[TAHC News] FYI: Info Regarding Disease Rumor in Texas

[flounder]

[TAHC News] FYI: Info Regarding Disease Rumor in Texas


Good afternoon:

Today we have received a number of calls regarding a reported case
of sporadic CJD, or Creutzfeldt-Jakob Disease, in Texas.
We have received no official written statement or announcement from
public health authorities regarding this case.

However, according to Texas' Department of State Health Services,
Texas had 11 sporadically occurring cases of CJD in 2007, and
5 in 2006. About 300 sporadic cases occur in the U.S. each year.

We have learned that this possible CJD case prompted another rumor
that Texas may have a BSE in cattle.
We have no indication of any pending BSE cases.

There is no known link between sporadic CJD in humans and BSE in cattle.

We wanted to ensure that you were provided the best information we
have regarding this rumor, in case you received calls
or were asked about the case.

Carla Everett
Public Information
Texas Animal Health Commission


===============================================END...TSS


News: Page (1) of 1 - 11/19/08 No human mad cow case in Texas panhandle: officials By Scott Malone CHICAGO (Reuters) - There has been no human case of mad cow disease in the Texas panhandle area in the past month, the Texas Department of State Health Services said on Wednesday in response to rumors in Chicago markets of a woman patient there with the disease.

Cattle futures plummeted at the Chicago Mercantile Exchange amid a variety of mad cow-related rumors, one of which was that a woman was being treated for the disease in an Amarillo hospital.

"We do not have any confirmed cases of CJD or variant CJD in that area in the last month," a health department spokesman said referring to the Panhandle area of Texas.

Mad cow is a fatal brain disease in cattle and it is believed humans can contract a fatal variation of it by eating infected parts of animals with the disease.

The United States has had three cases of mad cow disease in cattle.

(Reporting by Bob Burgdorfer; Editing by Christian Wiessner)

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http://theusdaily.com/articles/viewarticle.jsp?id=586147&type=home#



EMBO J. 2002 December 2; 21(23): 6358–6366.
doi: 10.1093/emboj/cdf653. PMCID: PMC136957

Copyright © 2002 European Molecular Biology Organization
BSE prions propagate as either variant CJD-like or sporadic CJD-like prion strains in transgenic mice expressing human prion protein
Emmanuel A. Asante, Jacqueline M. Linehan, Melanie Desbruslais, Susan Joiner, Ian Gowland, Andrew L. Wood, Julie Welch, Andrew F. Hill, Sarah E. Lloyd, Jonathan D.F. Wadsworth, and John Collinge1
MRC Prion Unit and Department of Neurodegenerative Disease, Institute of Neurology, University College, Queen Square, London WC1N 3BG, UK 1Corresponding author e-mail: [email protected].
Received August 1, 2002; Revised September 24, 2002; Accepted October 17, 2002.


ABSTRACT


Variant Creutzfeldt–Jakob disease (vCJD) has been recognized to date only in individuals homozygous for methionine at PRNP codon 129. Here we show that transgenic mice expressing human PrP methionine 129, inoculated with either bovine spongiform encephalopathy (BSE) or variant CJD prions, may develop the neuropathological and molecular phenotype of vCJD, consistent with these diseases being caused by the same prion strain. Surprisingly, however, BSE transmission to these transgenic mice, in addition to producing a vCJD-like phenotype, can also result in a distinct molecular phenotype that is indistinguishable from that of sporadic CJD with PrPSc type 2. These data suggest that more than one BSE-derived prion strain might infect humans; it is therefore possible that some patients with a phenotype consistent with sporadic CJD may have a disease arising from BSE exposure.

http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=136957



Tuesday, November 11, 2008


Transmission of atypical bovine prions to mice transgenic for human prion protein
DOI: 10.3201/eid1412.080941


http://bse-atypical.blogspot.com/2008/11/transmission-of-atypical-bovine-prions.html


2:00 Sporadic CJD and Atypical BSE: Two Children of One Protein Maurizio Pocchiari, Ph.D., Director of Research, Virology, Istituto Superiore Di Sanita The identification of forms of TSE diseases in cattle caused by prion strains different from BSE has raised new concerns on the possibility that these novel agents might induce disease in humans with a phenotype resembling sporadic CJD. The analysis of the distribution of the different molecular subtypes of sporadic CJD might give some answers.

