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BSE CASE CONFIRMED IN ALBERTA OTTAWA, August 15, 2008

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BSE CASE CONFIRMED IN ALBERTA OTTAWA, August 15, 2008 - The Canadian Food Inspection Agency (CFIA) has confirmed bovine spongiform encephalopathy (BSE) in a six-year-old beef cow from Alberta. No part of the animal's carcass entered the human food or animal feed systems.

The animal's birth farm has been identified, and an investigation is underway. The CFIA is tracing the animal's herdmates at the time of birth and examining possible sources of infection. The age and location of the infected animal are consistent with previous cases detected in Canada.

This case was detected through the national BSE surveillance program, which has been highly successful in demonstrating the low level of BSE in Canada. The program continues to play an important role in Canada's strategy to manage BSE.

Canada remains a Controlled Risk country for BSE, as recognized by the World Organisation for Animal Health (OIE). Accordingly, this case should not affect exports of Canadian cattle or beef.

- 30 -

For information:

Canadian Food Inspection Agency Media relations: 613-228-6682

http://www.inspection.gc.ca/english/anima/heasan/disemala/bseesb/ab2008/14notavie.shtml


Amazing what you will find when you look. The Canadians are doing a far better job than the US in confirming BSE/TSE cases. ...TSS



Wednesday, June 11, 2008

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

snip...SEE FULL TEXT with facts and sources @ ;

http://usdavskorea.blogspot.com/2008/06/oie-recognition-of-bse-status-of.html

http://organicconsumers.org/forum/index.php?showtopic=1566

Friday, April 25, 2008

Substances Prohibited From Use in Animal Food or Feed [Docket No. 2002N-0273] (Formerly Docket No. 02N-0273) RIN 0910-AF46

http://madcowfeed.blogspot.com/2008/04/substances-prohibited-from-use-in.html

Review on the epidemiology and dynamics of BSE epidemics

Cases of atypical BSE have only been found in countries having implemented large active surveillance programs. As of 1st September 2007, 36 cases (16 H, 20 L) have been described all over the world in cattle: Belgium (1 L) [23], Canada (1 H)15, Denmark (1 L)16, France (8 H, 6 L)17, Germany (1 H, 1 L) [13], Italy (3 L)18, Japan (1 L) [71], Netherlands (1 H, 2 L)19, Poland (1 H, 6 L)20, Sweden (1 H)21, United Kingdom (1 H)22, and USA (2 H)23. Another H-type case has been found in a 19 year old miniature zebu in a zoological park in Switzerland [56]. It is noteworthy that atypical cases have been found in countries that did not experience classical BSE so far, like Sweden, or in which only few cases of classical BSE have been found, like Canada or the USA.

And last but not least, similarities of PrPres between Htype BSE and human prion diseases like CJD or GSS have been put forward [10], as well as between L-type BSE and CJD [17]. These findings raise questions about the origin and inter species transmission of these prion diseases that were discovered through the BSE active surveillance.

full text 18 pages ;

http://www.vetres.org/index.php?option=article&access=standard&Itemid=129&url=/articles/vetres/pdf/2008/04/v07232.pdf

USA BSE ACTIVE SURVEILLANCE ???

http://bse-atypical.blogspot.com/2008/06/review-on-epidemiology-and-dynamics-of.html

Please remember, the last two mad cows documented in the USA i.e. Alabama and Texas, both were of the 'atypical' BSE strain, and immediately after that, the USDA shut down the testing from 470,000 to 40,000 in the U.S. in 2007 out of about 35 million cattle slaughtered. also, science is showing that some of these atypical cases are more virulent to humans than the typical UK BSE strain ;

***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.***

Progress Report from the National Prion Disease Pathology Surveillance Center

An Update from Stephen M. Sergay, MB, BCh & Pierluigi Gambetti, MD

April 3, 2008

http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45

In this context, a word is in order about the US testing program. After the discovery of the first (imported) cow in 2003, the magnitude of testing was much increased, reaching a level of >400,000 tests in 2005 (Figure 4). Neither of the 2 more recently indigenously infected older animals with nonspecific clinical features would have been detected without such testing, and neither would have been identified as atypical without confirmatory Western blots. Despite these facts, surveillance has now been decimated to 40,000 annual tests (USDA news release no. 0255.06, July 20, 2006) and invites the accusation that the United States will never know the true status of its involvement with BSE.

In short, a great deal of further work will need to be done before the phenotypic features and prevalence of atypical BSE are understood. More than a single strain may have been present from the beginning of the epidemic, but this possibility has been overlooked by virtue of the absence of widespread Western blot confirmatory testing of positive screening test results; or these new phenotypes may be found, at least in part, to result from infections at an older age by a typical BSE agent, rather than neonatal infections with new "strains" of BSE. Neither alternative has yet been investigated.

http://www.cdc.gov/ncidod/EID/vol12no12/06-0965.htm

Tuesday, June 3, 2008

SCRAPIE USA UPDATE JUNE 2008 NOR-98 REPORTED PA

http://nor-98.blogspot.com/2008/06/scrapie-usa-update-june-2008-nor-98.html

Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518
 
Pssstttt Terry,Albertas a province....Ottawa's a city in the province of Ontario..... :wink:
 
There must be some mistake. A six-year old would mean it was born in 2002 but the USDA says the feed ban was effective in 1997. Somebody isn't telling the truth here....
 
