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China reopens border to Canadian beef

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China reopens border to Canadian beef



By Philip Ling , Canwest News Service - Calgary Herald - June 24, 2010



OTTAWA — China will resume importing Canadian beef in a "staged approach," Prime Minister Stephen Harper announced on Thursday night following bilateral discussions with Chinese President Hu Jintao.



Canada is the first country affected by Bovine Spongiform Encephalopathy (BSE), commonly known as mad cow disease, to regain access to the Chinese market for beef.



The first step in this process is access for boneless beef derived from animals less than 30 months old, as well as beef tallow for industrial use.



The two leaders signed the agreement during the first full day of the president's visit to Canada, a tour that will climax Saturday when the Chinese leader participates in the G20 summit being hosted by Harper in Toronto.



"This visit by President Hu confirms that the Canada-China strategic partnership is gaining momentum as we mark 40 years of diplomatic relations and chart a course for the future," Harper said in a release.



The announcement is "very welcome news" for the Canada Beef Export Federation, which has long advocated for the reopening of the Chinese border to Canadian beef since the country's first case of BSE in 2003.



Federation president Ted Haney credits the strengthening of political relationships between Harper and Hu for Thursday's announcement.



"They've liked each other, but they need to love each other," he said referring to Chinese trade negotiations. "I guess this is a sure sign of a little love coming into the relationship."



Haney said Canada exported about $2.2 billion worth of processed beef products to world markets pre-BSE. The value of beef exports in 2009 reached $1.3 billion — or 59 per cent of where they stood in 2003, prior to BSE being found in Canada.



The Chinese market for Canadian beef and tallow is expected to be worth $110 million once full market access is achieved, significantly benefiting the beef industry, he said.



"Between 250,000 to 300,000 cattle will now be raised just to service this mainland China opportunity," Haney said. "This will help our industry move from our current survival mode to one of stability and then growth."



While Haney would not provide an estimate as to when Canadian beef exports to China will surpass the pre-BSE levels in terms of value, he said: "We will be in a much better position in China within one year than where we are today."



Only one market — South Korea — still imposes an outright ban on Canadian beef products.



calgaryherald.com
Click to expand...
 
Just think, it only took 7 years :roll:

Hopefully we see some benefit at the producer level in the next year or so. Any good news is welcome.
 
gcreekrch said:
I would think R-Quack would be applauding this agreement. :wink:
Click to expand...

Good for you guys. A buyer and a seller reached a mutual agreement without any threats or grandstanding - the way it should be.
 
Sandhusker said:
gcreekrch said:
I would think R-Quack would be applauding this agreement. :wink:
Click to expand...

Good for you guys. A buyer and a seller reached a mutual agreement without any threats or grandstanding -the way it should be.
Click to expand...

But you upsetthe NCBAer's (what few of them that are left :wink: )....

China opens door to Canadian beef; NCBA not happy

June 25, 2010 by Ken Anderson - Brownfield

China has lifted a ban on imports of Canadian beef, making Canadathe first among countries affected by BSE to regain market access to that country. And that's not sitting well withthe National Cattlemen's Beef Association (NCBA).[/b]

NCBA president Steve Fogelsong blames boththe Bush and Obama administrations for failing to aggressively addressthe issue with China. In his words, "China is thwarting us almost acrossthe board on animal agriculture trade issues, whilethe level of U.S. engagement has been poor at best."

Fogelsong also criticized Congress for what he calls its "do as we say, not as we do" attitude when it comes to science-based trade protocols. He citesthe case ofthe cooked poultry issue with China, saying those types of actions are hurtingthe United States' reputation with international trading partners.

Fogelson says it's "absolutely critical" to expand opportunities to sell beef inthe international marketplace in order to keep American family farms in business. brownfieldagnews.com
Click to expand...
 
So the answer is to strong arm China rather than ask them why they accepted Canadian beef and not American. :roll: :roll: :roll:

Quiz>>>

Can anyone here see why they would???? Think hard. There must be a reason.......
 
Kato said:
So the answer is to strong arm China rather than ask them why they accepted Canadian beef and not American. :roll: :roll: :roll:

Quiz>>>

Can anyone here see why they would???? Think hard. There must be a reason.......
Click to expand...

Well let's start by the process of elimination.

1) It can't be the rcalf influence because they've always supported a fair and open border position . . .

:lol: :lol: :lol: :lol:

2) The US cattle industry is world renowned for its open, broad and accurate BSE testing protocols . . .

:lol: :lol: :lol: :lol:

3) The US has THE BEST feed ban in the industry . . .