http://www.healthtech.com/Conferences_Overview.aspx?c=518&id=59662&c=518



USA PRION UNIT BLOG

http://prionunitusaupdate2008.blogspot.com/


Sunday, April 20, 2008 Progress Report from the National Prion Disease Pathology Surveillance Center April 3, 2008

Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.

see full text ;

http://prionunitusaupdate2008.blogspot.com/2008/04/progress-report-from-national-prion.html


CJD TEXAS (cjd clusters)

http://cjdtexas.blogspot.com/


USA WRITTEN CJD QUESTIONNAIRE ???

http://cjdquestionnaire.blogspot.com/


The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html


Tuesday, August 19, 2008

Atypical BSE (BASE) Transmitted from Asymptomatic Aging Cattle to a Primate

http://bse-atypical.blogspot.com/2008/08/atypical-bse-base-transmitted-from.html


Review on the epidemiology and dynamics of BSE epidemics

Vet. Res. (2008) 39:15 www.vetres.org DOI: 10.1051/vetres:2007053 c INRA, EDP Sciences, 2008 Review article

snip...

And last but not least, similarities of PrPres between Htype BSE and human prion diseases like CJD or GSS have been put forward [10], as well as between L-type BSE and CJD [17]. These findings raise questions about the origin and inter species transmission of these prion diseases that were discovered through the BSE active surveillance.

snip...

Cases of atypical BSE have only been found in countries having implemented large active surveillance programs. As of 1st September 2007, 36 cases (16 H, 20 L) have been described all over the world in cattle: Belgium (1 L) [23], Canada (1 H)15, Denmark (1 L)16, France (8 H, 6 L)17, Germany (1 H, 1 L) [13], Italy (3 L)18, Japan (1 L) [71], Netherlands (1 H, 2 L)19, Poland (1 H, 6 L)20, Sweden (1 H)21, United Kingdom (1 H)22, and USA (2 H)23. Another H-type case has been found in a 19 year old miniature zebu in a zoological park in Switzerland [56]. It is noteworthy that atypical cases have been found in countries that did not experience classical BSE so far, like Sweden, or in which only few cases of classical BSE have been found, like Canada or the USA.

And last but not least, similarities of PrPres between Htype BSE and human prion diseases like CJD or GSS have been put forward [10], as well as between L-type BSE and CJD [17]. These findings raise questions about the origin and inter species transmission of these prion diseases that were discovered through the BSE active surveillance.

full text 18 pages ;

http://www.vetres.org/index.php?option=article&access=standard&Itemid=129&url=/articles/vetres/pdf/2008/04/v07232.pdf


please see full text ;

http://bse-atypical.blogspot.com/2008/06/review-on-epidemiology-and-dynamics-of.html


***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.***

Progress Report from the National Prion Disease Pathology Surveillance Center

An Update from Stephen M. Sergay, MB, BCh & Pierluigi Gambetti, MD

April 3, 2008

http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45


Sunday, March 16, 2008

MAD COW DISEASE terminology UK c-BSE (typical), atypical BSE H or L, and or Italian L-BASE

http://bse-atypical.blogspot.com/2008/03/mad-cow-disease-terminology-uk-c-bse.html


Wednesday, October 08, 2008

Idiopathic Brainstem Neuronal Chromatolysis (IBNC): a novel prion protein related disorder of cattle?

http://bse-atypical.blogspot.com/2008/10/idiopathic-brainstem-neuronal.html

HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD only theory JUNE 2008

snip...

Tissue infectivity and strain typing of the many variants of the human and animal TSEs are paramount in all variants of all TSE. There must be a proper classification that will differentiate between all these human TSE in order to do this. With the CDI and other more sensitive testing coming about, I only hope that my proposal will some day be taken seriously. ...

snip...

http://cjdmadcowbaseoct2007.blogspot.com/2008/06/human-and-animal-tse-classifications-ie.html


ONE HUNDRED AND FIRST MEETING OF THE SPONGIFORM ENCEPHALOPATHY ADVISORY COMMITTEE
Oct 23, 2008 at 9:00 AM

http://seac992007.blogspot.com/2008/10/one-hundred-and-first-meeting-of_23.html

http://flounder068.vox.com/library/post/one-hundred-and-first-meeting-of-the-spongiform-encephalopathy-advisory-committee.html