Sandhusker said:
There must be some mistake. A six-year old would mean it was born in 2002 but the USDA says the feed ban was effective in 1997. Somebody isn't telling the truth here....

And that none born after the " magic 1999" number could be postive- so none would be imported to the US :roll: :wink: :lol: :( :( :mad:
 
Oldtimer said:
Sandhusker said:
There must be some mistake. A six-year old would mean it was born in 2002 but the USDA says the feed ban was effective in 1997. Somebody isn't telling the truth here....

And that none born after the " magic 1999" number could be postive- so none would be imported to the US :roll: :wink: :lol: :( :( :mad:


And obviously discontinuing testing in the US was a stroke of genius, because the "magic 1997" number must have done it's job in the US :roll: :wink: :lol: :cry: :cry: :mad:
 
If you're asking us to defend the USDA, you're not getting it, Silver.

Can you explain how positives can be born 5 years after the ban was declared effective?
 
Can you explain how positives can be born 5 years after the ban was declared effective?

I'll take a wild guess. Uhhh lemme think..... uhh .... contaminated feed is NOT the cause of BSE???
Maybe Mark Purdy was right all this time????
 
Imagine how many positives might show up if all animals were tested??? Not just a very very small percentage!! Truly laughable testing protocols in both countries. Not to mention misleading, dangerous and totally irresponsible on the part of the so-called governments.

So, the feed ban and yet another BSE case rears its ugly head again? Oh well, let the games begin - all over again.

It amazes me to hear the CFIA claim that their testing protocol actually works. Sure, every once in a while, they come forward and say - "Hey people, we got one - another one - but hey people, we caught it before it went into the food chain. Do they do this just to make you guys feel comfy about how they manage that dumb-ass system they seem to have us all relying on??

But hey people - if they do not test all of them - hey people, how many other positive got through??? You know, the ones that had not even began to show clinical signs of carrying the disease????

Like I have said many times before - smoke and mirrors folks - smoke and mirrors.
 
Bward said:
Can you explain how positives can be born 5 years after the ban was declared effective?

I'll take a wild guess. Uhhh lemme think..... uhh .... contaminated feed is NOT the cause of BSE???
Maybe Mark Purdy was right all this time????

:shock: Na that would make way to much sense.
Same cluster area,,, well, lets keep testing ,the more we test the more we will find and hopefully we will find the true cause to BSE in cattle. I really think they made the right call to skew the testing group to younger animals hopefully we will get some new information.


Oldtimer I agree 100% with you, our cattle has no business going into the States because your government doesn't test a damn thing, so guess what all the crap everyone is eating. I mean with all the BSE cases that you haven't found of your own, as well as our imports ,,,,,
Maybe you all would be better off eating Argentinian beef :roll: .
 
BSE tester, the game has never ended, to there is no restarting!  For all those that feel BSE is a non-issue in North America and lives, WAKE UP.Your federal, state, and provincial governments are in a struggle with you and one and other, over the implementation of restriction on beef producers.  Premise ID, animal identification, age verification.... all being pushed upon our ranchers by the bureaucrats and politicians; all the while they tell us it is "industry lead".  Thats some real BS.Mark Purdey, by comparison to the other prion researchers, is a bloody genius!  Nobody..... absolutely nobody else, took the time to travel to all the TSE clusters in the world and examine the environmental conditions and test the soils, waters, tissues, vegetation... for metal abnormalities.  His book, "Animal Pharm" is a must read.  As, are his 14 peer-reviewed papers and many articles, available on his websites and from other websites, like Acres USA.

Once our respective governments have us all signed up for our Premise ID numbers, and registering all the animals for birth dates, etc..... they will very likely implement a mandatory testing of all animals over 30 months of age. Then things could get quite interesting and dirty.

CFIA will have all they need to quarantine 'premises'; putting ranchers and land out of the cattle business. After Alberta implements their mandatory testing for over 30 months, the pressure will be on the rest of Canada and the USA to follow suit.

Meanwhile, everyone will be put off of eating beef and we will be crying for more handouts. When all is said and done, and testing becomes a mandatory practice across the "North American Union".... only a handful of very large multinational companies will be able to afford the privilege of raising cattle. We can either co-operate or leave the business, just as the Alberta Agriculture Minister stated (June 5, 2008) when he announced the Alberta Livestock and Meat Strategy.

Once testing is implemented and we begin to see a clear picture of what source is the likely cause of most of the TSEs across this continent (aka the military industrial complex's use of, and manufacture of, toxic depleted uranium weapons and nuclear explosives, chemical weapons et al), it will be too late to lay the blame at their feet and demand restitution. Most ranchers will have given up the fight by then.

Therefore, the time to fight this BS is now! Their feed story is crap. Start looking at the environmental conditions of these clusters and don't let our secretive and corrupt governments pull the wool over our eyes.

Fool us once = shame on you. Fool us twice = shame on us!