:lol: :lol: :lol: :lol:
 
U.S. Beef Exports May Jump 20% on Rising Asian Demand



Whitney McFerron - Bloomberg Businessweek - June 29, 2010



U.S. beef exports may jump 20 percent to $3.7 billion this year, as demand for the meat increases in Asia, said Erin Daley, an economist with the Denver-based U.S. Meat Export Federation.



The volume of U.S. beef exports may increase by 12 percent to 1 million metric tons (2.2 billion pounds), Daley said today on a conference call with reporters. Shipments were valued at $3.08 billion last year.



Beef demand in Asia is increasing as countries recover from recession and ease trade restrictions imposed following the 2003 discovery of mad cow disease in the U.S., Daley said. South Korea, which resumed imports of some U.S. beef in June 2008, has boosted purchases by 86 percent this year from 2009, she said. Overall U.S. beef exports this year may still trail 2003 volume by 3 percent, she said.



"This year exports have been tremendous," Daley said. "Recovery in U.S. exports has continued to go back toward 2003 volumes."



The U.S. has lost about $14 billion since 2004, when countries including Korea and Japan curtailed U.S. beef imports after mad cow disease, or bovine spongiform encephalopathy, broke out in the U.S., according to the Meat Export Federation. Scientists say humans who eat certain parts of infected animals may contract variant Cruetzfeldt-Jakob disease, a similar brain- wasting ailment.



U.S. shipments of beef-muscle-cuts may increase 17 percent this year, while beef-variety-meat exports will be steady, Daley said. In the first four months of 2010, the U.S. exported 11 percent of its beef production.



Demand for U.S. beef also may increase as competitors including Argentina and Canada cut production, she said. Of the world's top 10 beef producers, only Brazil, India and Mexico are expanding output this year.



--Editors: Michael Arndt, Steve Stroth.



businessweek.com
Click to expand...
 
Kato said:
So the answer is to strong arm China rather than ask them why they accepted Canadian beef and not American. :roll: :roll: :roll:

Quiz>>>

Can anyone here see why they would???? Think hard. There must be a reason.......
Click to expand...





Can you say TOYOTA. IT is a sad day when trade trumps human and animal health. as the case with the BSE MRR policy. Behind closed doors, the BSe spin machine is working i.e. Vilsack saying that 'The U.S. has had no cases in the last three years, and only three in two decades.' i can tell you with absolute certainty, that is only part of the story. i can tell you that in fact, the USDA BSE surveillance and testing have failed the consumer here in the USA, and abroad, and that we have been exposed to the TSE agent, i.e. USA atypical BSE, which laboratory studies show is more virulent. i can tell you with absolute certainty they infamous June 2004 enhanced BSE surveillance program, where some 800,000+ cattle were tested over many years of testing, was fraught with fraud, and in short, a failed program, and proven to be so by the GAO and the OIG, where it was proven that some of the testing program was using perfectly healthy cattle in their BSE testing program. Where some 9,200+ BSE test on suspect questionable cattle, only the IHC test were used. THE IHC is the least likely test to find BSE. IT only tells you if that part of the tissue sample is in fact infected or not, but it does not tell you about the rest of the brain. By only using the IHC, you miss many cases (Detwiler et al 2003 BSE ROUNDTABLE). i can tell you with absolute certainty, that when pressed, the USDA et al will say that even if we are missing cases of BSE, that the BSE mad cow feed ban of August 4, 1997, will stop BSE, but the ban was nothing more than ink on paper. This mad cow feed ban was only partial and voluntary. i can tell you with absolute certainty that in 2010, since 8/4/97, banned mad cow feed is in commerce here in the USA, BY THE TONS. i can tell you with absolute certainty, that when the BSE MRR policy was put in place, that this exposed everyone around the globe with the TSE agent, either by consumption and or friendly fire there from, and that decision was based NOT on science, but on trade. i can tell you with absolute certainty, that SINCE the USDA and the NSLP did in fact expose our children across the Nation with BSE via the NSLP USDA DEAD STOCK DOWNER COW SCHOOL LUNCH PROGRAM, that if they are capable of this, they are capable of exposing any person, in any country with the BSE TSE agent. North America has documented the so called typical c-BSE, l-BSE, and h-BSE. typical scrapie strains are rampant in the USA in sheep and goats, and the atypical Nor-98 scrapie is spreading, and CWD in deer and elk is spreading, with now documented a 2nd strain, and two strains of TME in mink. all this over the years have been fed back to food producing animals for animals and humans. Confucius ask, IF USA sheep Scrapie transmitted to USA cattle does not produce the same pathology as the U.K. c-BSE, why then would human CJD there from look like the U.K. nvCJD ??? what the USA has done defies all science and logic i.e. NO MAD COW DISEASE and or any human TSE there from. Either the BSE Mad Cow outbreak and human infection nvCJD there from, that happened in the U.K. was totally false, or the same thing is happening in the USA as we speak. sCJD of unknown phenotypes are rising in the USA. sporadic CJD is not a single strain, but multiple strains of unknown routes and sources of the TSE agent. IF the federal government can lie for almost a century about asbestos, and or tobacco, just to protect those two industry giants, i can guarantee you that they are doing it with mad cow type disease i.e. Transmissible Spongiform Encephalopathy. or just ask the Indians. ...TSS