Wednesday, October 08, 2008

Idiopathic Brainstem Neuronal Chromatolysis (IBNC): a novel prion protein related disorder of cattle?

http://bse-atypical.blogspot.com/2008/10/idiopathic-brainstem-neuronal.html

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

snip...SEE FULL TEXT with facts and sources @ ;

http://usdavskorea.blogspot.com/2008/06/oie-recognition-of-bse-status-of.html

http://organicconsumers.org/forum/index.php?showtopic=1566

Friday, April 25, 2008

Substances Prohibited From Use in Animal Food or Feed [Docket No. 2002N-0273] (Formerly Docket No. 02N-0273) RIN 0910-AF46

http://madcowfeed.blogspot.com/2008/04/substances-prohibited-from-use-in.html


***The pathology features of Nor98 in the cerebellum of the affected sheep showed similarities with those of sporadic Creutzfeldt-Jakob disease in humans.

http://www.prion2007.com/pdf/Prion%20Book%20of%20Abstracts.pdf


Here we report that both Nor98 and discordant cases, including three sheep homozygous for the resistant PrPARR allele (A136R154R171), efficiently transmitted the disease to transgenic mice expressing ovine PrP, and that they shared unique biological and biochemical features upon propagation in mice. These observations support the view that a truly infectious TSE agent, unrecognized until recently, infects sheep and goat flocks and may have important implications in terms of scrapie control and public health.

Edited by Stanley B. Prusiner, University of California, San Francisco, CA, and approved September 12, 2005 (received for review March 21, 2005)

http://www.pnas.org/cgi/content/abstract/0502296102v1


Tuesday, June 3, 2008 SCRAPIE USA UPDATE JUNE 2008 NOR-98 REPORTED PA

http://nor-98.blogspot.com/2008/06/scrapie-usa-update-june-2008-nor-98.html


NOR-98 ATYPICAL SCRAPIE 5 cases documented in USA in 5 different states USA 007

http://nor-98.blogspot.com/2008/04/seac-spongiform-encephalopathy-advisory.html


http://nor-98.blogspot.com/


SCRAPIE USA

http://scrapie-usa.blogspot.com/


Sunday, September 07, 2008

CWD LIVE TEST, and the political aspects or fallout of live testing for BSE in cattle in the USA

http://chronic-wasting-disease.blogspot.com/2008/09/cwd-live-test-and-political-aspects-or.html

Saturday, October 18, 2008 WYOMING STAR VALLEY MOOSE TESTS POSITIVE FOR CWD

http://chronic-wasting-disease.blogspot.com/2008/10/wyoming-star-valley-moose-tests.html

http://chronic-wasting-disease.blogspot.com/


Wednesday, August 20, 2008


Bovine Spongiform Encephalopathy Mad Cow Disease typical and atypical strains, was there a cover-up ?

http://bse-atypical.blogspot.com/2008/08/bovine-spongiform-encephalopathy-mad.html


Public release date: 9-Jul-2008

Contact: Claire Bowles [email protected]. 44-207-611-1210 New Scientist

10 people killed by new CJD-like disease A NEW form of fatal dementia has been discovered in 16 Americans, 10 of whom have already died of the condition. It resembles Creutzfeldt-Jakob disease - with patients gradually losing their ability to think, speak and move - but has features that make it distinct from known forms of CJD.

No one yet knows how the disease originates, or under what conditions it might spread. Nor is it clear how many people have the condition. "I believe the disease has been around for many years, unnoticed," says Pierluigi Gambetti, director of the US National Prion Disease Pathology Surveillance Center at Case Western Reserve University in Cleveland, Ohio. Cases may previously have been mistaken for other forms of dementia.

Since Gambetti's team wrote a paper describing an initial 11 cases referred to his centre between 2002 and 2006 (Annals of Neurology, vol 63, p 697), another five have come to light. "So it is possible that it could be just the tip of the iceberg," Gambetti says.

As in other spongiform encephalopathies, such as CJD and mad cow disease (BSE), the brain tissue of victims is full of tiny holes. This damage is thought to be caused by the accumulation of prions, misfolded versions of a brain protein called PrP that can convert normal PrP molecules into their own misshapen form.