Remember this also, if their 'story' about the feed bans proves incorrect (which it has), then their 'stories' about vCJD being caused by consuming meat may also be very incorrect. Just as Dr. David Brown, and Mark Purdey (as well as others) have stated.... it is more likely that the humans have been exposed to the same environmental toxins as the cattle were, and these disease are occurring separately, without any correlation to one and other via "consumption" of beef.
 
Alberta's latest mad-cow case shakes U.S. cattle futures

Bloomberg
Published: 2:02 am
American cattle futures fell to the lowest price this month on speculation that export demand for U.S. beef will slow after Canada's discovery of its 14th case of mad cow disease since 2003.

Last Friday, Canada confirmed the brain-wasting disease in a six-year-old beef cow from Alberta. The U.S. imported 110,319 head of cattle from Canada in June, up 84 per cent from the same month last year, government data show. Cattle futures gained 15 per cent in the past year on increased shipments of U.S. beef to Asia as economic growth spurs meat consumption.

"There's a little concern long-term that some Pacific Rim countries may associate Canada with us," said Lane Broadbent, a vice-president of KIS Futures Inc. in Oklahoma City. "We're closely associated as a trading partner and we're bringing in some cattle from Canada as well." Cattle for October delivery fell 1.125 cent, or 1.1 per cent, to $1.04825 a pound on the Chicago Mercantile Exchange, after earlier reaching $1.047, the lowest for a most-active contract since July 29. The price still is up 9.2 per cent this year.

The Canadian animal diagnosed with mad cow disease, also known as bovine spongiform encephalopathy, or BSE, was born years after Canada and the U.S. banned the use of feed containing ground-up cattle tissue, the government said.

Japan, formerly the largest buyer of U.S. beef, and South Korea, the third-biggest purchaser, along with 60 other nations banned imports in 2003 after the U.S. found its first case in December of that year. Most countries, including the two Asian nations, have since resumed imports on a limited basis.

http://www.canada.com/edmontonjournal/news/business/story.html?id=a72e321f-b557-40b7-9d13-7c0052fb14a5
 
Feeder cattle prices are stronger here this week than they have been since last September. Don't think that would be happening if the concern for BSE was strong.
 
"There's a little concern long-term that some Pacific Rim countries may associate Canada with us," said Lane Broadbent, a vice-president of KIS Futures Inc. in Oklahoma City. "We're closely associated as a trading partner and we're bringing in some cattle from Canada as well." Cattle for October delivery fell 1.125 cent, or 1.1 per cent, to $1.04825 a pound on the Chicago Mercantile Exchange, after earlier reaching $1.047, the lowest for a most-active contract since July 29. The price still is up 9.2 per cent this year.

I'm sure they do associate Canada with the US, what they are thinking is that the US has the same problems, the difference is that Canada is being open and honest with BSE.

"the incidence rate is probably the same, why is Canada finding them and the US not finding them?, OT, (the average caring citizen) is saying the US is being dishonest, and hiding the truth about food safety,, but preaching that their product is safer, and finding a scapegoat in foreign product.?" I'd think it was a little hypocritical also.

Who do you trust? That's the $1000 question.
 
You guys need to get your storys straight. You claim the positives came from isolated incidents where a simple mistake was made, and then you claim the incidence rate between the US and Canada is the same. That doesn't make any sense.
 
Now Sandy, you know the answer to that. The "experts" still don't know for sure the where,what, why, hows, but at least they are fumbling around and actually finding the odd one.
If you boys are worried about importing some maybe you could talk the USDA into running a few more tests down there.
 
Kathy said:
BSE tester, the game has never ended, to there is no restarting!  For all those that feel BSE is a non-issue in North America and lives, WAKE UP.Your federal, state, and provincial governments are in a struggle with you and one and other, over the implementation of restriction on beef producers.  Premise ID, animal identification, age verification.... all being pushed upon our ranchers by the bureaucrats and politicians; all the while they tell us it is "industry lead".  Thats some real BS.Mark Purdey, by comparison to the other prion researchers, is a bloody genius!  Nobody..... absolutely nobody else, took the time to travel to all the TSE clusters in the world and examine the environmental conditions and test the soils, waters, tissues, vegetation... for metal abnormalities.  His book, "Animal Pharm" is a must read.  As, are his 14 peer-reviewed papers and many articles, available on his websites and from other websites, like Acres USA.

Once our respective governments have us all signed up for our Premise ID numbers, and registering all the animals for birth dates, etc..... they will very likely implement a mandatory testing of all animals over 30 months of age. Then things could get quite interesting and dirty.

CFIA will have all they need to quarantine 'premises'; putting ranchers and land out of the cattle business. After Alberta implements their mandatory testing for over 30 months, the pressure will be on the rest of Canada and the USA to follow suit.

Meanwhile, everyone will be put off of eating beef and we will be crying for more handouts. When all is said and done, and testing becomes a mandatory practice across the "North American Union".... only a handful of very large multinational companies will be able to afford the privilege of raising cattle. We can either co-operate or leave the business, just as the Alberta Agriculture Minister stated (June 5, 2008) when he announced the Alberta Livestock and Meat Strategy.