full text ;

Saturday, April 10, 2010

TOYOTA VS MAD COW DISEASE USA OIE BSE MRR IMPORT AND EXPORT TRADE WARS



http://usdameatexport.blogspot.com/2010/04/toyota-vs-mad-cow-disease-usa-oie-bse.html




When the OIE and the USDA et al collaborated to make legal the trading of Transmissible Spongiform Encephalopathy, when they did away with the BSE GBR risk assessments, where the USA, Canada, and Mexico were categorized as BSE GBR III. please see ;

EFSA concludes that the current GBR level of USA is III, i.e. it is likely but not confirmed that domestic cattle are (clinically or pre-clinically) infected with the BSE-agent. As long as there are no significant changes in rendering or feeding, the stability remains extremely/very unstable. Thus, the probability of cattle to be (pre-clinically or clinically) infected with the BSE-agent persistently increases.




http://www.efsa.europa.eu/EFSA/efsa_locale-1178620753812_1211902594180.htm




Annex to the EFSA Scientific Report (2004) 3, 1-17 on the Assessment of the Geographical BSE Risk of USA

please see full text ;




http://www.efsa.europa.eu/en/scdocs/doc/3rax1.pdf





YET, in 2010, tons and tons of banned mad cow protein are still in commerce here in the USA, scientific studies are being misconstrued and manipulated by ARS USDA, which are still going by TSE science that is decades old, while refusing to acknowledge new scientific studies, and FOIA requests are still being held up by the USDA et al on these urgent matters (see source related materials below). CJD of unknown phenotype, in victims that are getting younger, with longer clinical course from first onset of symptoms to death are occurring, in fact, sporadic CJD is still rising, where the TSEs in the different species are mutating here in the USA, and we still have this same dog and pony show by the OIE and USDA et al. IF you go back and look at the Countries that went by these OIE BSE guidelines, most all came down with BSE. I have said it before, I was say it again now, OIE should hang up there jock strap now, since it appears they will buckle every time a country makes some political hay about trade protocol, commodities and futures. IF they are not going to be science based, they should do everyone a favor and dissolve there organization. ...TSS



Saturday, June 19, 2010

U.S. DENIED UPGRADED BSE STATUS FROM OIE




http://usdameatexport.blogspot.com/2010/06/us-denied-upgraded-bse-status-from-oie.html





****************PLEASE READ THE FOLLOWING CAREFULLY************



To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.



http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2





14th International Congress on Infectious Diseases H-type and L-type Atypical BSE January 2010 (special pre-congress edition)

18.173 page 189

Experimental Challenge of Cattle with H-type and L-type Atypical BSE

A. Buschmann1, U. Ziegler1, M. Keller1, R. Rogers2, B. Hills3, M.H. Groschup1. 1Friedrich-Loeffler-Institut, Greifswald-Insel Riems, Germany, 2Health Canada, Bureau of Microbial Hazards, Health Products & Food Branch, Ottawa, Canada, 3Health Canada, Transmissible Spongiform Encephalopathy Secretariat, Ottawa, Canada

Background: After the detection of two novel BSE forms designated H-type and L-type atypical BSE the question of the pathogenesis and the agent distribution of these two types in cattle was fully open. From initial studies of the brain pathology, it was already known that the anatomical distribution of L-type BSE differs from that of the classical type where the obex region in the brainstem always displays the highest PrPSc concentrations. In contrast in L-type BSE cases, the thalamus and frontal cortex regions showed the highest levels of the pathological prion protein, while the obex region was only weakly involved.