Some features of the new disease are different, however. All known disease-causing prions resist degradation by proteases - enzymes which digest the normal form of PrP. But prions from patients with the new disease are broken down by the enzymes.

Some very rare forms of CJD run in families and are caused by mutations in the gene for PrP. Six of the cases described in Gambetti's paper were from families with a history of dementia, suggesting a genetic cause. However, these people had no mutations in their PrP genes. "Maybe there are other genes that have an influence on the disease," suggests James Ironside of the UK's National CJD Surveillance Unit in Edinburgh.

Most forms of CJD develop spontaneously, for unknown reasons, but can be spread if someone is exposed to brain material from people with CJD, for instance, by neurosurgery using inadequately sterilised instruments.

One variant of CJD has been linked to the consumption of contaminated meat from cattle with mad cow disease. If the new condition is similarly caused by something in the victims' diet, or another environmental cause, new measures might be needed to protect public health.

Gambetti is now conducting experiments in mice to see how the disease is transmitted. He suspects that there is no cause for alarm. "I believe the disease occurs naturally, and is not due to environmental causes," he says.

###

New Scientist Reporter: Andy Coghlan

IF REPORTING ON THIS STORY, PLEASE MENTION NEW SCIENTIST AS THE SOURCE AND, IF REPORTING ONLINE, PLEASE CARRY A LINK TO: http://www.newscientist.com

UK CONTACT - Claire Bowles, New Scientist Press Office, London: Tel: +44(0)20 7611 1210 or email [email protected].

US CONTACT – New Scientist Boston office: Tel: +1 617 386 2190 or email [email protected].

This article is posted on this site to give advance access to other authorised media who may wish to report on this story, or quote extracts as part of fair dealing with this copyrighted material. Full attribution is required, and if reporting online a link to www.newscientist.com is also required. The story posted here is copyrighted therefore advance permission is required before any and every reproduction of each article in full. Please contact [email protected].

THIS ARTICLE APPEARS IN NEW SCIENTIST MAGAZINE ISSUE: 12 JULY 2008. EMBARGOED UNTIL WED, 9 JULY 2008, 13:00 HRS EDT (18:00 HRS BST)

EDITOR'S NOTE: PRIOR PERMISSION IS REQUIRED BEFORE ANY REPRODUCTION OF THIS STORY IN FULL


http://www.eurekalert.org/pub_releases/2008-07/ns-tpk070908.php


A New Prionopathy OR more of the same old BSe and sporadic CJD

http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html


Communicated by: Terry S. Singeltary Sr.

[In submitting these data, Terry S. Singeltary Sr. draws attention to the steady increase in the "type unknown" category, which, according to their definition, comprises cases in which vCJD could be excluded. The total of 26 cases for the current year (2007) is disturbing, possibly symptomatic of the circulation of novel agents. Characterization of these agents should be given a high priority. - Mod.CP]

http://pro-med.blogspot.com/2007/11/proahedr-prion-disease-update-2007-07.html

http://www.promedmail.org/pls/askus/f?p=2400:1001:6833194127530602005::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1010,39963


There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection.

He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf


sporadic Fatal Familial Insomnia

http://sporadicffi.blogspot.com/


JOURNAL OF NEUROLOGY

MARCH 26, 2003

RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob

disease in the United States

Email Terry S. Singeltary:

[email protected]

I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?

http://www.neurology.org/cgi/eletters/60/2/176#535


THE PATHOLOGICAL PROTEIN

Hardcover, 304 pages plus photos and illustrations. ISBN 0-387-95508-9

June 2003

BY Philip Yam

CHAPTER 14 LAYING ODDS

Answering critics like Terry Singeltary, who feels that the U.S. under- counts CJD, Schonberger conceded that the current surveillance system has errors but stated that most of the errors will be confined to the older population.

http://www.thepathologicalprotein.com/


Diagnosis and Reporting of Creutzfeldt-Jakob Disease Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.