Once testing is implemented and we begin to see a clear picture of what source is the likely cause of most of the TSEs across this continent (aka the military industrial complex's use of, and manufacture of, toxic depleted uranium weapons and nuclear explosives, chemical weapons et al), it will be too late to lay the blame at their feet and demand restitution. Most ranchers will have given up the fight by then.

Therefore, the time to fight this BS is now! Their feed story is crap. Start looking at the environmental conditions of these clusters and don't let our secretive and corrupt governments pull the wool over our eyes.

Fool us once = shame on you. Fool us twice = shame on us!

Remember this also, if their 'story' about the feed bans proves incorrect (which it has), then their 'stories' about vCJD being caused by consuming meat may also be very incorrect. Just as Dr. David Brown, and Mark Purdey (as well as others) have stated.... it is more likely that the humans have been exposed to the same environmental toxins as the cattle were, and these disease are occurring separately, without any correlation to one and other via "consumption" of beef.


shame on you kathy, you have been fooled more than twice with your old non-sense of nuclear proliferation or metals of any kind _causing_ any TSE. this theory was proven wrong long ago.


for Gods sake kathy, for once will you use some logic. or is that too much?
the tainted feed to bse is not a theory. it's fact. transmission studies do not lie. look at the reduction in cases of BSE in the UK and other countries that have been following the feed ban. you cannot argue this. oh, some can, but with no facts to back them up. also, in fact, with the atypical cases, you may in fact see this feed ban and the srms enhanced.



Estimation of the basic reproduction number of BSE: the intensity of transmission in British cattle Issue Volume 266, Number 1414/January 7, 1999 Pages 23-32 DOI 10.1098/rspb.1999.0599 Add to marked items Add to saved items Recommend this article

PDF (222.8 KB) Authors N. M. Ferguson, C. A. Donnelly, M. E. J. Woolhouse, R. M. Anderson

Abstract The basic reproduction number, R0, of an infectious agent is a key factor determining the rate of spread and the proportion of the host population affected. We formulate a general mathematical framework to describe the transmission dynamics of long incubation period diseases with complex pathogenesis. This is used to derive expressions for R0 of bovine spongiform encephalopathy (BSE) in British cattle, and backcalculation methods are used to estimate R0 throughout the time-course of the BSE epidemic. We show that the 1988 meat and bonemeal ban was effective in rapidly reducing R0 below 1, and demonstrate that this indicates that BSE will be unable to become endemic in the UK cattle population even when case clustering is taken into account. The analysis provides some insight into absolute infectiousness for bovine-to-bovine transmission, indicating maximally infectious animals may have infected up to 400 animals each. The relationship between R0 and the early stages of the BSE epidemic and the requirements for additional research are also discussed.


http://journals.royalsociety.org/content/ld82xpl8q9pkbne9/


and look at all the exotics in zoos that died from a TSE, these animals were fed split beef heads from BSE infected cattle for Pete's sake.


The 82 zoo animals with BSE:

Id TSE Genus Species Subsp Birth Origin Death Place of Death
654 x Microcebus murinus - 1997 U.Montpellier 1998 U.Montpellier
656 x Microcebus murinus - 1997 U.Montpellier 1998 U.Montpellier
481 + Eulemur fulvus mayottensis 1974 Madagascar 1992 Montpellier zoo
474 + Eulemur fulvus mayottensis 1974 Madagascar 1990 Montpellier zoo
584 - Eulemur fulvus mayottensis 1984 Montpellier 1991 Montpellier zoo
455 + Eulemur fulvus mayottensis 1983 Montpellier 1989 Montpellier zoo
- + Eulemur fulvus mayottensis 1988 Montpellier 1992 Montpellier zoo
- + Eulemur fulvus mayottensis 1995 Montpellier 1996 Montpellier zoo
- + Eulemur fulvus albifrons 1988 Paris 1992 Montpellier zoo
- + Eulemur fulvus albifrons 1988 Paris 1990 Montpellier zoo
- + Eulemur fulvus albifrons 1988 Paris 1992 Montpellier zoo
456 + Eulemur fulvus albifrons 1988 Paris 1990 Montpellier zoo
586 + Eulemur mongoz - 1979 Madagascar 1998 Montpellier zoo
- p Eulemur mongoz - 1989 Mulhouse 1991 Montpellier zoo
- p Eulemur mongoz - 1989 Mulhouse 1990 Montpellier zoo
- p Eulemur macaco - 1986 Montpellier 1996 Montpellier zoo
- p Lemur catta - 1976 Montpellier 1994 Montpellier zoo
- p Varecia variegata variegata 1985 Mulhouse 1990 Montpellier zoo
- p Varecia variegata variegata 1993 xxx 1994 Montpellier zoo
455 + Macaca mulatta - 1986 Ravensden UK 1992 Montpellier zoo
- p Macaca mulatta - 1986 Ravensden UK 1993 Montpellier zoo
- p Macaca mulatta - 1988 Ravensden UK 1991 Montpellier zoo
- p Saimiri sciureus - 1987 Frejus France 1990 Frejus zoo
700 pc eulemur hybrid - - Besancon zoo 1998 Besancon zoo
701 pc eulemur hybrid - - Besancon zoo 1998 Besancon zoo
702 pc eulemur hybrid - - Besancon zoo 1998 Besancon zoo
703 pc eulemur hybrid - - Besancon zoo 1998 Besancon zoo
704 pc eulemur hybrid - - Besancon zoo 1998 Besancon zoo
705 pc eulemur hybrid - - Besancon zoo 1998 Besancon zoo
706 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
707 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
708 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
709 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
710 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
711 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
712 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
713 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
714 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
715 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
716 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
717 pc eulemur hybrid - - Strasbourg zoo 1998 Strasbourg zoo
x p genus species - - Lille zoo 1996 Lille zoo
y p genus species - - Lille zoo 1996 Lille zoo
z p genus species - - Lille zoo 1996 Lille zoo