Methods:We performed intracranial inoculations of cattle (five and six per group) using 10%brainstemhomogenates of the two German H- and L-type atypical BSE isolates. The animals were inoculated under narcosis and then kept in a free-ranging stable under appropriate biosafety conditions.At least one animal per group was killed and sectioned in the preclinical stage and the remaining animals were kept until they developed clinical symptoms. The animals were examined for behavioural changes every four weeks throughout the experiment following a protocol that had been established during earlier BSE pathogenesis studies with classical BSE.

Results and Discussion: All animals of both groups developed clinical symptoms and had to be euthanized within 16 months. The clinical picture differed from that of classical BSE, as the earliest signs of illness were loss of body weight and depression. However, the animals later developed hind limb ataxia and hyperesthesia predominantly and the head. Analysis of brain samples from these animals confirmed the BSE infection and the atypical Western blot profile was maintained in all animals. Samples from these animals are now being examined in order to be able to describe the pathogenesis and agent distribution for these novel BSE types. Conclusions: A pilot study using a commercially avaialble BSE rapid test ELISA revealed an essential restriction of PrPSc to the central nervous system for both atypical BSE forms. A much more detailed analysis for PrPSc and infectivity is still ongoing.



http://www.isid.org/14th_icid/

http://ww2.isid.org/Downloads/IMED2009_AbstrAuth.pdf

http://www.isid.org/publications/ICID_Archive.shtml



14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

see page 114 ;



http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf





International Society for Infectious Diseases Web: http://www.isid.org



I ask Professor Kong ;

Thursday, December 04, 2008 3:37 PM Subject: RE: re--Chronic Wating Disease (CWD) and Bovine Spongiform Encephalopathies (BSE): Public Health Risk Assessment

''IS the h-BSE more virulent than typical BSE as well, or the same as cBSE, or less virulent than cBSE? just curious.....''

Professor Kong reply ;


.....snip


''As to the H-BSE, we do not have sufficient data to say one way or another, but we have found that H-BSE can infect humans. I hope we could publish these data once the study is complete.

Thanks for your interest.''

Best regards,

Qingzhong Kong, PhD Associate Professor Department of Pathology Case Western Reserve University Cleveland, OH 44106 USA

END...TSS



P02.35

Molecular Features of the Protease-resistant Prion Protein (PrPres) in H-type BSE

Biacabe, A-G1; Jacobs, JG2; Gavier-Widén, D3; Vulin, J1; Langeveld, JPM2; Baron, TGM1 1AFSSA, France; 2CIDC-Lelystad, Netherlands; 3SVA, Sweden

Western blot analyses of PrPres accumulating in the brain of BSE-infected cattle have demonstrated 3 different molecular phenotypes regarding to the apparent molecular masses and glycoform ratios of PrPres bands. We initially described isolates (H-type BSE) essentially characterized by higher PrPres molecular mass and decreased levels of the diglycosylated PrPres band, in contrast to the classical type of BSE. This type is also distinct from another BSE phenotype named L-type BSE, or also BASE (for Bovine Amyloid Spongiform Encephalopathy), mainly characterized by a low representation of the diglycosylated PrPres band as well as a lower PrPres molecular mass. Retrospective molecular studies in France of all available BSE cases older than 8 years old and of part of the other cases identified since the beginning of the exhaustive surveillance of the disease in 20001 allowed to identify 7 H-type BSE cases, among 594 BSE cases that could be classified as classical, L- or H-type BSE. By Western blot analysis of H-type PrPres, we described a remarkable specific feature with antibodies raised against the C-terminal region of PrP that demonstrated the existence of a more C-terminal cleaved form of PrPres (named PrPres#2 ), in addition to the usual PrPres form (PrPres #1). In the unglycosylated form, PrPres #2 migrates at about 14 kDa, compared to 20 kDa for PrPres #1. The proportion of the PrPres#2 in cattle seems to by higher compared to the PrPres#1. Furthermore another PK–resistant fragment at about 7 kDa was detected by some more N-terminal antibodies and presumed to be the result of cleavages of both N- and C-terminal parts of PrP. These singular features were maintained after transmission of the disease to C57Bl/6 mice. The identification of these two additional PrPres fragments (PrPres #2 and 7kDa band) reminds features reported respectively in sporadic Creutzfeldt-Jakob disease and in Gerstmann-Sträussler-Scheinker (GSS) syndrome in humans.



http://www.neuroprion.com/pdf_docs/conferences/prion2007/abstract_book.pdf





Wednesday, March 31, 2010

Atypical BSE in Cattle


http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html





Thursday, June 24, 2010

Accumulation of L-type Bovine Prions in Peripheral Nerve Tissues

Volume 16, Number 7–July 2010


http://bse-atypical.blogspot.com/2010/06/accumulation-of-l-type-bovine-prions-in.html