Terry S. Singeltary, Sr Bacliff, Tex

1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323. FREE FULL TEXT

http://jama.ama-assn.org/cgi/content/extract/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT

http://jama.ama-assn.org/cgi/content/full/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT


2 January 2000 British Medical Journal U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well

http://www.bmj.com/cgi/eletters/320/7226/8/b#6117


15 November 1999 British Medical Journal vCJD in the USA * BSE in U.S.

http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406


Creutzfeldt Jakob Disease

http://creutzfeldt-jakob-disease.blogspot.com/


USA PRION UNIT BLOG

http://prionunitusaupdate2008.blogspot.com/


Sunday, April 20, 2008 Progress Report from the National Prion Disease Pathology Surveillance Center April 3, 2008

Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.

see full text ;

http://prionunitusaupdate2008.blogspot.com/2008/04/progress-report-from-national-prion.html


CJD TEXAS (cjd clusters)

http://cjdtexas.blogspot.com/


USA WRITTEN CJD QUESTIONNAIRE ???

http://cjdquestionnaire.blogspot.com/


The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html


http://madcowtesting.blogspot.com/



TSS

 

[Tex]

The blood bank is sure interested in this issue as it had several questions about it. Wonder why the USDA and the blood bank disagree over "science"?

 

[hypocritexposer]

USDA?, why are the beef producers within Texas denying it. They should be demanding testing.

Haymaker demands it everytime there is a positive in Canada, forgetting that it was that very testing that found it in the first place, not chance!

 

[flounder]

Friday, November 21, 2008

Amarillo-area (suspect sporadic CJD) case linked to mad cow disease Rumor in Texas

http://cjdtexas.blogspot.com/2008/11/amarillo-area-suspect-sporadic-cjd-case.html


TSS

 

[Tex]

Friday, November 21, 2008

Amarillo-area (suspect sporadic CJD) case linked to mad cow disease Rumor in Texas

http://cjdtexas.blogspot.com/2008/11/amarillo-area-suspect-sporadic-cjd-case.html


TSS

Anyone on the chain of command responsible for the irresponsible govt. policy on bse should be held responsible for the deaths and or deterioration of anyone coming down with this disease, both financially and criminally. Getting appointed to the job and doing the bidding of money over their government responsibilities should have a cost.

 

[mrj]

Where is that link showing a POSITIVE cause and effect betweena human eating beef and having vCJD????

mrj

 

[flounder]

Where is that link showing a POSITIVE cause and effect betweena human eating beef and having vCJD????

mrj

the closest you have is oral transmission of bse to primate, which has been proven. it's against the law to do the same studies on humans, which i have argued against. atypical bse is even more virulent. you know all this there mrj. i think studies should be done on humans as opposed to primates for ''bse transmissions studies to humans for dummies''. long ago i proposed a 'death row inmate' trade off of such, instead of primates, as such studies we speak of, with a compensation package for living relatives of such 'death row inmates', for scientific research for 'dummies that just don't get it', and such, to finally prove, something of which to date is impossible to prove but only 99% out of 100% i.e. the difference between human and primate study. but i for one would rather see a human in the study as opposed to the helpless primate. you should step up to the plate there mrj, and eat a handful of srm's from a fresh case of atypical texas style bse, or better yet, feed it to one of your family members for a long case study. ...TSS

 

[hypocritexposer]

Pretty sure Texans don't eat SRM's, but they do eat BSE contaminated beef. No body has proven to me yet that BSE contaminated beef is a danger though.

They are taking all precauations in Texas, aren't they? If not, they should be taking an example from Canada, and do the same as us.

The world would trust them more, that's for sure

 

[Tex]

Where is that link showing a POSITIVE cause and effect betweena human eating beef and having vCJD????

mrj

MRJ, why is the medical community worried about this as evidenced by the blood donor questionaire? If the concern wasn't "sound" science, it wouldn't be on the blood donor questionaire. I guess the the only "proof" you accept is your own opinion. Do something good for others and go give blood. See for yourself.

Tex

 

[PORKER]

Ron, How is the status on your live BSE urine test ?

 

[hypocritexposer]

Urine test, who needs it to prove that a geographic area has had BSE within it's boundaries.

Texas has tested positive how many times, within it's cattle population?

They should be cut off from the world market, just like Alberta, unless they haven't been honest about it, and aren't testing!

 

[MsSage]

Since she is in Amarillo I will guess to say she came from farther out and not a city resident.
Let me fill you in on the Drs/hospitals out here. :shock: :shock: :shock:
One lady went in ( a Vet) told them her apendex ruptured it took them 6 cat scans then they said no it was leaking and for her to go home. She went back the next morning and they had to rush her to Amarillo because she was septic. :roll: I can give 100's of examples of major mistakes.
The joke at the farm is if you want me to die take me to the local hospital.