1 + Actinonyx jubatus cheetah 1986 Marwell zoo 1991 Pearle Coast AU
Duke + Actinonyx jubatus cheetah 1984 Marwell zoo 1992 Colchester zoo? UK
Saki + Actinonyx jubatus cheetah 1986 Marwell zoo 1993 unknown UK
Mich + Actinonyx jubatus cheetah 1986 Whipsnade 1993 Whipsnade UK
Fr1 + Actinonyx jubatus cheetah 1987 Whipsnade 1997 Safari de Peaugres FR
Fr2 + Actinonyx jubatus cheetah 1991 Marwell zoo 1997 Safari de Peaugres Fr
xx + Actinonyx jubatus cheetah 19xx xxx zoo 199x Fota zoo IR
yy + Actinonyx jubatus cheetah 19xx yyy zoo 1996+ yyyy zoo UK
zz + Actinonyx jubatus cheetah 19xx zzz zoo 1996+ yyyy zoo UK

aaa + Felis concolor puma 1986 Chester zoo 1991 Chester zoo UK
yy + Felis concolor puma 1980 yyy zoo 1995 yyyy zoo UK
zz + Felis concolor puma 1978 zzz zoo 1995 zzzz zoo UK

xxx + Felis pardalis ocelot 1987 xxx 1994 Chester zoo UK
zzz + Felis pardalis ocelot 1980 zzz 1995 zzzz zoo UK

85 + Felis catus cat 1990+ various 1999+ various UK LI NO
19 + Canis familia. dog 1992+ various 1999+ various UK

Fota + Panthera tigris tiger 1981 xxx zoo 1995 xxxx zoo UK
yy + Panthera tigris tiger 1983 yyy zoo 1998 yyyy zoo UK

Lump + Panthera leo lion 1986 Woburn SP 1998 Edinburgh zoo UK [since 1994]

1 + Taurotragus oryx eland 1987 Port Lympne 1989 Port Lympne zoo UK
Moll + Taurotragus oryx eland 1989 xx UK 1991 not Port Lympne UK
Nedd + Taurotragus oryx eland 1989 xx UK 1991 not Port Lympne UK
Elec + Taurotragus oryx eland 1990 xx UK 1992 not Port Lympne Uk
Daph p Taurotragus oryx eland 1988 xx UK 1990 not Port Lympne UK
zzz + Taurotragus oryx eland 1991 zz UK 1994 zzz UK
yyy + Taurotragus oryx eland 1993 yy UK 1995 yyy UK


Fran p Tragelaphus strepsi. kudu 1985 London zoo 1987 London zoo UK
Lind + Tragelaphus strepsi. kudu 1987 London zoo 1989 London zoo UK
Karl + Tragelaphus strepsi. kudu 1988 London zoo 1990 London zoo UK
Kaz + Tragelaphus strepsi. kudu 1988 London zoo 1991 London zoo UK
Bamb pc Tragelaphus strepsi. kudu 1988 London zoo 1991 London zoo UK
Step - Tragelaphus strepsi. kudu 1984 London zoo 1991 London zoo UK
346 pc Tragelaphus strepsi. kudu 1990 London zoo 1992 London zoo UK
324 + Tragelaphus strepsi. kudu 1989 Marwell zoo 1992 London zoo UK

xxx + Tragelaphus angasi nyala 1983 Marwell zoo 1986 Marwell zoo UK

yy + Oryx gazella gemsbok 1983 Marwell zoo 1986 Marwell zoo UK
zz + Oryx gazella gemsbok 1994+ zzz zoo 1996+ zzzz zoo UK

xx + Oryx dammah scim oryx 1990 xxxx zoo 1993 Chester zoo UK

yy + Oryx leucoryx arab oryx 1986 Zurich zoo 1991 London zoo UK

yy + Bos taurus ankole cow 1987 yyy zoo 1995 yyyy zoo UK
zz + Bos taurus ankole cow 1986 zzz zoo 1991 zzzz zoo UK

xx + Bison bison Eu bison 1989 xxx zoo 1996 xxxx zoo UK


http://www.mad-cow.org/zoo_cites_annotated.html



How many TSE deaths these zoos have had, how many are silently incubating the disease, and whether BSE in exported primates transmits horizontally as efficiently as CWD are the questions of the hour. A close reading of the PNAS study gives support to a truly catastrophic scenario.