******$$$$$$******


Saturday, June 12, 2010

PUBLICATION REQUEST AND FOIA REQUEST Project Number: 3625-32000-086-05 Study of Atypical Bse



http://bse-atypical.blogspot.com/2010/06/publication-request-and-foia-request.html





TSS
 
ALSO, COULD BE, maybe it had something to do with 23 million pounds of uranium concentrate that Canada plan to send to CHINA, in turn, China takes some of Canada's BSE risk cattle ? that would be a win, win for Canada. Course, we know that China cares less about what it feeds to their people, or people they export to.
OR maybe, it's like ''if China takes some of Canada's BSE, Canada will take more of China's melamine tainted products. i am telling you folks, it's all about trade now, and science has taken a back seat to the show $$$



Friday, June 25, 2010

China-Canada trade mission generates Cameco-China Nuclear Energy uranium deal

By Dorothy Kosich, Mineweb.com

During his visit to Canada this week ahead of the G20 meeting, Chinese President Hu Jintao brought with him more than 300 Chinese deals, especially in Canada's energy sector.

Top uranium miner Cameco announced Thursday it had signed an agreement with the China Nuclear Energy Industry Corporation (CNEIC), a subsidiary of China largest nuclear generator China National Nuclear Corporation (CNNC).

The agreement between Cameco and CNEIC calls for the Saskatchewan-based uranium miner to supply 23 million pounds of uranium concentrate under a long-term agreement through 2020.

"This is Cameco's first long-term supply agreement with a major Chinese nuclear utility," said Jerry Grandey, Cameco's CEO. "The agreement indicates clearly that we intend to be very active in the world's fastest-growing uranium market."

"The rapid expansion of nuclear capacity demonstrates the excellent prospects for nuclear energy and the solid underpinning of our plan to double uranium production by 2018," he added.

The agreement is subject to Chinese government approval.

President Hu said his country wanted to double its trade with Canada to $60 billion by 2015. Trade between the two nations totaled $29 billion last year.

Canadian energy company Encana and the China National Petroleum Corporation announced Thursday that they are negotiating a joint venture to develop Encana's oil shale-gas properties in northern British Columbia.

Chinese officials also reached a deal to open the Chinese market to Canadian beef.


http://proactiveinvestors.com/companies/news/6667/china-canada-trade-mission-generates-cameco-china-nuclear-energy-uranium-deal-6667.html




Canada's Merchandise Trade with China
9953908954 Canada's Trade with China (C$)
January - December January - March
2008 2009 2009 2010
Exports 10,084,234,904 10,933,193,228 2,830,666,402 2,545,056,665
Imports 42,623,483,767 39,656,943,226 9,953,908,954 9,109,746,525
Trade Balance -32,539,248,863 -28,723,749,998 -7,123,242,552 -6,564,689,860

Source: Trade Data Online. Industry Canada. 4 June 2010. http://www.ic.gc.ca/eic/site/tdo-dcd.nsf/eng/Home
Reproduced with the permission of the Minister of Public Works and Government Services Canada.

Canada's Merchandise Trade with China, 2009

Canadian Imports from China Canadian Exports to China

Merchandise Classification % Merchandise Classification
%

1 Electrical machinery and equipment 21.53 Oil seeds and misc. fruit, grain, etc. 14.33
2 Boilers, mechanical appliances, etc. 17.92 Woodpulp; paper or paperboard scraps 13.34
3 Toys, games, sports equipment 7.92 Ores, slag and ash 12.81
4 Furniture and stuffed furnishings 6.24 Mineral fuels, oils 8.01
5 Woven clothing and apparel articles 5.26 Boilers, mechanical appliances, etc. 7.78
6 Knitted or crocheted apparel 5.05 Nickel and nickel articles 6.38
7 Iron or steel articles 4.05 Fats, oils and waxes 3.68
8 Footwear 3.42 Wood and wood articles, charcoal 3.48
9 Plastic and plastic articles 3.12 Organic chemicals 3.43
10 Motor vehicles, trailers, bicycles, motorcycles 2.42 Plastic and plastic articles 2.92

Top 10 as % of total from China 76.92 Top 10 as % of total to China 76.17
Chinese imports as % of Cdn total 10.86 Chinese exports as % of Cdn total 3.27



Source: Trade Data Online. Industry Canada. 10 May 2010. http://www.ic.gc.ca/eic/site/tdo-dcd.nsf/eng/Home
Reproduced with the permission of the Minister of Public Works and Government Services Canada.