Texas Public Health didnt even look at the xrays that were taken here for TB. Amarillo Public Health just put me on the meds ie ..Treat me for it.
I ams sure they are running more complet tests but in the mean time they have to report it to Public Health as a person being treated.
I pray she does NOT have it.

 

[hillsdown]

Urine test, who needs it to prove that a geographic area has had BSE within it's boundaries.

Texas has tested positive how many times, within it's cattle population?

They should be cut off from the world market, just like Alberta, unless they haven't been honest about it, and aren't testing!

Do you actually read your posts after you have typed them in or do you just type in gibberish and hope that they post.

 

[mrj]

Tex, I donate blood frequently, the last time in Oct. and will do so again when eligible due to time since previous donation. I don't recall being asked any questions about BSE, but plenty of others to protect the blood supply.

Such foolishness posted on this thread. We sure don't need any enemies of beef with all these "friends" posting on this site!!!!

The best of science and some common sense is my criteria for what I believe about BSE, e coli's, and other animal diseases, for the record.

mrj

 

[Kato]

:agree:

It's all a matter of perspective and personal agendas. I always found it amazing that something like BSE, which after all these years, the cause of it is still being debated, can be used as a tool to grind trade to a halt, and cause an immense amount of damage to entire sections of an economy.

After so many years, we can't even agree on what causes this disease, and the actual number of people who've contracted and died from the new variant CJD probably doesn't add up to how many people in the U.S. are shot by guns on any given day. :shock:

Think about this, and how silly it is....

There's a big car accident on the freeway. It involves a bus carrying 23 people, and perhaps, a VW Rabbit. No one has proven it was the car involved, but since there was one on the freeway that day, it was a possibility. The brass at GM runs to Washington to insist that the government close the border to all car imports from Germany. After all, it could have been a German car that caused this accident, so therefore all German cars are deadly, and will cause the end of the world as we know it. America instantly shuts it's doors to German cars, and all Americans can rest easy knowing those deadly Rabbits are off the road now. All Americans that is, except those working for VW dealerships, and those driving VW cars that are now worth less than before. No matter about that, no one needs a job selling these dangerous Rabbits anyway. After all one of them just might have killed 23 people.

In the meantime, the evening news reports that thousands of people a year die of smoking related illnesses, and drunk driving accidents. But that doesn't matter, does it. After all, those needless deaths are not the problem of the brass at GM. They don't need attention because it's far more important to close the door to Rabbits.

Silly, right? :???: :???:

Just fill in the blanks with the appropriate subject matter of cows and trade, and you get the picture. You have a disease that even if anyone could quit debating it's cause, has been a much smaller player in the big picture than the hype would like us to believe. I would bet that there are many many more people killed in a year by mad cows, by which I mean actual angry cows, than have been killed by so called Mad Cow disease. I bet we all know of more than one story of someone we know being actually physically killed by a cow than we will ever know having had CJD.

All this sensationalism over this is wearing pretty thin. The only bright spot is that if you hear this garbage repeated enough times, and don't see the apocolyptic follow up, you quit listening to it. Just like I've quit reading certain posts on this subject. I think everyone knows which ones I mean.

Keep it in perspective. There are much more dangerous things in this world than theoretical diseases. :!:

 

[Tex]

Tex, I donate blood frequently, the last time in Oct. and will do so again when eligible due to time since previous donation. I don't recall being asked any questions about BSE, but plenty of others to protect the blood supply.

Such foolishness posted on this thread. We sure don't need any enemies of beef with all these "friends" posting on this site!!!!

The best of science and some common sense is my criteria for what I believe about BSE, e coli's, and other animal diseases, for the record.

mrj

MRJ, this is from Canada:

http://www.phac-aspc.gc.ca/cjd-mcj/vcjd-faq-03-eng.php

This is from the CDC:

http://www.cdc.gov/ncidod/dvrd/vcjd/qa.htm

This is from the High Plains Journal:

BSE questions raised after Brit infected via transfusion

Doctor: Safety of U.S. blood supply not in doubt

WASHINGTON (AP)--Federal health advisers unanimously agreed Oct. 14 that current safeguards on blood donations in the U.S. are sufficient despite the disclosure that a second British resident most likely acquired the human form of bovine spongiform encephalopathy through a tainted transfusion.