Number of primates by zoo for 8/56 genera
France
114 Mulhouse
67 La Palmyr
54 Romagne
50 Fontaine
50 Beauval
42 Lille Zo
35 Besancon
34 Peaugres
31 Paris Zoo
15 Thoiry
14 Lisieux Z
13 Amiens
13 La Plaine
12 Paris Jp
8 Touroparc
3 Obterre

UK
94 Twycross
83 Banham
52 Colchestr
48 London Rp
47 Jersey
37 Marwell
37 Edinburgh
33 Chester
30 Chard
26 Dudley
24 Burford
24 Blackpool
22 Paignton
20 Bristol
15 Southport
15 Battersea
13 Bekesbrne
11 Chesingtn
9 So Lakes
8 Lympne
8 Alfriston
5 Colwynbay
4 Whipsnade



Breakdown by number of animals by country for each genus and zoo


snip...see full text ;


http://www.mad-cow.org/mar99_mid_sci.html#wh


The BSE Inquiry / Statement No 324
Dr James Kirkwood
(not scheduled to give oral evidence)
Statement to the BSE Inquiry
James K Kirkwood BVSc PhD FIBiol MRCVS
[This witness has not been asked to give oral evidence in Phase 1 of the Inquiry]
1. I became involved in the field of TSEs through my work as Head of the Veterinary
Science Group at the Zoological Society of London's Institute of Zoology. I held this post from
November 1984 until June 1996, when I took up my present post at UFAW. During this time,
concurrent with the BSE epidemic, cases of scrapie-like spongiform encephalopathies occurred
in animals at the Zoological Society of London's collections at Regent's Park and Whipsnade
and in other zoos. It was appropriate to investigate the epidemiology of these cases in order to try
to determine the possible impact on zoo animals and breeding programmes, and to consider how
the disease in zoo animals might be controlled.
2. Throughout the period from 1985 to March 1996, I worked at the Institute of Zoology
(IoZ). I was Head of the Veterinary Science Group of the IoZ and Senior Veterinary Officer of
the Zoological Society of London (ZSL). I was responsible for the provision of the veterinary
service for the ZSL collections.
3. During the period from 1985 to March 1996, scrapie-like spongiform encephalopathies
were diagnosed in the following animals which died, or were euthanased, at London Zoo and
Whipsnade:
Animal Sex Date of Death Age (mos)
Arabian Oryx Oryx leucoryx F 24.3.89 38
Greater kudu Tragelaphus strepsiceros (Linda) F 18.8.89 30
Greater kudu (Karla) F 13.11.90 19
Greater kudu (Kaz) M 6.6.91 37
Greater kudu (Bambi) M 24.10.91 36
Greater kudu (346/90) M 26.2.92 18
Greater kudu (324/90) F 22.11.92 38
Cheetah Acinonyx jubatus (Michelle) F 22.12.93 91
All these cases were described in papers published in the scientific literature (as cited below).

snip...