Note: All merchandise is assigned Harmonised System (HS) codes in order that accurate trade records can be kept, tariffs collected appropriately, customs procedures followed, and so forth. The HS system of codes, at its most disaggregated, assigns a 10-digit code to a given item. As the degree of aggregation increases, the number of digits in the code decreases. Thus, for example, the 6-digit HS code assigned to a motor vehicle with a spark-type ignition and a cylinder capacity of more than 3000 cc is 870323. The 2-digit HS code (in this case it is 87) includes not only this type of vehicle, but also many other types of vehicles and a wide variety of vehicle accessories and parts. The table above is based on Harmonised System (HS) codes at the 2-digit level. To learn more about the HS Code system used by Statistics Canada go to http://www.ic.gc.ca/eic/site/tdo-dcd.nsf/eng/h_00048.html





http://www.asiapacific.ca/statistics/trade/bilateral-trade-asia-product/canadas-merchandise-trade-china




TSS



flounder said:
Kato said:
So the answer is to strong arm China rather than ask them why they accepted Canadian beef and not American. :roll: :roll: :roll:

Quiz>>>

Can anyone here see why they would???? Think hard. There must be a reason.......
Click to expand...





Can you say TOYOTA. IT is a sad day when trade trumps human and animal health. as the case with the BSE MRR policy. Behind closed doors, the BSe spin machine is working i.e. Vilsack saying that 'The U.S. has had no cases in the last three years, and only three in two decades.' i can tell you with absolute certainty, that is only part of the story. i can tell you that in fact, the USDA BSE surveillance and testing have failed the consumer here in the USA, and abroad, and that we have been exposed to the TSE agent, i.e. USA atypical BSE, which laboratory studies show is more virulent. i can tell you with absolute certainty they infamous June 2004 enhanced BSE surveillance program, where some 800,000+ cattle were tested over many years of testing, was fraught with fraud, and in short, a failed program, and proven to be so by the GAO and the OIG, where it was proven that some of the testing program was using perfectly healthy cattle in their BSE testing program. Where some 9,200+ BSE test on suspect questionable cattle, only the IHC test were used. THE IHC is the least likely test to find BSE. IT only tells you if that part of the tissue sample is in fact infected or not, but it does not tell you about the rest of the brain. By only using the IHC, you miss many cases (Detwiler et al 2003 BSE ROUNDTABLE). i can tell you with absolute certainty, that when pressed, the USDA et al will say that even if we are missing cases of BSE, that the BSE mad cow feed ban of August 4, 1997, will stop BSE, but the ban was nothing more than ink on paper. This mad cow feed ban was only partial and voluntary. i can tell you with absolute certainty that in 2010, since 8/4/97, banned mad cow feed is in commerce here in the USA, BY THE TONS. i can tell you with absolute certainty, that when the BSE MRR policy was put in place, that this exposed everyone around the globe with the TSE agent, either by consumption and or friendly fire there from, and that decision was based NOT on science, but on trade. i can tell you with absolute certainty, that SINCE the USDA and the NSLP did in fact expose our children across the Nation with BSE via the NSLP USDA DEAD STOCK DOWNER COW SCHOOL LUNCH PROGRAM, that if they are capable of this, they are capable of exposing any person, in any country with the BSE TSE agent. North America has documented the so called typical c-BSE, l-BSE, and h-BSE. typical scrapie strains are rampant in the USA in sheep and goats, and the atypical Nor-98 scrapie is spreading, and CWD in deer and elk is spreading, with now documented a 2nd strain, and two strains of TME in mink. all this over the years have been fed back to food producing animals for animals and humans. Confucius ask, IF USA sheep Scrapie transmitted to USA cattle does not produce the same pathology as the U.K. c-BSE, why then would human CJD there from look like the U.K. nvCJD ??? what the USA has done defies all science and logic i.e. NO MAD COW DISEASE and or any human TSE there from. Either the BSE Mad Cow outbreak and human infection nvCJD there from, that happened in the U.K. was totally false, or the same thing is happening in the USA as we speak. sCJD of unknown phenotypes are rising in the USA. sporadic CJD is not a single strain, but multiple strains of unknown routes and sources of the TSE agent. IF the federal government can lie for almost a century about asbestos, and or tobacco, just to protect those two industry giants, i can guarantee you that they are doing it with mad cow type disease i.e. Transmissible Spongiform Encephalopathy. or just ask the Indians. ...TSS

full text ;

