according to size and glycoform ratios, whichmay represent different prion strains. Type-1 and type-2 PrPres areassociated with sporadic Creutzfeldt-Jakob disease (CJD), type 3 withiatrogenic CJD, and type 4 with variant CJD.1,2 There is evidence thatvariant CJD is caused by the bovine spongiform encephalopathy (BSE)-prionstrain.2-4 The BSE strain has been identified in three cats with felinespongiform encephalopathy (FSE), a prion disease which appeared in 1990 inthe UK.5 We report the simultaneous occurrence of sporadic CJD in a man anda new variety of FSE in his cat. A 60-year-old man, with no unusual dietary habits, was admitted in November,1993, because of dysarthria, cerebellar ataxic gait, visual agnosia, andmyoclonus. An electroencephalogram (EEG) showed diffuse theta-deltaactivity. A brain magnetic resonance imaging scan was unremarkable. 10 dayslater, he was speechless and able to follow only simple commands. RepeatEEGs showed periodic triphasic complexes. 2 weeks after admission, he wasmute, akinetic, and unable to swallow. He died in early January, 1994. His 7-year-old, neutered, female shorthaired cat presented in November,1993, with episodes of frenzy, twitching of its body, and hyperaesthesia.The cat was usually fed on canned food and slept on its owner's bed. Nobites from the cat were recalled. In the next few days, the cat becameataxic, with hindquarter locomotor dysfunction; the ataxia got worse andthere was diffuse myoclonus. The cat was killed in mid-January, 1994. No pathogenic mutations in the patient's PrP gene were found. The patientand the cat were methionine homozygous at codon 129. Histology of thepatient's brain showed neocortical and cerebellar neuronal loss,astrocytosis, and spongiosis (figure A). PrP immunoreactivity showed apunctate pattern and paralleled spongiform changes (figure B). The cat'sbrain showed mild and focal spongiosis in deeper cortical layers of all fourlobes (figure C), vacuolated cortical neurons (figure D), and mildastrogliosis. The cerebellar cortex and the dentate nucleus were gliosed.Immunoreactive PrP showed a punctate pattern in neocortex, allocortex, andcaudate nucleus (figure E). Western blot analysis of control and affectedhuman and cat brain homogenates showed 3 PrP bands of 27-35 kDa. Afterdigestion with proteinase K and deglycosylation, only samples from theaffected patient and cat showed type-1 PrPres, with PrP glycoform ratioscomparable to those observed in sporadic CJD1 (details available fromauthor). [Image] Microscopic sections of patient and cat brains A: Occipital cortex of the patient showing moderate spongiformdegeneration and neuronal loss (haematoxylin and eosin) and B: punctateperineuronal pattern of PrP immunoreactivity; peroxidaseimmunohistochemistry with monoclonal antibody 3F4. C: cat parietal cortexshowing mild spongiform degeneration (haematoxylin and eosin).D:vacuolated neurons (arrow, haematoxylin and eosin), E: peroxidaseimmunohistochemistry with antibody 3F4 shows punctate perineuronaldeposition of PrP in temporal cortex. This study shows a spatio-temporal association between human and felineprion diseases. The clinical features of the cat were different frompreviously reported cases of FSE which were characterised by gradual onsetof behavioural changes preceding locomotor dysfunction and ataxia.5Neuropathological changes were also at variance with the diffuse spongiosisand vacuolation of brainstem neurons, seen in FSE.5 The synaptic pattern ofPrP deposition, similar in the cat and in the patient, was atypical for aBSE-related condition. Evidence of a new type of FSE was further provided bythe detection of a type-1 PrPres, other than the BSE-associated type 4.2Taken together, our data suggest that the same agent strain of sporadic CJDwas involved in the patient and in his cat. It is unknown whether these TSE occurred as the result of horizontaltransmission in either direction, infection from an unknown common source,or the chance occurrence of two sporadic forms.
1 Parchi P, Castellani R, Capellari S, et al. Molecular basis of phenotypicvariablity in sporadic Creutzfeldt-Jakob disease. Ann Neurol 1996; 39:767-78 [PubMed]. 2 Collinge J, Sidle KCL, Meads J, Ironside J, Hill AF. Molecular analysis ofprion strain variation and the aetiology of 'new variant' CJD. Nature 1996;383: 685-90 [PubMed]. 3 Bruce ME, Will RG, Ironside JW, et al. Transmissions to mice indicate that'new variant' CJD is caused by the BSE agent. Nature 1997; 389: 498-501[PubMed]. 4 Hill AF, Desbruslais M, Joiner S, et al. The same prion strain causes vCJDand BSE. Nature 1997; 389: 448-50 [PubMed]. 5 Pearson GR, Wyatt JM, Henderson JP, Gruffydd-Jones TJ. Feline spongiformencephalopathy: a review. Vet Annual 1993; 33: 1-10.
------------------------------------------------------------------------
Sezione di Neurologie Clinica, Dipartimento di Scienze Neurologiche e dellaVisione, Università di Verona, Policlinico Borgo Roma, 37134 Verona, Italy(S Monaco; e mail
[email protected]); and Istituto ZooprofilatticoSperimentale della Lombardia e dell' Emilia, Brescia
=========================================TSS
indeed there have been 4 documented cases of TSE in Lions to date.
Lion 32 December 98 Born November 86
Lion 33 May 1999 (euthanased) Born November 81.
Lion 36 Euthanased August 2000 Born July 87. Deteriorating hind limb
ataxia.
Lion 37 Euthanased November 2001 Male, 14 years. Deteriorating hind
limb ataxia since September 2001. (Litter mate to Ref. 36.)
http://www.defra.gov.uk/animalh/bse/index.html
go to the url above, on the bar at the top, click on _statistics_,
then in middle of next page, click on _other TSEs_.
or go here;
http://www.defra.gov.uk/animalh/bse/bse-statistics/level-3-tsestat.html
and
http://www.defra.gov.uk/animalh/bse/bse-science/level-4-othertses.html
http://www.bseinquiry.gov.uk/files/yb/1992/11/13001001.pdf
also;
Reports on the clinical symptoms presented by these cats give a
relatively homogeneous picture: Affected cats show a lack of
coordination with an ataxia mainly of the hind limbs, they often fall
and miss their target when jumping. Fear and increased aggressiveness
against the owner and also other animals is often seen. They do not
longer tolerate to be touched (stroked) and start hiding. These
behavioural chances might be the result of a hypersensibility to touch
and noise, but also to increased fear. Excessive salivation is another
more frequently seen symptom. Cats with FSE in general show severe
behavioural disturbances, restlessness and depression, and a lack of
coat cleaning. Symptoms in large cats in general are comparable to those
in domestic cats. A
report on FSE (in german) has been presented in 2001 in the Swiss FVO
Magazin. A paper on the first FSE case in a domestic cat in Switzerland
is currently in press in the Journal Schweizer Archiv für Tierheilkunde
(SAT).
http://www.neurocenter-bern.ch/tse_e.shtml
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0612&L=sanet-mg&T=0&F=&S=&P=8385
>>>And people wonder why beef demand is dropping! <<<
as you so kindly put it ;
Stop shooting yourselves in the foot & bitching about the bleeding! :wink:
why not try fixing the problems ???
OH, thats right, USA beef is safe, fedup2 says so.
i say it could and should be a hell of a lot safer :nod:
TSS