10. The case of SE in a cheetah that occurred during the period, involved a 7 year-old female which had been born and lived all her life at Whipsnade (except for the final stages when she was moved to the Animal Hospital at Regent's Park for diagnosis and treatment). This animal, which died in December 1993, had been fed on cuts of meat and bone from carcases of cattle unfit for human consumption and it was thought likely that she had been exposed to spinal cord (Kirkwood, J.K., Cunningham, A.A., Flach, E.J., Thornton, S.M. & Wells, G.A.H. (1995) Spongiform encephalopathy in another captive cheetah (Acinonyx jubatus): evidence for variation in susceptibility or incubation periods between species. Journal of Zoo and Wildlife Medicine 26, 577-582: J/ZWM/26/577). 11. During the period we also collated information on cases of SE that occurred in wild animals at or from other zoos in the British Isles. The total number of cases of which I was aware in June 1996, when I presented a review on occurrence of spongiform encephalopathies in zoo animals (at the Royal College of Pathologists' Symposium on Transmitting prions: BSE, CJD, and other TSEs, The Royal Society, London, 4th July 1996), was 25, involving 10 species. The animals involved were all from the families Bovidae and Felidae, and comprised: 1 Nyala Tragelaphus angasi, 5 Eland Taurotragus oryx, 6 greater kudu Tragelaphus strepsiceros, 1 Gemsbok Oryx gazella, 1 Arabian oryx Oryx leucoryx, 1 Scimitar-horned oryx Oryx dammah, 4 Cheetah Acinonyx jubatus, 3 Puma Felis concolor 2 Ocelot Felis pardalis, and 1 Tiger Panthera tigris. (A spongiform encephalopathy, which was thought probably to have a different aetiology, had also been reported in 3 ostriches Struthio camelus in Germany). This list did not include cases of BSE in domesticated species in zoos (ie BSE in Ankole or other cattle, or SEs, assumed to be scrapie, in mouflon sheep Ovis musimon). 12. Since the time the above statistics were published, a few further cases have occurred in animals at or from zoos in the British Isles. The total number of cases in cheetah that have now been documented has, as far as I am aware, risen to seven (Vitaud, C., Flach, E.J., Thornton, S.M. & Capello, R. (1998) Clinical observations on four cases of feline spongiform encephalopathy in cheetahs (Acinonyx jubatus). Proceedings of the European Association of Zoo and Wildlife Veterinarians, Chester, UK, 21st-24th May 1998. Pp 133-138). There has also been a case in a bison. 13. Epidemiological aspects of the majority of these cases (those diagnosed up to the end of 1993) were considered in paper published in 1994 (Kirkwood, J.K. & Cunningham, A.A. (1994) Epidemiological observations on spongiform encephalopathies in captive wild animals in the British Isles. Veterinary Record 135, 296-303:J/VR/135/296.) This paper was based on a paper presented at the Consultation on BSE with the Scientific Veterinary Committee of the Commission of the European Communities held in Brussels, 14-15th September 1993 (Kirkwood, J.K. & Cunningham, A.A. (1993) Spongiform encephalopathy in captive wild animals in Britain: epidemiological observations. In R. Bradley & B Marchant (Eds) Transmissible spongiform encephalopathies. Proceedings of a Consultation on BSE with the Scientific Veterinary Committee of the Commission of the European Communities, 14-15 September 1993, Brussels. European Commission. Pp 29-47:M9 tab 46). It was thought likely that at least some, and probably all, of the cases in zoo animals were caused by the BSE agent. Strong support for this hypothesis came from the findings of Bruce and others (1994) ( Bruce, M.E., Chree, A., McConnell, I., Foster, J., Pearson, G. & Fraser, H. (1994) Transmission of bovine spongiform encephalopathy and scrapie to mice: strain variation and species barrier. Philosophical Transactions of the Royal Society B 343, 405-411: J/PTRSL/343/405), who demonstrated that the pattern of variation in incubation period and lesion profile in six strains of mice inoculated with brain homogenates from an affected kudu and the nyala, was similar to that seen when this panel of mouse strains was inoculated with brain from cattle with BSE. The affected zoo bovids were all from herds that were exposed to feeds that were likely to have contained contaminated ruminant-derived protein and the zoo felids had been exposed, if only occasionally in some cases, to tissues from cattle unfit for human consumption. 14. Among the affected bovids were others (including scimitar horned oryx and eland) which, like some of the kudu, were born some considerable time after the July 1988 ban on inclusion of RDP in ruminant feeds (Kirkwood, J.K. & Cunningham, A.A. (1994) Epidemiological observations on spongiform encephalopathies in captive wild animals in the British Isles. Veterinary Record 135, 296-303:J/VR/135/296). The source of infection to these animals was puzzling. However, as it emerged that many cases of BSE were continuing to occur in domestic cattle born after the July 1988 ban on inclusion of RDP in ruminant feeds, it was clear that the ban had not been immediately effective, and it was therefore possible (or, at least, impossible to rule out) that the late cases in zoo ungulates were also due to exposure to contaminated feeds. 15. We drew attention to the fact that, from a taxonomic perspective, the incidence of cases was strikingly patchy (Kirkwood, J.K. & Cunningham, A.A. (1994) Epidemiological observations on spongiform encephalopathies in captive wild animals in the British Isles. Veterinary Record 135, 296-303. Also Kirkwood, J.K., Cunningham, A.A., Flach, E.J., Thornton, S.M. & Wells, G.A.H. (1995) Spongiform encephalopathy in another captive cheetah (Acinonyx jubatus): evidence for variation in susceptibility or incubation periods between species? Journal of Zoo and Wildlife Medicine 26, 577-582) Compared with many other species of exotic ruminants, few kudu were present in the UK but there had been 6 cases of SE among them. The picture seemed similar in the felids. Compared with other species of exotic felids (eg lions in which no cases had occurred), there were relatively small numbers of puma and cheetah in the UK but (at that time) there had been 3 and 4 cases among these respectively. Almost certainly a wider range of species were exposed to contaminated feeds than those in which cases have occurred or been detected. However, we were cautious about drawing firm conclusions about variation in susceptibility between species because (i) incubation periods vary between species and we thought other cases may emerge and (ii) because the variation might be related to differences in intensity of exposure.

16. The number of cases of SE each year in zoo hoofed-stock reached a peak around 1991 and has declined since as shown in the figure below (this and the subsequent figure are updates of those I showed at the Royal College of Pathologist's Meeting on TSEs at the Royal Society on 4th July 1996). This pattern suggests that the ban on including RDP in ruminant feeds has been an important factor in the control of the disease in zoo bovids.

snip...

Since it was considered from all the evidence available, to be very unlikely that the feed offered to the animals from 1988 onwards could contain RDP, the pattern of incidence in the group between 1989 and 1992 suggested that transmission between animals might have occurred. However, in 1995, it became clear from further discussions with the manufacturers that the possibility that the pelleted feed could have contained RDP for some years after the July 1988 ban, could not be entirely ruled out.


http://www.bseinquiry.gov.uk/files/ws/s324.pdf


TSEs in Exotic Ruminants
TSEs have been detected in exotic ruminants in UK zoos since 1986. These include antelopes (Eland, Gemsbok, Arabian and Scimitar oryx, Nyala and Kudu), Ankole cattle and Bison. With hindsight the 1986 case in a Nyala was diagnosed before the first case of BSE was identified. The TSE cases in exotic ruminants had a younger onset age and a shorter clinical duration compared to that in cattle with BSE. All the cases appear to be linked to the BSE epidemic via the consumption of feed contaminated with the BSE agent. The epidemic has declined as a result of tight controls on feeding mammalian meat and bone meal to susceptible animals, particularly from August 1996.