Saturday, April 10, 2010

TOYOTA VS MAD COW DISEASE USA OIE BSE MRR IMPORT AND EXPORT TRADE WARS



http://usdameatexport.blogspot.com/2010/04/toyota-vs-mad-cow-disease-usa-oie-bse.html




When the OIE and the USDA et al collaborated to make legal the trading of Transmissible Spongiform Encephalopathy, when they did away with the BSE GBR risk assessments, where the USA, Canada, and Mexico were categorized as BSE GBR III. please see ;

EFSA concludes that the current GBR level of USA is III, i.e. it is likely but not confirmed that domestic cattle are (clinically or pre-clinically) infected with the BSE-agent. As long as there are no significant changes in rendering or feeding, the stability remains extremely/very unstable. Thus, the probability of cattle to be (pre-clinically or clinically) infected with the BSE-agent persistently increases.




http://www.efsa.europa.eu/EFSA/efsa_locale-1178620753812_1211902594180.htm




Annex to the EFSA Scientific Report (2004) 3, 1-17 on the Assessment of the Geographical BSE Risk of USA

please see full text ;




http://www.efsa.europa.eu/en/scdocs/doc/3rax1.pdf





YET, in 2010, tons and tons of banned mad cow protein are still in commerce here in the USA, scientific studies are being misconstrued and manipulated by ARS USDA, which are still going by TSE science that is decades old, while refusing to acknowledge new scientific studies, and FOIA requests are still being held up by the USDA et al on these urgent matters (see source related materials below). CJD of unknown phenotype, in victims that are getting younger, with longer clinical course from first onset of symptoms to death are occurring, in fact, sporadic CJD is still rising, where the TSEs in the different species are mutating here in the USA, and we still have this same dog and pony show by the OIE and USDA et al. IF you go back and look at the Countries that went by these OIE BSE guidelines, most all came down with BSE. I have said it before, I was say it again now, OIE should hang up there jock strap now, since it appears they will buckle every time a country makes some political hay about trade protocol, commodities and futures. IF they are not going to be science based, they should do everyone a favor and dissolve there organization. ...TSS



Saturday, June 19, 2010

U.S. DENIED UPGRADED BSE STATUS FROM OIE




http://usdameatexport.blogspot.com/2010/06/us-denied-upgraded-bse-status-from-oie.html





****************PLEASE READ THE FOLLOWING CAREFULLY************



To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.



http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2





14th International Congress on Infectious Diseases H-type and L-type Atypical BSE January 2010 (special pre-congress edition)

18.173 page 189

Experimental Challenge of Cattle with H-type and L-type Atypical BSE

A. Buschmann1, U. Ziegler1, M. Keller1, R. Rogers2, B. Hills3, M.H. Groschup1. 1Friedrich-Loeffler-Institut, Greifswald-Insel Riems, Germany, 2Health Canada, Bureau of Microbial Hazards, Health Products & Food Branch, Ottawa, Canada, 3Health Canada, Transmissible Spongiform Encephalopathy Secretariat, Ottawa, Canada

Background: After the detection of two novel BSE forms designated H-type and L-type atypical BSE the question of the pathogenesis and the agent distribution of these two types in cattle was fully open. From initial studies of the brain pathology, it was already known that the anatomical distribution of L-type BSE differs from that of the classical type where the obex region in the brainstem always displays the highest PrPSc concentrations. In contrast in L-type BSE cases, the thalamus and frontal cortex regions showed the highest levels of the pathological prion protein, while the obex region was only weakly involved.

Methods:We performed intracranial inoculations of cattle (five and six per group) using 10%brainstemhomogenates of the two German H- and L-type atypical BSE isolates. The animals were inoculated under narcosis and then kept in a free-ranging stable under appropriate biosafety conditions.At least one animal per group was killed and sectioned in the preclinical stage and the remaining animals were kept until they developed clinical symptoms. The animals were examined for behavioural changes every four weeks throughout the experiment following a protocol that had been established during earlier BSE pathogenesis studies with classical BSE.