"The United States is not Great Britain," said Dr. Stephen DeArmond, a University of California-San Francisco pathology professor. DeArmond pointed to the lack of evidence that so-far symptomless infections of the human variant of bovine spongiform encephalopathy, suspected in thousands of British residents are an issue in this country.

The Food and Drug Administration advisory committee agreed that donor restrictions are still warranted. But it stopped short of asking for additional safeguards in the U.S. to further prevent the risk of transmission of BSE through blood.

The agency already bars blood donations from people who lived more than three months in Great Britain or who received transfusions there after 1979.

In both confirmed British infections, recipients got blood from donors who were young and apparently healthy at the time of donation. The chances that two confirmed infections among people who received blood was caused instead by eating BSE-tainted beef were remote--1 in 80,000 to 1 in 1 billion, the panel was told.

Getting the human form of BSE, variant Creutzfeldt-Jakob disease, through tainted blood "is a reality. And we've got to treat it like that," said Steven Anderson, an FDA risk assessment expert.

The new case involves a woman in her 80s who died from an unrelated medical condition early this year, according to Dr. Robert Will, a neurologist who founded the British BSE surveillance team.

Because the woman had received a blood transfusion in 1999 from a donor who later died from variant CJD, the woman's doctor did a more thorough analysis after her death.

The woman had no outward signs of neurological disease and her brain, spinal cord, tonsils and appendix were normal, Will told the panel. In her spleen and cervical lymph nodes, however, the woman harbored malformed, infective prion proteins.

The disease occurs when normal proteins found in the brain, known as prions, change shape and coax adjacent healthy prions to do the same. When enough prions are altered, they deposit a plaque on the brain and surround the mark with spongy holes, killing the victim.

By testing 14,964 appendices of normal surgical patients in England, researchers learned that three were tainted with the infective proteins. That implies 237 people per million British residents--or 3,808 people age 10 to 30--could silently incubate Creutzfeldt-Jakob disease, Will said.

G. Michael Fitzpatrick, chief policy officer at America's Blood Centers, acknowledged worries about a potential second wave of BSE cases in England.

Still, current safeguards appear to be working in the U.S., Fitzpatrick said. Already, the nation's major blood suppliers, ABC and the American Red Cross, estimate current deferrals block 400,000 previous blood donors.

Instead of additional donor restrictions, the FDA should consider an "exit strategy" that would set thresholds for ending the blood supply sapping measures, Fitzpatrick said.

The federal advisers soundly reject that notion, agreeing unanimously that the current donor deferral strategies are still warranted to reduce the risk in blood.

Before the second blood transfusion infection was reported in England, Dr. Arthur Bracey wondered whether restrictions in the U.S. might ease.

"I think that would be premature now," said Bracey, associate chief of pathology at St. Luke's Episcopal Hospital in Houston. He pointed to anecdotal information sent to the committee about a high-risk donor thwarting restrictions in order to sell his blood.

Current restrictions ban blood donations from people who lived for more than five years in much of Western Europe, where Creutzfeldt-Jakob cases among humans have been reported.

Still the advisory panel didn't decide to extend blood donor restrictions to other countries that recently discovered BSE in cattle. That includes Canada, Israel and Japan, which confirmed its 14th case of BSE on Oct. 14.

Date: 10/28/04

MRJ, you were either asleep when they asked you the questions or have a memory like my kids when I ask them who did it.



Tex

 

[flounder]

TRANSFUSION MEDICINE

Prion diseases are efficiently transmitted by blood transfusion in sheep


Fiona Houston1, Sandra McCutcheon1, Wilfred Goldmann2, Angela Chong2, James Foster2, Silvia Sisó3, Lorenzo González3, Martin Jeffrey3, and Nora Hunter2 1 Neuropathogenesis Division, Roslin Institute, Compton, United Kingdom; 2 Neuropathogenesis Division, Roslin Institute, University of Edinburgh, Edinburgh, United Kingdom; and 3 Lasswade Laboratory, Veterinary Laboratories Agency, Penicuik, United Kingdom