References:
Jeffrey, M. and Wells, G.A.H, (1988) Spongiform encephalopathy in a nyala (Tragelaphus angasi). Vet.Path. 25. 398-399

Kirkwood, J.K. et al (1990) Spongiform encephalopathy in an Arabian oryx (Oryx leucoryx) and a Greater kudu (Tragelaphus strepsiceros) Veterinary Record 127. 418-429.

Kirkwood, J.K. (1993) Spongiform encephalopathy in a herd of Greater kudu (Tragelaphus strepsiceros): epidemiological observations. Veterinary Record 133. 360-364

Kirkwood, J. K. and Cunningham, A.A. (1994) Epidemiological observations on spongiform encephalopathies in captive wild animals in the British Isles. Veterinary Record. 135. 296-303.

Food and Agriculture Organisation (1998) Manual on Bovine Spongiform Encephalopathy.

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Page last modified: 15 August, 2008
Page last reviewed: 15 August, 2008


http://www.defra.gov.uk/animalh/bse/othertses/index.html


worse still, there is serious risk the media could get
to hear of such a meeting...

snip...

Crushed heads (which inevitably involve brain and spinal cord material)
are used to a limited extent but will also form one of the constituent
raw materials of meat and bone meal, which is used extensively in
pet food manufacturer...

http://www.bseinquiry.gov.uk/files/yb/1989/03/17004001.pdf

2. The Parliamentary Secretary said that he was concerned
about the possibility that countries in which BSE had not
yet been detected could be exporting raw meat materials
(in particular crushed heads) contaminated with the disease
to the UK for use in petfood manufacture...

snip...

YOU explained that imported crushed heads were extensively used in the
petfood industry...

http://www.bseinquiry.gov.uk/files/yb/1989/04/14001001.pdf

In particular I do not believe one can say that the levels of
the scrapie agent in pet food are so low that domestic animals are
not exposed...

http://www.bseinquiry.gov.uk/files/yb/1989/04/24003001.pdf

http://www.bseinquiry.gov.uk/files/yb/1989/04/25001001.pdf

some 100+ _documented_ TSE cats of all types later...tss

on occassions, materials obtained from slaughterhouses
will be derived from sheep affected with scrapie or
cattle that may be incubating BSE for use in petfood manufacture...

http://www.bseinquiry.gov.uk/files/yb/1989/05/03007001.pdf

Meldrum's notes on pet foods and materials used

http://www.bseinquiry.gov.uk/files/yb/1989/05/16001001.pdf

http://www.bseinquiry.gov.uk/files/yb/1989/05/16002001.pdf

IN CONFIDENCE CJD TO CATS...

http://www.bseinquiry.gov.uk/files/yb/1989/05/18002001.pdf

Confidential BSE and __________________

http://www.bseinquiry.gov.uk/files/yb/1989/05/22012001.pdf

1st case natural FSE

http://www.bseinquiry.gov.uk/files/yb/1990/05/09002001.pdf

FSE and pharmaceuticals

http://www.bseinquiry.gov.uk/files/yb/1990/05/10005001.pdf

confidential cats/dogs and unsatisfactory posture
MAFFs failure to assure key research

http://www.bseinquiry.gov.uk/files/yb/1990/06/14006001.pdf


and when you have the feed industry itself banning together thick as thieves, to continue to use all the tainted feed in commerce ;

STRICTLY PRIVATE AND CONFIDENTIAL 25, AUGUST 1995

snip...

To minimise the risk of farmers' claims for compensation from feed compounders.

To minimise the potential damage to compound feed markets through adverse publicity.

To maximise freedom of action for feed compounders, notably by maintaining the availability of meat and bone meal as a raw material in animal feeds, and ensuring time is available to make any changes which may be required.

snip...

THE FUTURE

4..........

MAFF remains under pressure in Brussels and is not skilled at handling potentially explosive issues.

5. Tests _may_ show that ruminant feeds have been sold which contain illegal traces of ruminant protein. More likely, a few positive test results will turn up but proof that a particular feed mill knowingly supplied it to a particular farm will be difficult if not impossible.

6. The threat remains real and it will be some years before feed compounders are free of it. The longer we can avoid any direct linkage between feed milling _practices_ and actual BSE cases, the more likely it is that serious damage can be avoided. ...

SEE full text ;

http://www.bseinquiry.gov.uk/files/yb/1995/08/24002001.pdf


THIS is what happens when you have the industry run the government.
It's a very deadly, leathal mix $$$


kathy dreams of this as fact, kathy wrote ;


>>>Once testing is implemented and we begin to see a clear picture of what source is the likely cause of most of the TSEs across this continent (aka the military industrial complex's use of, and manufacture of, toxic depleted uranium weapons and nuclear explosives, chemical weapons et al), it will be too late to lay the blame at their feet and demand restitution. Most ranchers will have given up the fight by then.

Therefore, the time to fight this BS is now! Their feed story is crap. <<<


NO, you are wrong again kathy, the story that is 'crap', is yours. and it has been proven to be just that, by science, time and time again. ...


kind regards,
terry
 

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