Results and Discussion: All animals of both groups developed clinical symptoms and had to be euthanized within 16 months. The clinical picture differed from that of classical BSE, as the earliest signs of illness were loss of body weight and depression. However, the animals later developed hind limb ataxia and hyperesthesia predominantly and the head. Analysis of brain samples from these animals confirmed the BSE infection and the atypical Western blot profile was maintained in all animals. Samples from these animals are now being examined in order to be able to describe the pathogenesis and agent distribution for these novel BSE types. Conclusions: A pilot study using a commercially avaialble BSE rapid test ELISA revealed an essential restriction of PrPSc to the central nervous system for both atypical BSE forms. A much more detailed analysis for PrPSc and infectivity is still ongoing.



http://www.isid.org/14th_icid/

http://ww2.isid.org/Downloads/IMED2009_AbstrAuth.pdf

http://www.isid.org/publications/ICID_Archive.shtml



14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

see page 114 ;



http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf





International Society for Infectious Diseases Web: http://www.isid.org



I ask Professor Kong ;

Thursday, December 04, 2008 3:37 PM Subject: RE: re--Chronic Wating Disease (CWD) and Bovine Spongiform Encephalopathies (BSE): Public Health Risk Assessment

''IS the h-BSE more virulent than typical BSE as well, or the same as cBSE, or less virulent than cBSE? just curious.....''

Professor Kong reply ;


.....snip


''As to the H-BSE, we do not have sufficient data to say one way or another, but we have found that H-BSE can infect humans. I hope we could publish these data once the study is complete.

Thanks for your interest.''

Best regards,

Qingzhong Kong, PhD Associate Professor Department of Pathology Case Western Reserve University Cleveland, OH 44106 USA

END...TSS



P02.35

Molecular Features of the Protease-resistant Prion Protein (PrPres) in H-type BSE

Biacabe, A-G1; Jacobs, JG2; Gavier-Widén, D3; Vulin, J1; Langeveld, JPM2; Baron, TGM1 1AFSSA, France; 2CIDC-Lelystad, Netherlands; 3SVA, Sweden

Western blot analyses of PrPres accumulating in the brain of BSE-infected cattle have demonstrated 3 different molecular phenotypes regarding to the apparent molecular masses and glycoform ratios of PrPres bands. We initially described isolates (H-type BSE) essentially characterized by higher PrPres molecular mass and decreased levels of the diglycosylated PrPres band, in contrast to the classical type of BSE. This type is also distinct from another BSE phenotype named L-type BSE, or also BASE (for Bovine Amyloid Spongiform Encephalopathy), mainly characterized by a low representation of the diglycosylated PrPres band as well as a lower PrPres molecular mass. Retrospective molecular studies in France of all available BSE cases older than 8 years old and of part of the other cases identified since the beginning of the exhaustive surveillance of the disease in 20001 allowed to identify 7 H-type BSE cases, among 594 BSE cases that could be classified as classical, L- or H-type BSE. By Western blot analysis of H-type PrPres, we described a remarkable specific feature with antibodies raised against the C-terminal region of PrP that demonstrated the existence of a more C-terminal cleaved form of PrPres (named PrPres#2 ), in addition to the usual PrPres form (PrPres #1). In the unglycosylated form, PrPres #2 migrates at about 14 kDa, compared to 20 kDa for PrPres #1. The proportion of the PrPres#2 in cattle seems to by higher compared to the PrPres#1. Furthermore another PK–resistant fragment at about 7 kDa was detected by some more N-terminal antibodies and presumed to be the result of cleavages of both N- and C-terminal parts of PrP. These singular features were maintained after transmission of the disease to C57Bl/6 mice. The identification of these two additional PrPres fragments (PrPres #2 and 7kDa band) reminds features reported respectively in sporadic Creutzfeldt-Jakob disease and in Gerstmann-Sträussler-Scheinker (GSS) syndrome in humans.



http://www.neuroprion.com/pdf_docs/conferences/prion2007/abstract_book.pdf





Wednesday, March 31, 2010

Atypical BSE in Cattle


http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html





Thursday, June 24, 2010

Accumulation of L-type Bovine Prions in Peripheral Nerve Tissues

Volume 16, Number 7–July 2010


http://bse-atypical.blogspot.com/2010/06/accumulation-of-l-type-bovine-prions-in.html





******$$$$$$******


Saturday, June 12, 2010

PUBLICATION REQUEST AND FOIA REQUEST Project Number: 3625-32000-086-05 Study of Atypical Bse



http://bse-atypical.blogspot.com/2010/06/publication-request-and-foia-request.html





TSS
Click to expand...
 
Flounder says: it's all about trade now, and science has taken a back seat to the show $$$
The science behind the BSE border closure has always been on the same level as the "settled science" behind Global Warming. Like Global Warmings tipping point being just around around the corner, I suppose we can expect the massive BSE induced human die off to begin any day now. Global Warming=Settled Science. Bullsh1t. BSE Border Closures=Science. Bullsh1t. It has always been about trade. And politics.Looking forward to your twenty two thousand word cut and paste reply.
 

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