The emergence of variant Creutzfeld-Jakob disease, following on from the bovine spongiform encephalopathy (BSE) epidemic, led to concerns about the potential risk of iatrogenic transmission of disease by blood transfusion and the introduction of costly control measures to protect blood supplies. We previously reported preliminary data demonstrating the transmission of BSE and natural scrapie by blood transfusion in sheep. The final results of this experiment, reported here, give unexpectedly high transmission rates by transfusion of 36% for BSE and 43% for scrapie. A proportion of BSE-infected tranfusion recipients (3 of 8) survived for up to 7 years without showing clinical signs of disease. The majority of transmissions resulted from blood collected from donors at more than 50% of the estimated incubation period. The high transmission rates and relatively short and consistent incubation periods in clinically positive recipients suggest that infectivity titers in blood were substantial and/or that blood transfusion is an efficient method of transmission. This experiment has established the value of using sheep as a model for studying transmission of variant Creutzfeld-Jakob disease by blood products in humans.


http://bloodjournal.hematologylibrary.org/cgi/content/abstract/112/12/4739?ct



Plasma & Serum Proteins Receive Continued FDA Approval

4/25/2008
APC, Inc. is pleased to advise our customers and industry partners that as anticipated, the Food and Drug Administration (FDA) will continue to allow the use of bovine blood, plasma and serum proteins in ruminant feeds.


In April 2008 FDA announced the publication of its Final Rule for 21 CFR Part 589.2001 - Substances Prohibited From Use in Animal Food or Feed. FDA specifically stated in their opinion that, "FDA is not prohibiting the use of blood and blood products in animal feed because we believe such a prohibition would do very little to reduce the risk of BSE transmission."


Known as a leader in developing nutritional products for the swine industry, where 95% of pig starter diets in the United States contain functional proteins, APC has more recently developed their line of colostrum replacers, supplements, feed additives and milk replacer ingredients for calves. Products include plasma, serum and immunoglobulin concentrate based Acquire®, Lifeline®, Gammulin® and Nutrapro® used to optimize the health and performance of calves.


To view the full report for Final Rule 21 CFR Part 589.2001 visit:

http://www.fda.gov/OHRMS/DOCKETS/98fr/FDA-2002-N-0031-nfr.pdf


To view the complete Feed Rule 21 CFR Part 589 visit:

http://www.accessdata.fda.gov/scripts/cdrh/cfdocs/cfcfr/CFRSearch.cfm?CFRPart=589&showFR=1


Thursday, November 27, 2008

Prion diseases are efficiently transmitted by blood transfusion in sheep

http://vcjdblood.blogspot.com/2008/11/prion-diseases-are-efficiently.html

TSS

 

[mrj]

Sorry, Tex, I'll pay more attention when I donate again in a couple of weeks, OK?

I'm quite sure it was not in the context of "have you been in a country rife with BSE in cattle?", but possibly question of whether I'd travelled to some specific country and I didn't put the two together.

mrj

 

[Kato]

There are all kinds of restrictions on who can and cannot donate blood in Canada. Most came about after a terrible disaster a few years ago with hepatitis and hiv. The following lawsuits were huge, and caused the Canadian Red Cross to get out of the blood bank services entirely.

The forms you need to fill in to donate have a very long list of different countries and time frames, and are aimed at a lot of health issues. For instance, they won't take blood from anyone who has handled monkeys. :shock: Go figure. I wonder how many people have been turned down because they've been in contact with a monkey. :???:

Basically I think they look at whatever potential problems may arise, and cover their bases accordingly. They err on the side of caution, and until someone finally settles this seemingly unending debate over the method of transmission of BSE, it will stay on the radar.

As long as there is debate, they will avoid any potential problems no matter how slim they may be.

And we all know the debate is a long way from being settled. :roll: :roll:

 

[Tex]

Sorry, Tex, I'll pay more attention when I donate again in a couple of weeks, OK?

I'm quite sure it was not in the context of "have you been in a country rife with BSE in cattle?", but possibly question of whether I'd travelled to some specific country and I didn't put the two together.

mrj

Good enough, MRJ. Just follow up and ask innocent questions about the questions they are asking you and see what you find out. Thanks for giving blood, you have a bonus point in my book for that effort, if that means anything.

Tex