We tested our bulls this past week for trich and don't have the results yet. I also took tissue samples just to rule out BVD-PIs.
What I am leaning towards is the possibility that it is nutritional as a result of the disease. I have done quite a bit of studying from various sources on the effects of lepto hardjo-bovis. There are several systems that it can affect. The most obvious one is reproductive. In addition to the reproductive is the possibility of mastitis. We have seen a fairly high rate of udder problems over the last several years, but I was attributing them to a shift in calving periods. We moved from February to May calving and I was thinking that the better nutrition when our cows were freshening was causing the problems with the udders. After studying some on HB I am now beginning to suspect that my udder problems may have its roots in lepto instead of nutritional. Thirdly there is also evidence that lepto can damage the kidneys in some cases.
This is where I am making some leaps that I am not able to completely defend with the research available to me but it seems fairly logical, others may disagree. I am thinking now that during the last couple of years we have had a fairly high rate of HB infection on these heifers, starting as calves. Because it was undiagnosed and untreated it ended up damaging the kidneys and as a result limits the animals overall ability to thrive on the feed they are getting. Calving in May means breeding in August. The last couple of years we have had unusually wet springs followed by very dry summers. This leads to pastures that are pretty well burned up by the time we are going into breeding season. On the cows that don't have damaged metabolic systems the feed is still adequate, because we are a short grass prairie ecosystem, to meet the nutritional requirements of the animals. The animals that have compromised systems, because of the damage from the lepto, may not be able to extract adequate nutrition from the dry grasses to meet their maintenance needs. That would put them into a decreasing plane of nutrition, and as a result an anestrous state that they don't recover from until the following spring when they are once again on abundant, highly nutritious grass.
I don't know if this is the correct explanation, but it is the best that I can come up with seeing as how our production system, i.e. the pastures we run on and the nutritional profile the cattle are exposed to, hasn't changed in any significant manner for quite some time. I would like it to be something as simple as putting out some mineral to solve the problem, but I am afraid that the effects of this infection are going to take a few years to be resolved.